Bobbin R P, May J G, Lemoine R L
Arch Otolaryngol. 1979 Aug;105(8):467-70. doi: 10.1001/archotol.1979.00790200029006.
Latency and amplitude intensity functions of two short latency (less than 5 ms) components (I and IV) of the brain stem auditory potentials were recorded from scalp electrodes in 18 paralyzed, hooded rats breathing with the assistance of artificial respirators. Averaged responses were obtained in three groups of rats before and 30 minutes after intraperitoneal injections of saline, ketamine hydrochloride (Ketalar), and pentobarbital sodium. Although both ketamine and pentobarbital significantly decreased heart rate and altered the gross EEG, they did not significantly change the latency or amplitude intensity functions of the two evoked-potential components.
在18只借助人工呼吸机呼吸的麻痹、戴头罩的大鼠中，通过头皮电极记录了脑干听觉电位的两个短潜伏期（小于5毫秒）成分（I和IV）的潜伏期和波幅强度函数。在三组大鼠腹腔注射生理盐水、盐酸氯胺酮（凯他敏）和戊巴比妥钠之前及之后30分钟获得平均反应。尽管氯胺酮和戊巴比妥均显著降低心率并改变了脑电图的总体情况，但它们并未显著改变两个诱发电位成分的潜伏期或波幅强度函数。
