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Enzymic activities and sodium, potassium and copper concentrations in mouse brain and liver after cuprizone treatment in vivo.

作者信息

Venturini G

出版信息

J Neurochem. 1973 Nov;21(5):1147-51. doi: 10.1111/j.1471-4159.1973.tb07569.x.

DOI:10.1111/j.1471-4159.1973.tb07569.x
PMID:4357499
Abstract
摘要

相似文献

1
Enzymic activities and sodium, potassium and copper concentrations in mouse brain and liver after cuprizone treatment in vivo.
J Neurochem. 1973 Nov;21(5):1147-51. doi: 10.1111/j.1471-4159.1973.tb07569.x.
2
Mechanism of the formation of megamitochondria induced by copper-chelating agents. II. Isolation and some properties of megamitochondria from the cuprizone-treated mouse liver.
Acta Pathol Jpn. 1975 Jan;25(1):39-49.
3
Monoamine oxidase inhibition and the activity of other oxidative enzymes in the brains of mice fed cuprizone.用双环已酮草酰二腙喂养的小鼠大脑中的单胺氧化酶抑制作用及其他氧化酶的活性
Toxicol Appl Pharmacol. 1971 Nov;20(3):386-95. doi: 10.1016/0041-008x(71)90281-x.
4
Effect of cuprizone feeding on hepatic superoxide dismutase and cytochrome oxidase activities in mice.用铜螯合剂喂养对小鼠肝脏超氧化物歧化酶和细胞色素氧化酶活性的影响。
Experientia. 1982 Jul 15;38(7):784-5. doi: 10.1007/BF01972269.
5
Biochemical effects of cuprizone on mouse liver and heart mitochondria.铜螯合剂对小鼠肝脏和心脏线粒体的生化作用。
Biochem Pharmacol. 1973 Sep 15;22(18):2311-8. doi: 10.1016/0006-2952(73)90012-9.
6
Mechanism of the formation of megamitochondria induced by copper-chelating agents. III. Formation and some biochemical properties of megamitochondria induced by diethyldithiocarbamate (DDC).
Acta Pathol Jpn. 1975 Mar;25(2):125-34. doi: 10.1111/j.1440-1827.1975.tb00852.x.
7
Division of giant mitochondria during recovery from cuprizone intoxication.从铜离子螯合剂中毒恢复过程中巨大线粒体的分裂
J Cell Biol. 1973 Jan;56(1):266-72. doi: 10.1083/jcb.56.1.266.
8
Giant hepatic mitochondria: production in mice fed with cuprizone.巨大的肝脏线粒体:在喂食双硫仑的小鼠中的产生
Science. 1969 Jan 3;163(3862):81-2. doi: 10.1126/science.163.3862.81.
9
Mechanism of the formation of megamitochondria by copper-chelating agents. V. Further studies on isolated megamitochondria.
Acta Pathol Jpn. 1978 Mar;28(2):215-23. doi: 10.1111/j.1440-1827.1978.tb00533.x.
10
Purification and properties of an amine oxidase in bovine dental pulp and its comparison with serum amine oxidase.牛牙髓中胺氧化酶的纯化、性质及其与血清胺氧化酶的比较。
Biochim Biophys Acta. 1974 Apr 25;341(2):366-77. doi: 10.1016/0005-2744(74)90229-0.

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Integrated elemental analysis supports targeting copper perturbations as a therapeutic strategy in multiple sclerosis.综合元素分析支持将铜扰动作为多发性硬化症的一种治疗策略。
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Microglial Neuroinflammation-Independent Reversal of Demyelination of Corpus Callosum by Arsenic in a Cuprizone-Induced Demyelinating Mouse Model.砷在 cuprizone 诱导的脱髓鞘小鼠模型中通过小胶质细胞神经炎症非依赖性逆转胼胝体脱髓鞘。
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在给予铜离子载体杯状霉素处理的小鼠的胼胝体中发现与脱髓鞘相关的铜含量降低。
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Glutamate delta-1 receptor regulates oligodendrocyte progenitor cell differentiation and myelination in normal and demyelinating conditions.谷氨酸 delta-1 受体调节正常和脱髓鞘条件下少突胶质前体细胞的分化和髓鞘形成。
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The Tryptophan-Kynurenine Metabolic System Is Suppressed in Cuprizone-Induced Model of Demyelination Simulating Progressive Multiple Sclerosis.在模拟进行性多发性硬化的铜离子螯合剂诱导的脱髓鞘模型中,色氨酸-犬尿氨酸代谢系统受到抑制。
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Sulforaphane, an Nrf-2 Agonist, Modulates Oxidative Stress and Inflammation in a Rat Model of Cuprizone-Induced Cardiotoxicity and Hepatotoxicity.萝卜硫素,一种 Nrf-2 激动剂,可调节 CPZ 诱导的心脏毒性和肝毒性大鼠模型中的氧化应激和炎症。
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