Djamgoz M B, Ruddock K H
Neurosci Lett. 1979 May;12(2-3):329-34. doi: 10.1016/0304-3940(79)96084-1.
Simultaneous recording of the light evoked electrical signals from different classes of slow(S)-potential unit in the isolated fish retina established that picrotoxin, an antagonist of GABA, selectively suppresses the depolarizing component of C-type S-potentials. In contrast, equivalent concentrations of strychnine have no effect on S-potentials, but suppress a component of transient amacrine cell responses. On the basis of these and other experimental data [3,4] we propose a network diagram for the C-type S-potential units in the cyprinid fish retina. This network includes a GABA-ergic feed-back loop and is essentially similar to that proposed by Fuortes and Simon [5].
对离体鱼视网膜中不同类型的慢（S）电位单位的光诱发电信号进行同步记录发现，γ-氨基丁酸（GABA）拮抗剂苦味毒可选择性地抑制C型S电位的去极化成分。相比之下，同等浓度的士的宁对S电位没有影响，但可抑制瞬态无长突细胞反应的一个成分。基于这些及其他实验数据[3,4]，我们提出了鲤科鱼视网膜中C型S电位单位的网络图。该网络包括一个γ-氨基丁酸能反馈回路，本质上与富尔特斯和西蒙[5]提出的网络相似。
