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[高碳酸血症后代谢性碱中毒的生理病理及临床资料。一例重度高碳酸血症经药物治疗及使用“铁肺”的病例]

[Physiopathological and clinical data on post-hypercapnic metabolic alkalosis. A case of severe hypercapnia treated with drugs and in an "iron lung"].

作者信息

Ferrara A, Ferrario E, Basani F, Sunseri S, Fumagalli G

出版信息

Minerva Med. 1979 Jan 7;70(1):67-73.

PMID:431833
Abstract

The Authors produce a further contribution on metabolic post-hypercapnic alkalosis on the basis of clinical observation of a patient with severe hypercapnia and respiratory failure undergoing intensive care as well as treatment with iron lung (Pulmolife). The improvement of respiratory acid-base umbalance was associated with alteration of electrolytic assessment, especially of the plasmatic chloride ratio. The Authors consider this remark as a starting point for a physiopathological pattern and a more complete analysis of the pathways which generate the metabolic post-hypercapnic alkalosis condition in such patients.

摘要

作者基于对一名患有严重高碳酸血症和呼吸衰竭且正在接受重症监护以及使用铁肺(Pulmolife)治疗的患者的临床观察,对代谢性高碳酸血症后碱中毒做出了进一步贡献。呼吸酸碱失衡的改善与电解质评估的改变有关,尤其是血浆氯化物比值。作者认为这一发现是一种生理病理模式的起点,也是对在此类患者中产生代谢性高碳酸血症后碱中毒状况的途径进行更全面分析的起点。

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