Zipes D P, Foster P R, Troup P J, Pedersen D H
Am J Cardiol. 1979 Jul;44(1):1-8. doi: 10.1016/0002-9149(79)90242-x.
Atrial stimulation induced a sustained ventricular tachycardia in two patients with mitral valve prolapse and in one patient who had mild hypertension without cardiac abnormalities. Exercise-induced sinus tachycardia also started the ventricular tachycardia in one patient. Evidence is presented to suggest that the mechanism of ventricular tachycardia in one patient was reentrant excitation and in another patient triggered automaticity. It is likely that the origin of the ventricular tachycardia was confined to a relatively protected small area near the posteroinferior portion of the left ventricle and was not due to macroreentry.
心房刺激在两名二尖瓣脱垂患者和一名无心脏异常的轻度高血压患者中诱发了持续性室性心动过速。运动诱发的窦性心动过速在一名患者中也引发了室性心动过速。有证据表明,一名患者室性心动过速的机制是折返激动,另一名患者是触发自律性。室性心动过速的起源可能局限于左心室后下部分附近相对受保护的小区域,而非大折返所致。