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Transsynaptic degeneration 'en cascade' in the cerebellar cortex of staggerer mutant mice.

作者信息

Sotelo C, Changeux J P

出版信息

Brain Res. 1974 Mar 8;67(3):519-26. doi: 10.1016/0006-8993(74)90499-5.

DOI:10.1016/0006-8993(74)90499-5
PMID:4470439
Abstract
摘要

相似文献

1
Transsynaptic degeneration 'en cascade' in the cerebellar cortex of staggerer mutant mice.
Brain Res. 1974 Mar 8;67(3):519-26. doi: 10.1016/0006-8993(74)90499-5.
2
Ultrastructural identification of noradrenergic boutons in mutant and normal mouse cerebellar cortex.
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3
Synaptic remodeling of the cerebellar circuitry in mutant mice and experimental cerebellar malformations. Study "in vivo" and "in vitro".突变小鼠和实验性小脑畸形中小脑回路的突触重塑。“体内”和“体外”研究。
Acta Neuropathol. 1978 Aug 7;43(1-2):19-34. doi: 10.1007/BF00684995.
4
Identification of climbing fibres in the cerebellar cortex of the rat by means of 3H-leucine EM-autoradiography.
Acta Biol Acad Sci Hung. 1974;25(3):217-26.
5
Fine structure of the cerebellum of "staggerer-reeler", a double mutant of mice affected by staggerer and reeler conditions. II. Purkinje cell anomalies.“蹒跚 - 摇晃”小鼠（一种受蹒跚和摇晃病症影响的双突变小鼠）小脑的精细结构。II. 浦肯野细胞异常。
J Neuropathol Exp Neurol. 1977 May;36(3):427-39. doi: 10.1097/00005072-197705000-00002.
6
Electron microscopic analysis of postnatal histogenesis in the cerebellar cortex of staggerer mutant mice.蹒跚突变小鼠小脑皮质出生后组织发生的电子显微镜分析
J Comp Neurol. 1978 Jun 15;179(4):831-63. doi: 10.1002/cne.901790408.
7
Freeze-fracture scanning electron microscopy and comparative freeze-etching study of parallel fiber-Purkinje spine synapses of vertebrate cerebellar cortex.脊椎动物小脑皮质平行纤维 - 浦肯野树突棘突触的冷冻断裂扫描电子显微镜及比较冷冻蚀刻研究
J Submicrosc Cytol Pathol. 1990 Apr;22(2):281-95.
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Dendritic abnormalities of Purkinje cells in the cerebellum of neurologic mutant mice (weaver and staggerer).
Adv Neurol. 1975;12:335-51.
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[Synapses with a different synaptic contact structure in the cerebellar cortex].[小脑皮质中具有不同突触接触结构的突触]
Tsitologiia. 1988 Mar;30(3):342-5.
10
The fine structure of staggerer cerebellum.蹒跚者小脑的精细结构。
J Neuropathol Exp Neurol. 1975 Jan;34(1):1-11. doi: 10.1097/00005072-197501000-00001.

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Loss of RAR-related orphan receptor alpha (RORα) selectively lowers docosahexaenoic acid in developing cerebellum.
视黄酸相关孤儿受体α（RORα）缺失选择性降低发育中小脑的二十二碳六烯酸。
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Single-Cell RNA-seq Reveals Profound Alterations in Mechanosensitive Dorsal Root Ganglion Neurons with Vitamin E Deficiency.单细胞RNA测序揭示维生素E缺乏时机械敏感背根神经节神经元的深刻变化。
iScience. 2019 Nov 22;21:720-735. doi: 10.1016/j.isci.2019.10.064. Epub 2019 Oct 31.
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An innate immune response and altered nuclear receptor activation defines the spinal cord transcriptome during alpha-tocopherol deficiency in Ttpa-null mice.在 Ttpa 基因敲除小鼠的α-生育酚缺乏症期间，固有免疫反应和核受体激活的改变定义了脊髓转录组。
Free Radic Biol Med. 2018 May 20;120:289-302. doi: 10.1016/j.freeradbiomed.2018.02.037. Epub 2018 Mar 9.
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Deletion of the GluRδ2 Receptor in the Hotfoot Mouse Mutant Causes Granule Cell Loss, Delayed Purkinje Cell Death, and Reductions in Purkinje Cell Dendritic Tree Area.热足小鼠突变体中GluRδ2受体的缺失导致颗粒细胞丢失、浦肯野细胞死亡延迟以及浦肯野细胞树突区域减少。
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The role of cerebellar circuitry alterations in the pathophysiology of autism spectrum disorders.小脑神经回路改变在自闭症谱系障碍病理生理学中的作用。
Front Neurosci. 2015 Sep 1;9:296. doi: 10.3389/fnins.2015.00296. eCollection 2015.
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The Spontaneous Ataxic Mouse Mutant Tippy is Characterized by a Novel Purkinje Cell Morphogenesis and Degeneration Phenotype.自发性共济失调小鼠突变体Tippy的特征是一种新型的浦肯野细胞形态发生和退化表型。
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Mature Purkinje cells require the retinoic acid-related orphan receptor-α (RORα) to maintain climbing fiber mono-innervation and other adult characteristics.成熟的浦肯野细胞需要维甲酸相关孤儿受体-α（RORα）来维持 climbing fiber 单神经支配和其他成年特征。
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Motor learning of mice lacking cerebellar Purkinje cells.缺乏小脑浦肯野细胞的小鼠的运动学习。
Front Neuroanat. 2013 Apr 23;7:4. doi: 10.3389/fnana.2013.00004. eCollection 2013.