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Loss of oxidative phosphorylation in mitochondria isolated from kidneys of mercury poisoned rats.

作者信息

Southard J H, Nitisewojo P

出版信息

Biochem Biophys Res Commun. 1973 Jun 8;52(3):921-7. doi: 10.1016/0006-291x(73)91025-5.

DOI:10.1016/0006-291x(73)91025-5
PMID:4710571
Abstract
摘要

相似文献

1
Loss of oxidative phosphorylation in mitochondria isolated from kidneys of mercury poisoned rats.
Biochem Biophys Res Commun. 1973 Jun 8;52(3):921-7. doi: 10.1016/0006-291x(73)91025-5.
2
[Oxidative phosphorylation in rat kidney mitochondria in mercuric chloride nephrosis].
Vopr Med Khim. 1973 Mar-Apr;19(2):186-91.
3
Mercurial toxicity and the perturbation of the mitochondrial control system.汞中毒与线粒体控制系统的紊乱
Fed Proc. 1974 Oct;33(10):2147-53.
4
Reserpine as an uncoupling agent.利血平作为一种解偶联剂。
Biochim Biophys Acta. 1974 Mar 26;333(3):481-6. doi: 10.1016/0005-2728(74)90132-7.
5
Ribonucleotides during the regeneration of the rat kidney after lesions by folic acid, HgCl2 and ligation of the ureter.大鼠肾脏在遭受叶酸、氯化汞损伤以及输尿管结扎后再生过程中的核糖核苷酸
Naunyn Schmiedebergs Arch Pharmacol. 1974;284(4):383-93. doi: 10.1007/BF00504707.
6
[Hg concentrations in blood and organs of the rat following Hg contamination of the skin].[皮肤汞污染后大鼠血液及器官中的汞浓度]
Arch Exp Veterinarmed. 1980;34(3):425-30.
7
[Disruption of the oxidative phosphorylation process in the heart and kidney mitochondria of rats in vitamin D2 poisoning].[维生素D2中毒大鼠心脏和肾脏线粒体氧化磷酸化过程的破坏]
Vopr Pitan. 1967 Mar-Apr;26(2):38-42.
8
Control of the energy coupling modes in mitochondria by mercurials.汞化合物对线粒体中能量偶联模式的调控
Biochem Biophys Res Commun. 1974 Dec 23;61(4):1310-6. doi: 10.1016/s0006-291x(74)80427-4.
9
[Acute mercury poisoning due to intravenous injection of metallic mercury].静脉注射金属汞导致的急性汞中毒
MMW Munch Med Wochenschr. 1977 Nov 25;119(47):1537-8.
10
Protective behavior of captopril on Hg(++)-induced toxicity on kidney mitochondria. In vivo and in vitro experiments.卡托普利对汞（++）诱导的肾脏线粒体毒性的保护作用。体内和体外实验。
J Pharmacol Exp Ther. 1991 Jan;256(1):385-90.

引用本文的文献

1
A near-infrared fluorescent probe with an improved Stokes shift achieved by tuning the donor-acceptor-donor character of the rhodamine skeleton and its applications.一种通过调节罗丹明骨架的供体-受体-供体特性实现斯托克斯位移改善的近红外荧光探针及其应用。
RSC Adv. 2020 Aug 10;10(49):29536-29542. doi: 10.1039/d0ra04373g. eCollection 2020 Aug 5.
2
Mechanisms of kidney cell injury from metals.金属导致肾细胞损伤的机制。
Environ Health Perspect. 1993 Apr;100:57-63. doi: 10.1289/ehp.9310057.
3
Time-dependent accumulation of inorganic mercury in subcellular fractions of kidney, liver, and brain of rats exposed to methylmercury.
暴露于甲基汞的大鼠肾脏、肝脏和大脑亚细胞组分中无机汞的时间依赖性积累。
Arch Toxicol. 1980 Apr;44(4):231-41. doi: 10.1007/BF00278031.
4
Effects of mercury bichloride on mouse kidney polyribosome structure and function.二氯化汞对小鼠肾脏多核糖体结构和功能的影响。
Arch Toxicol. 1981 Sep;48(2-3):167-72. doi: 10.1007/BF00310485.
5
Ribonucleotides during the regeneration of the rat kidney after lesions by folic acid, HgCl2 and ligation of the ureter.大鼠肾脏在遭受叶酸、氯化汞损伤以及输尿管结扎后再生过程中的核糖核苷酸
Naunyn Schmiedebergs Arch Pharmacol. 1974;284(4):383-93. doi: 10.1007/BF00504707.
6
Regeneration of renal proximal tubules after mercuric chloride injury is accompanied by increased binding of aminoacyl-transfer ribonucleic acid.氯化汞损伤后肾近端小管的再生伴随着氨酰转移核糖核酸结合增加。
Biochem J. 1976 Nov 15;160(2):357-65. doi: 10.1042/bj1600357.