Rugenius Iu, Steponenene R, Zhvironaĭte V
Kardiologiia. 1978 Aug;18(8):108-14.
Seventy-nine patients with ischemic heart disease were examined. Weakness of the sinus node was suspected in 32 patients according to the clinical findings and the ECG; diagnostic atrial stimulation was conducted. Sinoatrial node potential was registered in the form of a spike in patients with ischemic heart disease. Direct stimulation from the region of the sinoatrial node was performed. The stimulator impulse was followed by a distinct potential of the sinoatrial node in the form of a spike which was continuous with the wave of atrial depolarization. The time of sinoatrial conduction (from the onset of the sinoatrial) node potential to the first atrial spike) was determined in 47 patients with ischemic heart disease. It was determined that under the effect of nitroglycerin the time of sinoatrial conduction becomes significantly shorter (reduced from 29 +/- 10.6 to 20 +/- 8.3 msec).
对79例缺血性心脏病患者进行了检查。根据临床表现和心电图，32例患者疑似窦房结功能减弱；进行了诊断性心房刺激。在缺血性心脏病患者中，窦房结电位以尖峰形式记录。对窦房结区域进行了直接刺激。刺激器脉冲之后是窦房结以尖峰形式出现的明显电位，该电位与心房去极化波连续。在47例缺血性心脏病患者中测定了窦房传导时间（从窦房结电位开始到第一个心房尖峰）。结果发现，在硝酸甘油的作用下，窦房传导时间显著缩短（从29±10.6毫秒降至20±8.3毫秒）。
