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Mechanisms of cell and tissue injury induced by group A streptococci: relation to poststreptococcal sequelae.

作者信息

Ginsburg I

出版信息

J Infect Dis. 1972 Sep;126(3):294-340. doi: 10.1093/infdis/126.3.294.

DOI:10.1093/infdis/126.3.294
PMID:4627328
Abstract
摘要

相似文献

1
Mechanisms of cell and tissue injury induced by group A streptococci: relation to poststreptococcal sequelae.A组链球菌诱导的细胞和组织损伤机制：与链球菌感染后后遗症的关系。
J Infect Dis. 1972 Sep;126(3):294-340. doi: 10.1093/infdis/126.3.294.
2
Some aspects of basic microbiology of streptococcal disease.链球菌疾病基础微生物学的某些方面。
N Y State J Med. 1968 Jun 1;68(11):1370-80.
3
[Antigens and antibodies of Streptococcus pyogenes in the laboratory and in clinical medicine. Clinico-experimental research on ASO, ASK, ASH and anti-M antibodies].[化脓性链球菌的抗原与抗体在实验室及临床医学中的研究。关于抗链球菌溶血素O、抗链球菌激酶、抗链球菌透明质酸酶及抗M抗体的临床实验研究]
Arch Sci Med (Torino). 1967 Dec;124(6):672-715.
4
Poststreptococcal diseases: pathogenetic aspects of rheumatic fever and acute poststreptococcal glomerulonephritis.链球菌感染后疾病：风湿热和急性链球菌感染后肾小球肾炎的发病机制
Pathobiol Annu. 1978;8:333-57.
5
Some characteristics of nontypable group A streptococci.不可分型A群链球菌的一些特征。
Yale J Biol Med. 1972 Dec;45(6):572-83.
6
[Release of extracellular metabolic products by Streptococci groups C and G].[C组和G组链球菌释放细胞外代谢产物]
Zentralbl Bakteriol Mikrobiol Hyg A Med Mikrobiol Infekt Parasitol. 1982 Oct;253(1):81-7.
7
[Progress in serological detection of hemolytic streptococcal infections].[溶血性链球菌感染血清学检测的进展]
Nihon Rinsho. 1970 Jun;28(6):1662-71.
8
Mechanisms of cell and tissue injury induced by group A streptococci: relation to poststreptococcal sequelae.A组链球菌诱导的细胞和组织损伤机制：与链球菌感染后后遗症的关系。
J Infect Dis. 1972 Oct;126(4):419-56. doi: 10.1093/infdis/126.4.419.
9
[Antibodies to streptozyme and antistreptolysin O in acute glomerulonephritis following streptococcal skin infection].链球菌皮肤感染后急性肾小球肾炎中抗链激酶和抗链球菌溶血素O抗体
Harefuah. 1972 Nov 15;83(10):414-5.
10
The antibody respones in man to infection with different serotypes of group-A streptococci.人类对A组链球菌不同血清型感染的抗体反应。
J Med Microbiol. 1974 Nov;7(4):483-96. doi: 10.1099/00222615-7-4-483.

引用本文的文献

1
Streptolysin S targets the sodium-bicarbonate cotransporter NBCn1 to induce inflammation and cytotoxicity in human keratinocytes during Group A Streptococcal infection.化脓性链球菌溶血素 S 通过靶向钠-碳酸氢盐共转运蛋白 NBCn1 诱导人角质形成细胞在 A 组链球菌感染期间发生炎症和细胞毒性。
Front Cell Infect Microbiol. 2022 Nov 1;12:1002230. doi: 10.3389/fcimb.2022.1002230. eCollection 2022.
2
Signaling Peptide SpoV Is Essential for Virulence, and Prophylaxis with Anti-SpoV Decreases Disease Severity.信号肽SpoV对毒力至关重要，使用抗SpoV进行预防可降低疾病严重程度。
Microorganisms. 2021 Nov 10;9(11):2321. doi: 10.3390/microorganisms9112321.
3
Pro-inflammatory agents released by pathogens, dying host cells, and neutrophils act synergistically to destroy host tissues: a working hypothesis.
病原体、濒死宿主细胞和中性粒细胞释放的促炎因子协同作用以破坏宿主组织：一个工作假说。
J Inflamm Res. 2019 Jan 23;12:35-47. doi: 10.2147/JIR.S190007. eCollection 2019.
4
The effect of leukocyte hydrolases on bacteria VIII. The combined effect of leukocyte extracts, lysozyme, enzyme "cocktails," and penicillin on the lysis ofStaphylococcus aureus and group a streptococci in vitro.白细胞水解酶对细菌的影响 VIII. 白细胞提取物、溶菌酶、酶“鸡尾酒”和青霉素对体外金黄色葡萄球菌和 A 组链球菌溶解的联合作用。
Inflammation. 1976 Dec;1(4):371-407. doi: 10.1007/BF00920338.
5
The effect of leukocyte hydrolases on bacteria : VII. Bactericidal and bacteriolytic reactions mediated by leukocyte and tissue extracts and their modifications by polyelectrolytes.白细胞水解酶对细菌的作用：VII. 白细胞和组织提取物介导的杀菌和溶菌反应及其被聚电解质修饰。
Inflammation. 1976 Jun;1(3):261-84. doi: 10.1007/BF00917867.
6
The effect of leukocyte hydrolases on bacteria : VI. The role played by leukocyte extracts in the sensitization of RBC by lipopolysaccharides and by the cell-sensitizing factor of group A streptococci.白细胞水解酶对细菌的影响：六、白细胞提取物在脂多糖致 RBC 致敏和 A 组链球菌细胞致敏因子中的作用。
Inflammation. 1976 Jun;1(3):247-60. doi: 10.1007/BF00917866.
7
Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae.多药策略对于抑制协同机制导致的感染后后遗症中的组织损伤和器官衰竭是必要的。
Inflammopharmacology. 1999;7(3):207-17. doi: 10.1007/s10787-999-0004-1.
8
Effect of Body Fluids and Macromolecular Substances on the Lysis of Group A Streptococci by Muramidases of Streptomyces albus.白链霉菌 muramidases 对 A 组链球菌裂解作用的体液和大分子物质的影响。
Infect Immun. 1975 Apr;11(4):869-72. doi: 10.1128/iai.11.4.869-872.1975.
9
Cytotoxic effects of streptolysin o and streptolysin s enhance the virulence of poorly encapsulated group a streptococci.链球菌溶血素O和链球菌溶血素S的细胞毒性作用增强了低荚膜A组链球菌的毒力。
Infect Immun. 2003 Jan;71(1):446-55. doi: 10.1128/IAI.71.1.446-455.2003.
10
Hypothesis: is a failure to prevent bacteriolysis and the synergy among microbial and host-derived pro-inflammatory agonists the main contributory factors to the pathogenesis of post-infectious sequelae?假设：无法预防细菌溶解以及微生物和宿主来源的促炎激动剂之间的协同作用是感染后后遗症发病机制的主要促成因素吗？
Inflammation. 2001 Feb;25(1):1-6. doi: 10.1023/a:1007024809661.