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On the origin of foam cells in cholesterol-induced atherosclerosis of the rabbit.

作者信息

Barbolini G, Scilabra G A, Botticelli A, Botticelli S

出版信息

Virchows Arch B Cell Pathol. 1969;3(1):24-32. doi: 10.1007/BF02901924.

DOI:10.1007/BF02901924
PMID:4239560
Abstract
摘要

相似文献

1
On the origin of foam cells in cholesterol-induced atherosclerosis of the rabbit.兔胆固醇诱导动脉粥样硬化中泡沫细胞的起源
Virchows Arch B Cell Pathol. 1969;3(1):24-32. doi: 10.1007/BF02901924.
2
[The activity of phosphomonoesterase, carboxylesterase, lactate dehydrogenase and beta-glucuronidase in blood serum, liver, kidney and aorta in rabbit atherosclerosis].[兔动脉粥样硬化时血清、肝脏、肾脏和主动脉中磷酸单酯酶、羧酸酯酶、乳酸脱氢酶和β-葡萄糖醛酸酶的活性]
Enzymol Biol Clin (Basel). 1970;11(6):521-30.
3
Lysosomes of the arterial wall. IV. Cytochemical localization of acid phosphatase and catalase in smooth muscle cells and foam cells from rabbit atheromatous aorta.动脉壁的溶酶体。IV. 兔动脉粥样硬化主动脉平滑肌细胞和泡沫细胞中酸性磷酸酶和过氧化氢酶的细胞化学定位
Am J Pathol. 1974 Jul;76(1):1-16.
4
[The histoenzymological characteristics of hydrolytic enzymes of the arterial wall at the initial stage of atherosclerosis].
Ann Anat Pathol (Paris). 1969 Jul-Sep;14(3):267-74.
5
Enzyme histochemical observations on the effect of pyridinol carbamate on cholesterol-induced atherosclerosis.关于吡啶醇氨基甲酸酯对胆固醇诱导的动脉粥样硬化影响的酶组织化学观察
Atherosclerosis. 1972 Jan-Feb;15(1):77-82. doi: 10.1016/0021-9150(72)90039-1.
6
[The enzyme-histochemical behavior of the liver after intravenous papain application].[静脉注射木瓜蛋白酶后肝脏的酶组织化学行为]
Acta Biol Med Ger. 1967;18(3):394-407.
7
Tissue fractionation. Past and present.组织分级分离:过去与现在
J Cell Biol. 1971 Jul;50(1):20d-55d.
8
Histochemical study of Mallory bodies.马洛里小体的组织化学研究。
Acta Pathol Microbiol Scand A. 1971;79(6):649-57. doi: 10.1111/j.1699-0463.1971.tb01867.x.
9
An electron microscopy study of phosphatase activity in the endothelial cells of rabbit aorta.兔主动脉内皮细胞中磷酸酶活性的电子显微镜研究。
J Histochem Cytochem. 1966 Oct;14(10):719-24. doi: 10.1177/14.10.719.
10
The effect of carbon tetrachloride on histochemical reactions in the rat liver.
Folia Histochem Cytochem (Krakow). 1972;10(1):57-84.

引用本文的文献

1
HIV-derived ssRNA binds to TLR8 to induce inflammation-driven macrophage foam cell formation.源自HIV的单链RNA与Toll样受体8(TLR8)结合,以诱导炎症驱动的巨噬细胞泡沫细胞形成。
PLoS One. 2014 Aug 4;9(8):e104039. doi: 10.1371/journal.pone.0104039. eCollection 2014.
2
The macrophage: the intersection between HIV infection and atherosclerosis.巨噬细胞:HIV 感染与动脉粥样硬化的交汇点。
J Leukoc Biol. 2010 Apr;87(4):589-98. doi: 10.1189/jlb.0809580. Epub 2009 Dec 1.
3
Initial characterization of a peripheral blood mononuclear cell chemoattractant derived from cultured arterial smooth muscle cells.
源自培养动脉平滑肌细胞的外周血单核细胞趋化因子的初步特性分析。
Am J Pathol. 1984 Dec;117(3):409-17.
4
Lysosomes of the arterial wall. IV. Cytochemical localization of acid phosphatase and catalase in smooth muscle cells and foam cells from rabbit atheromatous aorta.动脉壁的溶酶体。IV. 兔动脉粥样硬化主动脉平滑肌细胞和泡沫细胞中酸性磷酸酶和过氧化氢酶的细胞化学定位
Am J Pathol. 1974 Jul;76(1):1-16.
5
Evolution of foam cells in subcutaneous rabbit carrageenan granulomas. II. Tissue and macrophage lipid composition.皮下注射角叉菜胶所致兔肉芽肿中泡沫细胞的演变。II. 组织和巨噬细胞的脂质组成。
Am J Pathol. 1985 Sep;120(3):391-401.
6
Evolution of foam cells in subcutaneous rabbit carrageenan granulomas: I. Light-microscopic and ultrastructural study.皮下注射角叉菜胶诱导的兔肉芽肿中泡沫细胞的演变:I. 光镜和超微结构研究
Am J Pathol. 1985 Jan;118(1):134-50.
7
Elevated expression of monocyte chemoattractant protein 1 by vascular smooth muscle cells in hypercholesterolemic primates.高胆固醇血症灵长类动物血管平滑肌细胞中单核细胞趋化蛋白1的表达升高。
Proc Natl Acad Sci U S A. 1992 Aug 1;89(15):6953-7. doi: 10.1073/pnas.89.15.6953.
8
Restitution of aortic wall after sustained necrotizing transmural ligation injury. Role of blood cells and artery cells.持续性坏死性透壁结扎损伤后主动脉壁的修复。血细胞和动脉细胞的作用。
Am J Pathol. 1975 Apr;79(1):7-30.
9
[Mesenchymal reactions of the aortic wall in atherogenesis by adrenalin-thyroxin and fat diet in rabbit. Ultrastructural and histoenzymologic study (author's transl)].[肾上腺素-甲状腺素及高脂饮食致兔动脉粥样硬化过程中主动脉壁的间充质反应。超微结构及组织酶学研究(作者译)]
Virchows Arch A Pathol Anat Histol. 1977 Dec 29;377(1):49-66. doi: 10.1007/BF00432698.