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Repair in arterial tissue. Morphological and biochemical changes in rabbit aorta after a single dilatation injury.

作者信息

Helin P, Lorenzen I, Garbarsch C, Matthiessen M E

出版信息

Circ Res. 1971 Nov;29(5):542-54. doi: 10.1161/01.res.29.5.542.

DOI:10.1161/01.res.29.5.542
PMID:4256306
Abstract
摘要

相似文献

1
Repair in arterial tissue. Morphological and biochemical changes in rabbit aorta after a single dilatation injury.动脉组织修复。单次扩张损伤后兔主动脉的形态学和生化变化。
Circ Res. 1971 Nov;29(5):542-54. doi: 10.1161/01.res.29.5.542.
2
Arteriosclerosis in rabbit aorta induced by mechanical dilatation. Biochemical and morphological studies.
Atherosclerosis. 1971 May-Jun;13(3):319-31. doi: 10.1016/0021-9150(71)90075-x.
3
Arteriosclerosis and hypoxia. 2. Biochemical changes in mucopolysaccharides and collagen of rabbit aorta induced by systemic hypoxia.动脉硬化与缺氧。2. 全身性缺氧诱导兔主动脉黏多糖和胶原蛋白的生化变化。
J Atheroscler Res. 1969 May-Jun;9(3):295-304. doi: 10.1016/s0368-1319(69)80024-4.
4
Repair processes in vascular connective tissue following experimental injury.
Angiology. 1972 Apr;23(4):183-7. doi: 10.1177/000331977202300401.
5
[The intermediate substance of the aorta in dissecting aneurysm in comparison with various diseases].
Acta Biol Med Ger. 1969;23(2):323-8.
6
[Acid mucopolysaccharides of the arterial wall, age and arteriosclerosis].[动脉壁的酸性粘多糖、年龄与动脉硬化]
Arkh Patol. 1968;30(11):3-14.
7
Arteriosclerosis in rabbit aorta induced by systemic hypoxia. Biochemical and morphologic studies.
Angiology. 1969 Jan;20(1):1-12. doi: 10.1177/000331976902000101.
8
Vascular injury compared to ageing of normal rabbit aorta. Biochemical and histochemical studies on time-dependent alterations of vascular connective tissue.
Blood Vessels. 1985;22(2):94-104.
9
Mucopolysaccharides of rabbit bone marrow cells. Identification and properties.兔骨髓细胞的粘多糖。鉴定与特性
Exp Cell Res. 1971 Aug;67(2):416-26. doi: 10.1016/0014-4827(71)90427-7.
10
The mucopolysaccharides (glycosaminoglycans) in fracture callus.
Isr J Med Sci. 1971 Mar;7(3):416-7.

引用本文的文献

1
Differences in the synthesis and secretion of sulfated glycosaminoglycans by aorta explant monolayers cultured from atherosclerosis-susceptible and -resistant pigeons.从易患和抗动脉粥样硬化的鸽子培养的主动脉外植体单层中硫酸化糖胺聚糖合成与分泌的差异。
Am J Pathol. 1980 Oct;101(1):127-42.
2
Suppression of experimental atherosclerosis by the Ca++-antagonist lanthanum. Possible role of calcium in atherogenesis.钙离子拮抗剂镧对实验性动脉粥样硬化的抑制作用。钙在动脉粥样硬化发生中的可能作用。
J Clin Invest. 1980 May;65(5):967-81. doi: 10.1172/JCI109783.
3
Atherosclerosis following balloon catheter injury to the carotid artery and the aorta of hypertensive rats with normolipidemia or hyperlipidemia.
正常血脂或高脂血症高血压大鼠颈动脉和主动脉球囊导管损伤后的动脉粥样硬化
Am J Pathol. 1981 Apr;103(1):105-15.
4
Susceptibility to atherosclerosis in aortas and coronary arteries of swine with von Willebrand's disease.患有血管性血友病的猪的主动脉和冠状动脉对动脉粥样硬化的易感性。
Am J Pathol. 1981 Feb;102(2):137-45.
5
Lipoprotein and albumin accumulation in reendothelialized and deendothelialized aorta.脂蛋白和白蛋白在再内皮化和去内皮化主动脉中的积聚。
Am J Pathol. 1984 Jan;114(1):112-20.
6
Effect of endothelium on glycosaminoglycan accumulation in injured rabbit aorta.内皮对兔主动脉损伤后糖胺聚糖积聚的影响。
Am J Pathol. 1983 Nov;113(2):156-64.
7
Repair in arterial tissue. Demonstration of fibrinogen/fibrin in the normal and healing rabbit thoracic aorta by the indirect immunoperoxidase technique.动脉组织修复。采用间接免疫过氧化物酶技术在正常及愈合中的兔胸主动脉中显示纤维蛋白原/纤维蛋白。
Virchows Arch A Pathol Anat Histopathol. 1984;403(2):163-71. doi: 10.1007/BF00695232.
8
Repair in arterial tissue. Demonstration of fibronectin in the normal and healing rabbit thoracic aorta by the indirect immunoperoxidase technique.动脉组织修复。通过间接免疫过氧化物酶技术显示正常及愈合中的兔胸主动脉中的纤连蛋白。
Virchows Arch A Pathol Anat Histopathol. 1983;399(3):307-16. doi: 10.1007/BF00612948.
9
Demonstration of fibronectin in normal and injured aorta by an indirect immunoperoxidase technique.采用间接免疫过氧化物酶技术对正常和损伤主动脉中的纤连蛋白进行检测。
Histochemistry. 1983;77(3):395-403. doi: 10.1007/BF00490900.
10
Glycosaminoglycans in the rat aorta. Ultrastructural localization with toluidine blue O and osmium--ferrocyanide procedure.大鼠主动脉中的糖胺聚糖。用甲苯胺蓝O和锇-亚铁氰化物法进行超微结构定位。
Am J Pathol. 1981 Dec;105(3):232-40.