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伪狂犬病病毒光复活作用的特性

Characteristics of the photoreactivation of pseudorabies virus.

作者信息

Pfefferkorn E R, Burge B W, Coady H M

出版信息

J Bacteriol. 1966 Oct;92(4):856-61. doi: 10.1128/jb.92.4.856-861.1966.

DOI:10.1128/jb.92.4.856-861.1966
PMID:4288801
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC276345/
Abstract

Pfefferkorn, E. R. (Harvard Medical School, Boston, Mass.), Boyce W. Burge, and Helen M. Coady. Characteristics of the photoreactivation of pseudorabies virus. J. Bacteriol. 92:856-861. 1966.-Pseudorabies virus, killed with ultraviolet light and then allowed to adsorb to chick embryo tissue culture cells, can be photoreactivated by light of longer wavelengths. The photoreactivable sector is approximately 0.5. Herpes simplex virus is equally photoreactivable. Photoreactivability is negligible during the first 20 min after adsorption and then rises to a maximum by 100 min. After this maximum is reached, photoreactivability decreases with increasing rate as the temperature of incubation is raised. Inhibition of protein synthesis immediately after absorption prevents the establishment of the photoreactivable state.

摘要

费弗科恩,E.R.(马萨诸塞州波士顿哈佛医学院),博伊斯·W.伯奇,海伦·M.科迪。伪狂犬病病毒光复活作用的特性。《细菌学杂志》92:856 - 861。1966年。——用紫外线灭活后再使其吸附于鸡胚组织培养细胞的伪狂犬病病毒,可被较长波长的光光复活。可光复活部分约为0.5。单纯疱疹病毒同样可光复活。吸附后最初20分钟内光复活能力可忽略不计,然后在100分钟时升至最大值。达到此最大值后,随着培养温度升高,光复活能力以递增速率下降。吸附后立即抑制蛋白质合成可阻止光复活状态的建立。

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Characteristics of the photoreactivation of pseudorabies virus.伪狂犬病病毒光复活作用的特性
J Bacteriol. 1966 Oct;92(4):856-61. doi: 10.1128/jb.92.4.856-861.1966.
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引用本文的文献

1
Mechanism of photeractivation of pseudorabies virus.伪狂犬病病毒光激活机制。
J Virol. 1968 May;2(5):474-9. doi: 10.1128/JVI.2.5.474-479.1968.
2
Photoreactivation in an established vertebrate cell line.在一个已建立的脊椎动物细胞系中的光复活作用。
Proc Natl Acad Sci U S A. 1967 Dec;58(6):2274-9. doi: 10.1073/pnas.58.6.2274.
3
Photoreactivation of a cytoplasmic virus.一种细胞质病毒的光复活作用。
J Virol. 1972 Mar;9(3):474-8. doi: 10.1128/JVI.9.3.474-478.1972.

本文引用的文献

1
Experiments on photoreactivation of bacteriophages inactivated with ultraviolet radiation.用紫外线辐射灭活的噬菌体的光复活实验。
J Bacteriol. 1950 Mar;59(3):329-47. doi: 10.1128/jb.59.3.329-347.1950.
2
THE INDUCTION OF THYMINE DIMERS IN ULTRAVIOLET-IRRADIATED MAMMALIAN CELLS.紫外线照射哺乳动物细胞时胸腺嘧啶二聚体的诱导形成
Radiat Res. 1965 Apr;24:667-72.
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CYCLOHEXIMIDE: ASPECTS OF INHIBITION OF PROTEIN SYNTHESIS IN MAMMALIAN CELLS.环己酰亚胺:哺乳动物细胞中蛋白质合成抑制的相关方面
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THE INTRACELLULAR FATE OF RABBITPOX VIRUS RENDERED NONINFECTIOUS BY VARIOUS REAGENTS.经各种试剂处理后失去感染性的兔痘病毒在细胞内的命运
Virology. 1964 Apr;22:620-33. doi: 10.1016/0042-6822(64)90084-4.
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RELEASE OF ULTRAVIOLET LIGHT-INDUCED THYMINE DIMERS FROM DNA IN E. COLI K-12.大肠杆菌K-12中DNA上紫外线诱导胸腺嘧啶二聚体的释放
Proc Natl Acad Sci U S A. 1964 Feb;51(2):293-300. doi: 10.1073/pnas.51.2.293.
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THE DISAPPEARANCE OF THYMINE DIMERS FROM DNA: AN ERROR-CORRECTING MECHANISM.胸腺嘧啶二聚体从DNA中的消失:一种纠错机制。
Proc Natl Acad Sci U S A. 1964 Feb;51(2):226-31. doi: 10.1073/pnas.51.2.226.
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PURIFICATION AND PARTIAL CHEMICAL ANALYSIS OF SINDBIS VIRUS.辛德毕斯病毒的纯化与部分化学分析
Virology. 1963 Jul;20:433-45. doi: 10.1016/0042-6822(63)90092-8.
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Disappearance of thymine photodimer in ultraviolet irradiated DNA upon treatment with a photoreactivating enzyme from baker's yeast.用来自面包酵母的光复活酶处理后,紫外线照射的DNA中胸腺嘧啶光二聚体的消失
Biochem Biophys Res Commun. 1962 Apr 20;7:237-40. doi: 10.1016/0006-291x(62)90181-x.
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Evidence that ultraviolet-induced thymine dimers in DNA cause biological damage.有证据表明,DNA中紫外线诱导的胸腺嘧啶二聚体会造成生物损伤。
Proc Natl Acad Sci U S A. 1962 Jul 15;48(7):1250-7. doi: 10.1073/pnas.48.7.1250.
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Photoreactivation.光复活作用
Bacteriol Rev. 1958 Jun;22(2):99-142. doi: 10.1128/br.22.2.99-142.1958.