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Duration of the enhanced activity of the microsomal ethanol-oxidizing enzyme system and rate of ethanol degradation in ethanol-fed rats after withdrawal.

作者信息

Mezey E

出版信息

Biochem Pharmacol. 1972 Jan 15;21(2):137-42. doi: 10.1016/0006-2952(72)90263-8.

DOI:10.1016/0006-2952(72)90263-8
PMID:4674908
Abstract
摘要

相似文献

1
Duration of the enhanced activity of the microsomal ethanol-oxidizing enzyme system and rate of ethanol degradation in ethanol-fed rats after withdrawal.撤酒后乙醇喂养大鼠微粒体乙醇氧化酶系统增强活性的持续时间及乙醇降解速率
Biochem Pharmacol. 1972 Jan 15;21(2):137-42. doi: 10.1016/0006-2952(72)90263-8.
2
Microsomal ethanol-oxidizing system (MEOS): purification and properties of a rat liver system free of catalase and alcohol dehydrogenase.微粒体乙醇氧化系统（MEOS）：不含过氧化氢酶和乙醇脱氢酶的大鼠肝脏系统的纯化及特性
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The role of the hepatic microsomal ethanol oxidizing system (MEOS) for ethanol metabolism in vivo.肝脏微粒体乙醇氧化系统（MEOS）在体内乙醇代谢中的作用。
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4
Hepatic microsomal ethanol oxidizing system (MEOS): respective roles of ethanol and carbohydrates for the enhanced activity after chronic alcohol consumption.肝微粒体乙醇氧化系统（MEOS）：慢性饮酒后乙醇和碳水化合物对该系统活性增强的各自作用。
Biochem Pharmacol. 1981 Jul 1;30(13):1745-51. doi: 10.1016/0006-2952(81)90004-6.
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Inhibitory effect of propylthiouracil on the development of metabolic tolerance to ethanol.丙硫氧嘧啶对乙醇代谢耐受性发展的抑制作用。
Biochem Pharmacol. 1985 Jul 1;34(13):2377-83. doi: 10.1016/0006-2952(85)90797-x.
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The combined effects of protein deficiency and chronic ethanol administration on rat ethanol metabolism.蛋白质缺乏和长期给予乙醇对大鼠乙醇代谢的联合影响。
Hepatology. 1986 Sep-Oct;6(5):823-9. doi: 10.1002/hep.1840060504.
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Ethanol oxidation by hepatic microsomes: adaptive increase after ethanol feeding.
Science. 1968 Nov 22;162(3856):917-8. doi: 10.1126/science.162.3856.917.
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Effect of phenobarbital administration on ethanol oxidizing enzymes and on the rates of ethanol degradation.苯巴比妥给药对乙醇氧化酶及乙醇降解速率的影响。
Biochem Pharmacol. 1971 Feb;20(2):508-10. doi: 10.1016/0006-2952(71)90094-3.
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Significance in vivo of the increase in microsomal ethanol-oxidizing system after chronic administration of ethanol, phenobarbital and chlorcyclizine.长期给予乙醇、苯巴比妥和氯环利嗪后微粒体乙醇氧化系统增加在体内的意义。
Biochem Pharmacol. 1972 Aug 15;21(16):2215-26. doi: 10.1016/0006-2952(72)90037-8.
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Increased sensitivity of the microsomal oxidation of ethanol to inhibition by pyrazole and 4-methylpyrazole after chronic ethanol treatment.
Biochem Pharmacol. 1987 Oct 1;36(19):3277-83. doi: 10.1016/0006-2952(87)90645-9.

引用本文的文献

1
Metabolic alterations produced in the liver by chronic ethanol administration. Comparison between the effects produced by ethanol and by thyroid hormones.慢性乙醇给药对肝脏产生的代谢改变。乙醇和甲状腺激素产生的作用的比较。
Biochem J. 1973 Jun;134(2):523-9. doi: 10.1042/bj1340523.
2
Metabolic alterations produced in the liver by chronic ethanol administration. Increased oxidative capacity.慢性乙醇给药对肝脏产生的代谢改变。氧化能力增强。
Biochem J. 1973 Jun;134(2):507-14. doi: 10.1042/bj1340507.
3
Genetic considerations in the effects of ethanol in mice. II. A trans-acting inducibility regulator(s) affecting alcohol dehydrogenase (ADH) activity.
乙醇对小鼠影响中的遗传学考量。II. 一种影响乙醇脱氢酶（ADH）活性的反式作用诱导调节因子。
Biochem Genet. 1984 Aug;22(7-8):597-609. doi: 10.1007/BF00485847.
4
Effects of ethanol on drug metabolism in the liver.乙醇对肝脏药物代谢的影响。
Trans Am Clin Climatol Assoc. 1974;85:175-84.
5
Lipid peroxidation in isolated hepatocytes from rats ingesting ethanol chronically.长期摄入乙醇的大鼠分离肝细胞中的脂质过氧化作用。
Naunyn Schmiedebergs Arch Pharmacol. 1977 Jun;298(2):107-13. doi: 10.1007/BF00508617.