Drexler H
J Virol. 1973 Nov;12(5):1072-7. doi: 10.1128/JVI.12.5.1072-1077.1973.
A 10- to 15-min derepression of a lambda prophage in a Gal(-) recipient during early infection with a transducing lysate of coliphage T1am will cause an increase in the efficiency of transduction of Gal(+). An increase in the efficiency of transduction occurs when the donor is either nonlysogenic or lysogenic for lambda; the increase is blocked by rifampin or chloramphenicol. With strain R901 it has been shown that efficient transduction can be blocked by treatment with rifampin after all chloramphenicol-sensitive steps have occurred.
在用大肠杆菌噬菌体T1am的转导裂解物进行早期感染期间,在Gal(-)受体中使λ原噬菌体去阻遏10至15分钟,会导致Gal(+)转导效率增加。当供体为非溶原性或对λ为溶原性时,转导效率都会增加;这种增加会被利福平或氯霉素阻断。对于R901菌株,已经表明,在所有氯霉素敏感步骤发生后,用利福平处理可阻断高效转导。