大肠杆菌素E3诱导16S核糖体核糖核酸的切割；某些抗生素的阻断作用。 - Suppr

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超能文献

Colicin E3 induced cleavage of 16S ribosomal ribonucleic acid; blocking effects of certain antibiotics.

作者信息

Dahlberg A E, Lund E, Kjeldgaard N O, Bowman C M, Nomura M

出版信息

Biochemistry. 1973 Feb 27;12(5):948-50. doi: 10.1021/bi00729a025.

DOI:10.1021/bi00729a025

PMID:4568771

Abstract

摘要

相似文献

Colicin E3 induced cleavage of 16S ribosomal ribonucleic acid; blocking effects of certain antibiotics.大肠杆菌素E3诱导16S核糖体核糖核酸的切割；某些抗生素的阻断作用。

Biochemistry. 1973 Feb 27;12(5):948-50. doi: 10.1021/bi00729a025.

Specific inactivation of 16S ribosomal RNA induced by colicin E3 in vivo.体内大肠菌素E3诱导的16S核糖体RNA的特异性失活

Proc Natl Acad Sci U S A. 1971 May;68(5):964-8. doi: 10.1073/pnas.68.5.964.

Effect of colicin E3 upon the 30S ribosomal subunit of Escherichia coli.大肠杆菌素E3对大肠杆菌30S核糖体亚基的作用。

Proc Natl Acad Sci U S A. 1971 May;68(5):959-63. doi: 10.1073/pnas.68.5.959.

Specific inactivation of ribosomes by colicin E3 in vitro and mechanism of immunity in colicinogenic cells.体外大肠杆菌素E3对核糖体的特异性失活作用及产大肠杆菌素细胞中的免疫机制

Nat New Biol. 1971 Dec 1;234(48):133-7. doi: 10.1038/newbio234133a0.

Change in methylation of 16S ribosomal RNA associated with mutation to kasugamycin resistance in Escherichia coli.与大肠杆菌对春雷霉素耐药性突变相关的16S核糖体RNA甲基化变化

Nat New Biol. 1971 Sep 1;233(35):12-4. doi: 10.1038/newbio233012a0.

Ribosome degradation in Escherichia coli induced by colicin E2.大肠杆菌中由大肠杆菌素E2诱导的核糖体降解

Biochem Biophys Res Commun. 1973 Oct 1;54(3):1185-90. doi: 10.1016/0006-291x(73)90817-6.

Colicin E3: a unique endoribonuclease.大肠杆菌素E3：一种独特的核糖核酸内切酶。

Proc Natl Acad Sci U S A. 1973 Jan;70(1):156-60. doi: 10.1073/pnas.70.1.156.

Stability of ribosomal and transfer ribonucleic acid in Escherichia coli B/r after treatment with ethylenedinitrilotetraacetic acid and rifampicin.用乙二胺四乙酸和利福平处理后大肠杆菌B/r中核糖体核糖核酸和转移核糖核酸的稳定性

J Bacteriol. 1975 May;122(2):425-32. doi: 10.1128/jb.122.2.425-432.1975.

Activity of colicin E3-treated ribosomes in initiation and in chain elongation.大肠杆菌素E3处理的核糖体在起始和链延伸中的活性。

Proc Natl Acad Sci U S A. 1974 Apr;71(4):1021-5. doi: 10.1073/pnas.71.4.1021.

Properties of 30S ribosomal particles reconstituted from precursor 16S ribonucleic acid.由前体16S核糖核酸重构的30S核糖体颗粒的特性

Biochemistry. 1974 Mar 12;13(6):1215-21. doi: 10.1021/bi00703a025.

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A structural basis for streptomycin-induced misreading of the genetic code.链霉素诱导遗传密码错读的结构基础。

Nat Commun. 2013;4:1355. doi: 10.1038/ncomms2346.

Colicin E3 cleavage of 16S rRNA impairs decoding and accelerates tRNA translocation on Escherichia coli ribosomes.大肠杆菌素E3对16S rRNA的切割会损害解码过程，并加速tRNA在大肠杆菌核糖体上的易位。

Mol Microbiol. 2008 Jul;69(2):390-401. doi: 10.1111/j.1365-2958.2008.06283.x.

Ribosomal protection proteins and their mechanism of tetracycline resistance.核糖体保护蛋白及其四环素抗性机制。

Antimicrob Agents Chemother. 2003 Dec;47(12):3675-81. doi: 10.1128/AAC.47.12.3675-3681.2003.

Mechanism of Tet(O)-mediated tetracycline resistance.Tet(O)介导的四环素耐药机制。

EMBO J. 2003 Feb 17;22(4):945-53. doi: 10.1093/emboj/cdg093.

Throwing a spanner in the works: antibiotics and the translation apparatus.破坏计划：抗生素与翻译装置

J Mol Med (Berl). 1996 Aug;74(8):423-39. doi: 10.1007/BF00217518.

Relationship between structure of the 5' noncoding region of viral mRNA and efficiency in the initiation step of protein synthesis in a eukaryotic system.病毒mRNA 5'非编码区结构与真核系统中蛋白质合成起始步骤效率之间的关系。

Proc Natl Acad Sci U S A. 1982 Feb;79(4):1012-6. doi: 10.1073/pnas.79.4.1012.

Studies on a factor enhancing colicin E3 activity in vitro.关于一种在体外增强大肠杆菌素E3活性的因子的研究。

Proc Natl Acad Sci U S A. 1974 Oct;71(10):4062-6. doi: 10.1073/pnas.71.10.4062.

Activity of colicin E3-treated ribosomes in initiation and in chain elongation.大肠杆菌素E3处理的核糖体在起始和链延伸中的活性。

Proc Natl Acad Sci U S A. 1974 Apr;71(4):1021-5. doi: 10.1073/pnas.71.4.1021.

How ribosomes select initiator regions in mRNA: base pair formation between the 3' terminus of 16S rRNA and the mRNA during initiation of protein synthesis in Escherichia coli.核糖体如何在mRNA中选择起始区域：大肠杆菌蛋白质合成起始过程中16S rRNA的3'末端与mRNA之间的碱基对形成

Proc Natl Acad Sci U S A. 1975 Dec;72(12):4734-8. doi: 10.1073/pnas.72.12.4734.

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