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Epileptic brain damage in adolescent baboons following seizures induced by allylgycine.

作者信息

Meldrum B S, Horton R W, Brierley J B

出版信息

Brain. 1974 Jun;97(2):407-18. doi: 10.1093/brain/97.1.407.

DOI:10.1093/brain/97.1.407
PMID:4434186
Abstract
摘要

相似文献

1
Epileptic brain damage in adolescent baboons following seizures induced by allylgycine.烯丙基甘氨酸诱发癫痫发作后青春期狒狒的癫痫性脑损伤
Brain. 1974 Jun;97(2):407-18. doi: 10.1093/brain/97.1.407.
2
Seizures induced by allylglycine, 3-mercaptopropionic acid and 4-deoxypyridoxine in mice and photosensitive baboons, and different modes of inhibition of cerebral glutamic acid decarboxylase.烯丙基甘氨酸、3-巯基丙酸和4-脱氧吡哆醇在小鼠和光敏狒狒中诱发的癫痫发作，以及脑谷氨酸脱羧酶的不同抑制模式。
Br J Pharmacol. 1973 Sep;49(1):52-63. doi: 10.1111/j.1476-5381.1973.tb08267.x.
3
Neuronal loss and gliosis in the hippocampus following repetitive epileptic seizures induced in adolescent baboons by allylglycine.烯丙基甘氨酸诱导青春期狒狒反复癫痫发作后海马中的神经元丢失和胶质增生。
Brain Res. 1972 Dec 24;48:361-5. doi: 10.1016/0006-8993(72)90190-4.
4
Physiological changes during prolonged seizures and epileptic brain damage.长时间癫痫发作及癫痫性脑损伤期间的生理变化。
Neuropadiatrie. 1978 Aug;9(3):203-12. doi: 10.1055/s-0028-1091481.
5
Systemic factors and epileptic brain damage. Prolonged seizures in paralyzed, artificially ventilated baboons.全身因素与癫痫性脑损伤。瘫痪、人工通气的狒狒的长时间癫痫发作。
Arch Neurol. 1973 Aug;29(2):82-7. doi: 10.1001/archneur.1973.00490260026003.
6
Cerebral metabolism during induced seizures in man and animals.人类和动物诱发性癫痫发作期间的脑代谢
Riv Patol Nerv Ment. 1970 Dec;91(6):345-56.
7
Hippocampal lesions produced by prolonged seizures in paralyzed artificially ventilated baboons.在人工通气的麻痹狒狒中，长时间癫痫发作所导致的海马损伤。
Experientia. 1973 May 15;29(5):561-3. doi: 10.1007/BF01926665.
8
Cerebral energy metabolism and the regulation of cerebral blood flow.脑能量代谢与脑血流调节
Trans Am Neurol Assoc. 1973;98:153-5.
9
Possible role of gangliosides in epilepsy: effects of epileptic seizures on cerebral gangliosides.神经节苷脂在癫痫中的可能作用：癫痫发作对脑内神经节苷脂的影响。
Trans Am Neurol Assoc. 1975;100:261-3.
10
Four models for studying cerebral lesions secondary to epileptic seizures.四种用于研究癫痫发作继发脑损伤的模型。
Adv Neurol. 1975;10:147-61.

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Effects of Prolonged Seizures on Basal Forebrain Cholinergic Neurons: Evidence and Potential Clinical Relevance.长时间癫痫发作对基底前脑胆碱能神经元的影响：证据与潜在临床相关性。
Neurotox Res. 2020 Aug;38(2):249-265. doi: 10.1007/s12640-020-00198-w. Epub 2020 Apr 21.
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Hippocampal neuropathology of domoic acid-induced epilepsy in California sea lions (Zalophus californianus).多莫酸诱导的加利福尼亚海狮（加州海狗）癫痫的海马神经病理学
J Comp Neurol. 2014 May 1;522(7):1691-706. doi: 10.1002/cne.23509.
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Classic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single "cryptic" episode of focal hippocampal excitation in awake rats.
清醒大鼠单一“隐匿性”海马局部兴奋发作致经典海马硬化和海马起始性癫痫
J Comp Neurol. 2010 Aug 15;518(16):3381-407. doi: 10.1002/cne.22406.
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Altered phase precession and compression of temporal sequences by place cells in epileptic rats.癫痫大鼠中位置细胞对时间序列的相位进动和压缩改变
J Neurosci. 2008 May 7;28(19):5053-62. doi: 10.1523/JNEUROSCI.5024-07.2008.
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The electroconvulsive therapy controversy: evidence and ethics.电休克治疗争议：证据与伦理
Neuropsychol Rev. 2003 Dec;13(4):199-219. doi: 10.1023/b:nerv.0000009484.76564.58.
6
Pathogenesis of brain lesions caused by experimental epilepsy. Light- and electron-microscopic changes in the rat cerebral cortex following bicuculline-induced status epilepticus.实验性癫痫所致脑损伤的发病机制。荷包牡丹碱诱发癫痫持续状态后大鼠大脑皮质的光镜和电镜变化。
Acta Neuropathol. 1981;54(3):219-31. doi: 10.1007/BF00687745.
7
Phenobarbitone dosage in neonatal convulsions.新生儿惊厥时苯巴比妥的剂量
Arch Dis Child. 1982 Sep;57(9):653-7. doi: 10.1136/adc.57.9.653.
8
Degenerative hippocampal pathology in mice infected with scrapie.感染羊瘙痒病的小鼠海马区的退行性病变
Acta Neuropathol. 1984;65(1):62-8. doi: 10.1007/BF00689829.
9
Pathogenesis of brain lesions caused by experimental epilepsy. Light- and electron-microscopic changes in the rat hippocampus following bicuculline-induced status epilepticus.实验性癫痫所致脑损伤的发病机制。荷包牡丹碱诱导癫痫持续状态后大鼠海马的光镜和电镜变化。
Acta Neuropathol. 1983;59(1):11-24. doi: 10.1007/BF00690312.
10
Structural abnormalities in neurones.神经元的结构异常。
J Clin Pathol Suppl (R Coll Pathol). 1977;11:155-69. doi: 10.1136/jcp.s3-11.1.155.