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Complement-mediated tissue damage: contribution of the classical and alternate complement pathways in the Forssman reaction.

作者信息

May J E, Frank M M

出版信息

J Immunol. 1972 Jun;108(6):1517-25.

PMID:4537434
Abstract
摘要

相似文献

1
Complement-mediated tissue damage: contribution of the classical and alternate complement pathways in the Forssman reaction.补体介导的组织损伤：经典和替代补体途径在福斯曼反应中的作用。
J Immunol. 1972 Jun;108(6):1517-25.
2
Studies of complement-mediated membrane damage: the influence of erythrocyte storage on susceptibility to cytolysis.补体介导的膜损伤研究：红细胞储存对细胞溶解敏感性的影响。
J Immunol. 1973 Feb;110(2):482-9.
3
Complement: effects of C1 binding affinity on the efficiency of the C2 reaction.补体：C1结合亲和力对C2反应效率的影响
J Immunol. 1972 Dec;109(6):1327-36.
4
The first component of human complement (C1): kinetics of reaction with its natural substrates.人类补体（C1）的首个成分：与其天然底物的反应动力学
J Immunol. 1974 Mar;112(3):905-10.
5
DNP-antigens activate the alternate pathway of the complement system.二硝基苯抗原激活补体系统的替代途径。
J Immunol. 1974 Aug;113(2):501-6.
6
Hemolysis of sheep erythrocytes in guinea pig serum deficient in the fourth component of complement. II. Evidence for involvement of C1 and components of the alternate complement pathway.豚鼠血清中缺乏补体第四成分时绵羊红细胞的溶血作用。II. C1及替代补体途径成分参与的证据
J Immunol. 1973 Dec;111(6):1668-76.
7
Complement and heterophile shock.补体与嗜异性休克。
Johns Hopkins Med J. 1970 Apr;126(4):210-6.
8
Observations on the mechanism of immune hemolysis: importance of immunoglobulin class and source of complement on the extent of damage.免疫溶血机制的观察：免疫球蛋白类别及补体来源对损伤程度的重要性
J Immunol. 1970 Jun;104(6):1502-10.
9
An ultrastructural analysis of the vascular damage in the lethal and sublethal Forssman reaction in the guinea-pig.豚鼠致死性和亚致死性福斯曼反应中血管损伤的超微结构分析。
Br J Exp Pathol. 1985 Dec;66(6):709-18.
10
IgG and complement-mediated tissue damage in the absence of C2: evidence of a functionally active C2-bypass pathway in a guinea pig model.在缺乏C2的情况下IgG和补体介导的组织损伤：豚鼠模型中功能活跃的C2旁路途径的证据
J Immunol. 1999 Sep 15;163(6):3549-58.

引用本文的文献

1
Animal models for complement deficiencies.补体缺陷的动物模型。
J Clin Immunol. 1995 Nov;15(6 Suppl):113S-121S. doi: 10.1007/BF01540901.
2
High-dose intravenous immunoglobulin exerts its beneficial effect in patients with dermatomyositis by blocking endomysial deposition of activated complement fragments.大剂量静脉注射免疫球蛋白通过阻断活化补体片段在肌内膜的沉积，对皮肌炎患者发挥有益作用。
J Clin Invest. 1994 Nov;94(5):1729-35. doi: 10.1172/JCI117520.
3
Proceedings of the British Pharmacological Society. 17th-19th December, 1979: University of London (Institute of Education).
英国药理学会会议记录。1979年12月17日至19日：伦敦大学（教育学院）
Br J Pharmacol. 1980 Jun;69(2):265P-346P.
4
Mechanisms of Forssman-induced bronchospasm and their inhibition.福斯曼抗原诱导支气管痉挛的机制及其抑制作用。
Br J Pharmacol. 1981 May;73(1):25-32. doi: 10.1111/j.1476-5381.1981.tb16767.x.
5
Studies on the in vivo effects of antibody. Interaction of IgM antibody and complement in the immune clearance and destruction of erythrocytes in man.抗体的体内效应研究。人类中IgM抗体与补体在红细胞免疫清除和破坏中的相互作用。
J Clin Invest. 1974 Aug;54(2):339-48. doi: 10.1172/JCI107769.
6
Heterophile antibodies and tissue injury. 3. A role for platelets in the development of lethal vascular injury during Forssman shock in guinea pigs.嗜异性抗体与组织损伤。3. 血小板在豚鼠福斯曼休克致死性血管损伤发展中的作用。
Am J Pathol. 1973 Aug;72(2):179-200.
7
Interactions of the classical and alternate complement pathway with endotoxin lipopolysaccharide. Effect on platelets and blood coagulation.经典补体途径和替代补体途径与内毒素脂多糖的相互作用。对血小板和血液凝固的影响。
J Clin Invest. 1973 Feb;52(2):370-6. doi: 10.1172/JCI107193.
8
A new complement-mediated cytolytic mechanism--the C1-bypass activation pathway.一种新的补体介导的细胞溶解机制——C1旁路激活途径。
Proc Natl Acad Sci U S A. 1973 Mar;70(3):649-52. doi: 10.1073/pnas.70.3.649.
9
An ultrastructural analysis of the vascular damage in the lethal and sublethal Forssman reaction in the guinea-pig.豚鼠致死性和亚致死性福斯曼反应中血管损伤的超微结构分析。
Br J Exp Pathol. 1985 Dec;66(6):709-18.
10
Mechanism of therapeutic effect of high-dose intravenous immunoglobulin. Attenuation of acute, complement-dependent immune damage in a guinea pig model.大剂量静脉注射免疫球蛋白的治疗作用机制。豚鼠模型中急性补体依赖性免疫损伤的减轻。
J Clin Invest. 1989 Dec;84(6):1974-81. doi: 10.1172/JCI114387.