Rugenius Iu Iu, Steponenene R V
Kardiologiia. 1979 Jul;19(7):53-9.
The authors developed a method for recording the electrical activity directly from the human sinus node through the right subclavian vein. Electrical activity of the sinoatrial node (SA) was studied in 179 patients with CHD (angina pectoris, myocardial infarction). The sick-sinus syndrome (SSS) was revealed in 86 patients. The duration of slow deflection Sd, Ss -- P interval (from the onset of Ss to the onset of P wave), P -- A1 interval (from the onset of P wave to the first atrial spike) was prolonged in cases with SSS. The duration of Sd, Ss -- P and P -- A1 decreased after nitroglycerine and atropine administration but increased after benzodixin administration. The data on direct pacing of the SA node and the changes in the duration of phase Sd and Ss after the administration of drugs confirm that slow deflection Sd and rapid preatrial deflection Ss reflect the electrical activity of the SA node.
作者研发出一种通过右锁骨下静脉直接记录人体窦房结电活动的方法。对179例冠心病（心绞痛、心肌梗死）患者的窦房结（SA）电活动进行了研究。86例患者被诊断为病态窦房结综合征（SSS）。在病态窦房结综合征患者中，缓慢偏转Sd、Ss - P间期（从Ss开始到P波开始）、P - A1间期（从P波开始到第一个心房波峰）的时长延长。给予硝酸甘油和阿托品后，Sd、Ss - P和P - A1的时长缩短，但给予苯二氮䓬后时长增加。关于窦房结直接起搏的数据以及给药后Sd和Ss期时长的变化证实，缓慢偏转Sd和快速房前期偏转Ss反映了窦房结的电活动。
