用甲基乙二醛双（脒腙）或溴化乙锭体内处理腹水L1210细胞后线粒体超微结构和功能损伤的相关性 - Suppr

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超能文献

Correlation of ultrastructural and functional damage to mitochondria of ascites L1210 cells treated in vivo with methylglyoxal-bis(guanylhydrazone) or ethidium bromide.

作者信息

Porter C W, Mikles-Robertson F, Kramer D, Dave C

出版信息

Cancer Res. 1979 Jul;39(7 Pt 1):2414-21.

PMID:445440

Abstract

摘要

相似文献

Correlation of ultrastructural and functional damage to mitochondria of ascites L1210 cells treated in vivo with methylglyoxal-bis(guanylhydrazone) or ethidium bromide.用甲基乙二醛双（脒腙）或溴化乙锭体内处理腹水L1210细胞后线粒体超微结构和功能损伤的相关性

Cancer Res. 1979 Jul;39(7 Pt 1):2414-21.

Ultrastructural changes in the mitochondria of intestinal epithelium of rodents treated with methylglyoxal-bis(guanylhydrazone).用甲基乙二醛双（脒腙）处理的啮齿动物肠上皮线粒体的超微结构变化

Cancer Res. 1983 Feb;43(2):646-52.

Morphological evidence for an antimitochondrial action by methylglyoxal-bis(guanylhydrazone).丙酮醛双（脒腙）抗线粒体作用的形态学证据。

Cancer Res. 1977 Jul;37(7 Pt 1):2246-50.

Independence of drug action on mitochondria and polyamines in L1210 leukemia cells treated with methylglyoxal-bis(guanylhydrazone).用甲基乙二醛双（脒腙）处理的L1210白血病细胞中药物作用对线粒体和多胺的独立性

Cancer Res. 1980 Dec;40(12):4533-40.

A selective effect of methylglyoxal-bis(guanylhydrazone) on the synthesis of mitochondrial DNA of cultured L1210 leukemia cells.甲基乙二醛双（脒腙）对培养的L1210白血病细胞线粒体DNA合成的选择性作用。

Cancer Res. 1979 Oct;39(10):4130-7.

Apparent autophagocytosis of mitochondria in L1210 leukemia cells treated in vitro with 4,4'-diacetyl-diphenylurea-bis(guanylhydrazone).

Cancer Res. 1980 Apr;40(4):1054-61.

Carnitine prevents the early mitochondrial damage induced by methylglyoxal bis(guanylhydrazone) in L1210 leukaemia cells.肉碱可预防甲基乙二醛双（胍腙）诱导的L1210白血病细胞早期线粒体损伤。

Biochem J. 1985 Jun 1;228(2):513-6. doi: 10.1042/bj2280513.

The generality of methylglyoxal bis(guanylhydrazone)-induced mitochondrial damage and the dependence of this effect on cell proliferation.甲基乙二醛双（脒腙）诱导的线粒体损伤的普遍性以及这种效应对细胞增殖的依赖性。

Cancer Res. 1979 Jun;39(6 Pt 1):1919-26.

Ethylglyoxal bis(guanylhydrazone) as an inhibitor of polyamine biosynthesis in L1210 leukemia cells.乙二醛双（脒腙）作为L1210白血病细胞中多胺生物合成的抑制剂。

Biochim Biophys Acta. 1984 Apr 16;803(4):331-7. doi: 10.1016/0167-4889(84)90125-3.

Methylglyoxal bis(guanylhydrazone) stimulates the cellular transport system of the polyamines.甲基乙二醛双（脒腙）刺激多胺的细胞转运系统。

FEBS Lett. 1982 Aug 23;145(2):182-6. doi: 10.1016/0014-5793(82)80163-4.

引用本文的文献

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Biochem J. 1981 Jan 15;194(1):79-89. doi: 10.1042/bj1940079.

Changes in mitochondrial structure and function in 9l rat brain tumor cells treated in vitro with alpha-difluoromethylornithine, a polyamine biosynthesis inhibitor.用多胺生物合成抑制剂α-二氟甲基鸟氨酸体外处理9l大鼠脑肿瘤细胞后线粒体结构和功能的变化

Proc Natl Acad Sci U S A. 1983 Feb;80(3):780-4. doi: 10.1073/pnas.80.3.780.

Irreversible inhibition of putrescine-stimulated S-adenosyl-L-methionine decarboxylase by berenil and pentamidine.贝尼尔和喷他脒对腐胺刺激的S-腺苷-L-蛋氨酸脱羧酶的不可逆抑制作用。

Biochem J. 1985 Oct 1;231(1):165-9. doi: 10.1042/bj2310165.

Acetylation of spermidine and methylglyoxal bis(guanylhydrazone) in baby-hamster kidney cells (BHK-21/C13).亚精胺和甲基乙二醛双（脒腙）在幼仓鼠肾细胞（BHK-21/C13）中的乙酰化作用

Biochem J. 1988 Jul 1;253(1):223-7. doi: 10.1042/bj2530223.

Antitumor effect of methylglyoxal bis(3-aminopropylamidinohydrazone), a new inhibitor of S-adenosylmethionine and ornithine decarboxylases, on human erythroid leukemia K562 cells.新型S-腺苷甲硫氨酸和鸟氨酸脱羧酶抑制剂双（3-氨丙基脒腙）甲基乙二醛对人红白血病K562细胞的抗肿瘤作用

Cancer Chemother Pharmacol. 1988;22(3):187-90. doi: 10.1007/BF00273408.

Combined use of alpha-difluoromethylornithine and an inhibitor of S-adenosylmethionine decarboxylase in mice bearing P388 leukemia or Lewis lung carcinoma.α-二氟甲基鸟氨酸与S-腺苷甲硫氨酸脱羧酶抑制剂联合用于携带P388白血病或Lewis肺癌的小鼠。

Jpn J Cancer Res. 1988 Apr;79(4):501-8. doi: 10.1111/j.1349-7006.1988.tb01619.x.

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