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Variations in the red cells in paroxysmal nocturnal haemoglobinuria.

作者信息

Rosse W F

出版信息

Br J Haematol. 1973 Mar;24(3):327-42. doi: 10.1111/j.1365-2141.1973.tb01657.x.

DOI:10.1111/j.1365-2141.1973.tb01657.x
PMID:4713634
Abstract
摘要

相似文献

1
Variations in the red cells in paroxysmal nocturnal haemoglobinuria.阵发性夜间血红蛋白尿症中红细胞的变异
Br J Haematol. 1973 Mar;24(3):327-42. doi: 10.1111/j.1365-2141.1973.tb01657.x.
2
Erythrocytes of patients with paroxysmal nocturnal haemoglobinuria acquire resistance to complement attack by purified 20-kD homologous restriction factor.阵发性夜间血红蛋白尿患者的红细胞通过纯化的20-kD同源限制因子获得对补体攻击的抗性。
Clin Exp Immunol. 1990 Apr;80(1):109-13. doi: 10.1111/j.1365-2249.1990.tb06449.x.
3
Study of in vitro lysis of paroxysmal nocturnal haemoglobinuria (PNH) and PNH-like red cells.阵发性夜间血红蛋白尿(PNH)及类PNH红细胞的体外溶解研究
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[Simultaneous assessment of the complement-sensitive cells of red cells and reticulocytes in paroxysmal nocturnal hemoglobinuria by complement lysis sensitivity test].[通过补体溶解敏感性试验同步评估阵发性夜间血红蛋白尿症中红细胞和网织红细胞的补体敏感细胞]
Nihon Ketsueki Gakkai Zasshi. 1986 Nov;49(7):1318-24.
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[Relation between the complement sensitive red cells and the frequency of exacerbation of hemoglobinuria in paroxysmal nocturnal hemoglobinuria].阵发性夜间血红蛋白尿中补体敏感红细胞与血红蛋白尿加重频率的关系
Nihon Naika Gakkai Zasshi. 1987 Aug;76(8):1224-9. doi: 10.2169/naika.76.1224.
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The life-span of complement-sensitive and -insensitive red cells in paroxysmal nocturnal hemoglobinuria.阵发性夜间血红蛋白尿中补体敏感和不敏感红细胞的寿命
Blood. 1971 May;37(5):556-62.
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[Changing complement sensitivity and PNH red cell population during the clinical course in a case of PNH with hypoplastic bone marrow].[1例伴有骨髓增生低下的阵发性睡眠性血红蛋白尿症临床病程中补体敏感性及PNH红细胞群体的变化]
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The role of complement in the sensitivity of the paroxysmal nocturnal haemoglobinuria red cell to immune lysis.
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The effect of iron therapy in paroxysmal nocturnal hemoglobinuria.铁疗法对阵发性夜间血红蛋白尿的疗效。
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Paroxysmal nocturnal hemoglobinuria. Hemolysis initiated by the C3 activator system.阵发性夜间血红蛋白尿。溶血由C3激活系统引发。
N Engl J Med. 1972 Jan 27;286(4):180-4. doi: 10.1056/NEJM197201272860403.

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Pharmacological Therapies in Paroxysmal Nocturnal Haemoglobinuria: Focus on Complement Inhibition.
Drugs. 2025 Sep 23. doi: 10.1007/s40265-025-02235-4.
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Paroxysmal Nocturnal Hemoglobinuria: An Underestimated Cause of Pediatric Thromboembolism.阵发性夜间血红蛋白尿症:小儿血栓栓塞症的一个被低估的病因。
TH Open. 2020 Feb 20;4(1):e36-e39. doi: 10.1055/s-0040-1702155. eCollection 2020 Jan.
3
Bone Marrow as a Source of Cells for Paroxysmal Nocturnal Hemoglobinuria Detection.作为用于阵发性夜间血红蛋白尿检测的细胞来源的骨髓
Am J Clin Pathol. 2018 Jul 31;150(3):273-282. doi: 10.1093/ajcp/aqy053.
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Pesg PNH diagnosis, follow-up and treatment guidelines.阵发性睡眠性血红蛋白尿症(PNH)诊断、随访及治疗指南。
Am J Blood Res. 2016 Aug 5;6(2):19-27. eCollection 2016.
5
The pathophysiology of paroxysmal nocturnal hemoglobinuria and treatment with eculizumab.阵发性睡眠性血红蛋白尿症的病理生理学与依库珠单抗治疗。
Ther Clin Risk Manag. 2009;5:911-21. doi: 10.2147/tcrm.s3334.
6
The management of paroxysmal nocturnal hemoglobinuria: recent advances in diagnosis and treatment and new hope for patients.阵发性夜间血红蛋白尿的管理:诊断与治疗的最新进展及给患者带来的新希望
Semin Hematol. 2009 Jan;46(1 Suppl 1):S1-S16. doi: 10.1053/j.seminhematol.2008.11.004.
7
Paroxysmal nocturnal haemoglobinuria: nature's gene therapy?阵发性夜间血红蛋白尿症:大自然的基因疗法?
Mol Pathol. 2002 Jun;55(3):145-52. doi: 10.1136/mp.55.3.145.
8
Specific defect in N-acetylglucosamine incorporation in the biosynthesis of the glycosylphosphatidylinositol anchor in cloned cell lines from patients with paroxysmal nocturnal hemoglobinuria.阵发性夜间血红蛋白尿症患者克隆细胞系中糖基磷脂酰肌醇锚生物合成过程中N-乙酰葡糖胺掺入的特异性缺陷。
Proc Natl Acad Sci U S A. 1993 Jun 1;90(11):5272-6. doi: 10.1073/pnas.90.11.5272.
9
Increased enzymatic activity of the alternative pathway convertase when bound to the erythrocytes of paroxysmal nocturnal hemoglobinuria.当与阵发性夜间血红蛋白尿症的红细胞结合时,替代途径转化酶的酶活性增加。
J Clin Invest. 1982 Feb;69(2):337-46. doi: 10.1172/jci110457.
10
Affected erythrocytes of patients with paroxysmal nocturnal hemoglobinuria are deficient in the complement regulatory protein, decay accelerating factor.阵发性夜间血红蛋白尿患者的受累红细胞缺乏补体调节蛋白衰变加速因子。
Proc Natl Acad Sci U S A. 1983 Aug;80(16):5066-70. doi: 10.1073/pnas.80.16.5066.