MacCarthy E P, Frost G W, Strokes G S
Med J Aust. 1979 Jun 16;1(12):550. doi: 10.5694/j.1326-5377.1979.tb127061.x.
A patient with mild chronic renal failure developed significant hyperkalaemia while receiving indomethacin therapy. The hyperkalaemia, which was reversed by discontinuing indomethacin, is attributed to a drug-induced defect in the cellular uptake of potassium.
一名轻度慢性肾衰竭患者在接受吲哚美辛治疗时出现了显著的高钾血症。停用吲哚美辛后高钾血症得到逆转，这归因于药物诱导的钾细胞摄取缺陷。
