Actions of 1,25-dihydroxycholecalciferol in patients with hypophosphatemic, vitamin-D-resistant rickets.

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作者信息

Brickman A S, Coburn J W, Kurokawa K, Bethune J E, Harrison H E, Norman A W

出版信息

N Engl J Med. 1973 Sep 6;289(10):495-8. doi: 10.1056/NEJM197309062891002.

DOI:10.1056/NEJM197309062891002

PMID:4353218

Abstract

摘要

相似文献

Actions of 1,25-dihydroxycholecalciferol in patients with hypophosphatemic, vitamin-D-resistant rickets.

N Engl J Med. 1973 Sep 6;289(10):495-8. doi: 10.1056/NEJM197309062891002.

Familial hypophosphatemia: the dilemma of treatment.家族性低磷血症：治疗的困境

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Pathogenesis of hereditary vitamin-D-dependent rickets. An inborn error of vitamin D metabolism involving defective conversion of 25-hydroxyvitamin D to 1 alpha,25-dihydroxyvitamin D.遗传性维生素D依赖性佝偻病的发病机制。一种维生素D代谢的先天性缺陷，涉及25-羟基维生素D向1α,25-二羟基维生素D的转化缺陷。

N Engl J Med. 1973 Oct 18;289(16):817-22. doi: 10.1056/NEJM197310182891601.

Bone response to phosphate salts, ergocalciferol, and calcitriol in hypophosphatemic vitamin D-resistant rickets.低磷性维生素D抵抗性佝偻病中骨骼对磷酸盐、麦角钙化醇和骨化三醇的反应

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X-linked hypophosphataemic rickets: Inadequate therapeutic response to 1,25-dihydroxycholecalciferol.

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Acceleration of growth with combined vitamin D-phosphate therapy of hypophosphatemic resistant rickets.

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[Administration of phosphates and 1 alpha-hydroxyvitamin D3 during the postoperative immobilization of children with familial hypophosphatemic rickets].

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[Hypophosphatemic vitamin D resistant rickets. Therapeutic management].[低磷性维生素D抵抗性佝偻病。治疗管理]

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Nature of defect responsible for familial vitamin D-resistant rickets (VDRR) based on radioimmunoassay for parathyroid hormone (PTH).基于甲状旁腺激素（PTH）放射免疫测定法的家族性维生素D抵抗性佝偻病（VDRR）相关缺陷的性质

Trans Assoc Am Physicians. 1972;85:172-80.

Hypophosphatemic vitamin D-resistant rickets: metabolic balance studies in a child receiving 1,25 dihydroxyvitamin D3, phosphate, and ascorbic acid.

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引用本文的文献

Therapeutics of X-linked hypophosphatemic rickets.X连锁低磷血症性佝偻病的治疗方法。

Pediatr Nephrol. 1993 Dec;7(6):744-8. doi: 10.1007/BF01213343.

Autosomal hypophosphataemic bone disease responds to 1,25-(OH)2D3.常染色体性低磷性骨病对1,25 -（OH）2D3有反应。

Arch Dis Child. 1981 Mar;56(3):203-7. doi: 10.1136/adc.56.3.203.

Evaluation of a role for 1,25-dihydroxyvitamin D3 in the pathogenesis and treatment of X-linked hypophosphatemic rickets and osteomalacia.评估1,25-二羟维生素D3在X连锁低磷性佝偻病和骨软化症的发病机制及治疗中的作用。

J Clin Invest. 1980 Nov;66(5):1020-32. doi: 10.1172/JCI109930.

Metabolism and action of the hormone vitamin D. Its relation to diseases of calcium homeostasis.激素维生素D的代谢与作用。及其与钙稳态疾病的关系。

West J Med. 1974 Jul;121(1):22-44.

Healing of bone disease in X-linked hypophosphatemic rickets/osteomalacia. Induction and maintenance with phosphorus and calcitriol.X连锁低磷性佝偻病/骨软化症中骨病的治疗。磷和骨化三醇的诱导与维持治疗。

J Clin Invest. 1985 Jun;75(6):1858-68. doi: 10.1172/JCI111900.

Phosphorus deficiency, parathyroid hormone and bone resorption in the growing rat.生长中大鼠的磷缺乏、甲状旁腺激素与骨吸收

Calcif Tissue Res. 1976 Jun 14;20(3):235-49. doi: 10.1007/BF02546412.

Recessive hypophosphataemic rickets, and possible aetiology of the 'vitamin D-resistant' syndrome.隐性低磷血症性佝偻病以及“维生素D抵抗”综合征的可能病因。

Arch Dis Child. 1976 May;51(5):360-5. doi: 10.1136/adc.51.5.360.

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