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感染蛙病毒3后宿主细胞核糖核酸聚合酶II的损伤

Impairment of host cell ribonucleic acid polymerase II after infection with frog virus 3.

作者信息

Campadelli-Fiume G, Costanzo F, Mannini-Palenzona A, La Placa M

出版信息

J Virol. 1972 Apr;9(4):698-700. doi: 10.1128/JVI.9.4.698-700.1972.

Abstract

Evidence is presented that, in frog virus 3-infected BHK cells, inhibition of ribonucleic acid (RNA) synthesis is paralleled by a decreased activity of host cell RNA polymerase II, while the template ability of host cell nuclei and chromatin is unaffected.

摘要

有证据表明,在蛙病毒3感染的BHK细胞中,核糖核酸(RNA)合成的抑制与宿主细胞RNA聚合酶II活性的降低同时出现,而宿主细胞核和染色质的模板能力未受影响。

相似文献

2
Selective inhibition of a nuclear RNA polymerase in cells infected with frog virus.
Nature. 1970 Jul 18;227(5255):294-5. doi: 10.1038/227294a0.

本文引用的文献

3
Selective inhibition of a nuclear RNA polymerase in cells infected with frog virus.
Nature. 1970 Jul 18;227(5255):294-5. doi: 10.1038/227294a0.
5
Specific nucleolar and nucleoplasmic RNA polymerases.特定的核仁与核质RNA聚合酶。
Proc Natl Acad Sci U S A. 1970 Mar;65(3):675-82. doi: 10.1073/pnas.65.3.675.
8
Action of rifamycins on RNA polymerase.
Biochim Biophys Acta. 1968 Mar 18;157(1):215-7. doi: 10.1016/0005-2787(68)90285-2.

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