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Inheritance of responsiveness to pertussis HSF in mice.

作者信息

Wardlaw A C

出版信息

Int Arch Allergy Appl Immunol. 1970;38(6):573-89. doi: 10.1159/000230313.

DOI:10.1159/000230313
PMID:4315845
Abstract
摘要

相似文献

1
Inheritance of responsiveness to pertussis HSF in mice.
Int Arch Allergy Appl Immunol. 1970;38(6):573-89. doi: 10.1159/000230313.
2
Further studies on the inheritance of responsiveness to pertussis HSF in mice.
Int Arch Allergy Appl Immunol. 1975;48(1):11-5. doi: 10.1159/000231288.
3
Some histamine sensitizing properties of soluble preparations of the histamine sensitizing factor(HSF) from Bordetella pertussis.
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4
Hypoproteinemia in mice after treatment with histamine-sensitizing factor from Bordetella pertussis.
Proc Soc Exp Biol Med. 1969 Jul;131(3):964-9. doi: 10.3181/00379727-131-34020.
5
Histamine-sensitising factor of Bordetella pertussis differentiated from immunogens by neutralisation and passive protection tests.通过中和试验和被动保护试验，区分百日咳博德特氏菌的组胺致敏因子与免疫原。
J Pathol Bacteriol. 1967 Apr;93(2):483-92. doi: 10.1002/path.1700930209.
6
Adjuvant activity of the histamine-sensitizing factor of Bordetella pertussis in different strains of mice.百日咳博德特氏菌组胺致敏因子在不同品系小鼠中的佐剂活性。
Int Arch Allergy Appl Immunol. 1975;49(6):796-813. doi: 10.1159/000231464.
7
Enhancing effect of Bordetella pertussis and propranolol on experimental immune hemolytic anemia.百日咳博德特氏菌与普萘洛尔对实验性免疫性溶血性贫血的增强作用。
Experientia. 1971 Mar 15;27(3):332-3. doi: 10.1007/BF02138180.
8
Immunologic and biochemical properties of the histamine-sensitizing factor from Bordetella pertussis.百日咳博德特氏菌组胺致敏因子的免疫学和生化特性。
J Immunol. 1975 Jan;114(1 Pt 1):34-9.
9
Histopathological changes in mice treated with extracts of the histamine-sensitizing factor of Bordetella pertussis.用百日咳博德特氏菌组胺致敏因子提取物处理的小鼠的组织病理学变化。
Int Arch Allergy Appl Immunol. 1977;54(2):129-42. doi: 10.1159/000231816.
10
Modified state of susceptibility to histamine in mice treated with Bordetella pertussis during the neonatal period.新生期感染百日咳博德特氏菌的小鼠对组胺敏感性的改变状态。
Z Immunitatsforsch Allerg Klin Immunol. 1970 Oct;140(4):326-34.

引用本文的文献

1
A genetic locus complements resistance to Bordetella pertussis-induced histamine sensitization.一个遗传基因座互补了对百日咳博德特氏菌诱导的组氨酸致敏的抗性。
Commun Biol. 2023 Mar 6;6(1):244. doi: 10.1038/s42003-023-04603-w.
2
G proteins Gαi1/3 are critical targets for Bordetella pertussis toxin-induced vasoactive amine sensitization.G蛋白Gαi1/3是百日咳博德特氏菌毒素诱导的血管活性胺致敏作用的关键靶点。
Infect Immun. 2014 Feb;82(2):773-82. doi: 10.1128/IAI.00971-13. Epub 2013 Dec 9.
3
von-Willebrand factor influences blood brain barrier permeability and brain inflammation in experimental allergic encephalomyelitis.
血管性血友病因子影响实验性变态反应性脑脊髓炎中的血脑屏障通透性和脑炎症。
Am J Pathol. 2008 Sep;173(3):892-900. doi: 10.2353/ajpath.2008.080001. Epub 2008 Aug 7.
4
Analysis of the role of Bphs/Hrh1 in the genetic control of responsiveness to pertussis toxin.分析Bphs/Hrh1在百日咳毒素反应性基因控制中的作用。
Infect Immun. 2003 Mar;71(3):1281-7. doi: 10.1128/IAI.71.3.1281-1287.2003.
5
Locus controlling Bordetella pertussis-induced histamine sensitization (Bphs), an autoimmune disease-susceptibility gene, maps distal to T-cell receptor beta-chain gene on mouse chromosome 6.控制百日咳博德特氏菌诱导的组胺致敏作用的基因座（Bphs）是一种自身免疫性疾病易感基因，定位于小鼠6号染色体上T细胞受体β链基因的远端。
Proc Natl Acad Sci U S A. 1993 Apr 15;90(8):3700-4. doi: 10.1073/pnas.90.8.3700.
6
Acute autoimmune encephalomyelitis in mice. II. Susceptibility is controlled by the combination of H-2 and histamine sensitization genes.小鼠急性自身免疫性脑脊髓炎。II. 易感性由H-2和组胺致敏基因的组合控制。
J Exp Med. 1982 Jul 1;156(1):31-40. doi: 10.1084/jem.156.1.31.
7
Independent segregation of the H-2 locus and the locus for responsiveness to histamine-sensitizing factor.H-2基因座与对组胺致敏因子反应性基因座的独立分离。
Proc Natl Acad Sci U S A. 1973 Sep;70(9):2500-1. doi: 10.1073/pnas.70.9.2500.
8
Experimental allergic orchitis in mice. II. Association of disease susceptibility with the locus controlling Bordetella pertussis-induced sensitivity to histamine.小鼠实验性变应性睾丸炎。II. 疾病易感性与控制百日咳博德特氏菌诱导的组胺敏感性的基因座的关联。
Immunogenetics. 1985;22(5):417-25. doi: 10.1007/BF00418088.
9
Inheritance of lipopolysaccharide-enhanced nonspecific resistance to infection and of susceptibility to endotoxic shock in lipopolysaccharide low-responder mice.脂多糖低反应小鼠中脂多糖增强的非特异性抗感染抵抗力及对内毒素休克易感性的遗传
Infect Immun. 1977 May;16(2):432-38. doi: 10.1128/iai.16.2.432-438.1977.