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Utilization of the releaxed symphysis pubis of guinea pigs for clues to the mechanism of ovulation.

作者信息

Chihal H J, Espey L L

出版信息

Endocrinology. 1973 Dec;93(6):1441-5. doi: 10.1210/endo-93-6-1441.

DOI:10.1210/endo-93-6-1441
PMID:4752894
Abstract
摘要

相似文献

1
Utilization of the releaxed symphysis pubis of guinea pigs for clues to the mechanism of ovulation.
Endocrinology. 1973 Dec;93(6):1441-5. doi: 10.1210/endo-93-6-1441.
2
The action of relaxin on the pubic symphysis of the guinea-pig, studied electrometrically.用电测法研究松弛素对豚鼠耻骨联合的作用。
J Endocrinol. 1952 Oct;8(4):377-85. doi: 10.1677/joe.0.0080377.
3
[Clomiphene and separation of the symphysis pubis in guinea pigs].[克罗米芬与豚鼠耻骨联合分离]
J Gynecol Obstet Biol Reprod (Paris). 1973 Mar;2(2):129-33.
4
Metabolic responses to relaxin in the mouse symphysis pubis.
Horm Metab Res. 1987 Dec;19(12):669. doi: 10.1055/s-2007-1011907.
5
Relaxin and collagen metabolism.
Horm Metab Res. 1979 Jun;11(6):408-10. doi: 10.1055/s-0028-1092750.
6
Effect of hormones on collagen metabolism and collagenase activity in the pubic symphysis ligament of the guinea pig.激素对豚鼠耻骨联合韧带中胶原代谢及胶原酶活性的影响。
Endocrinology. 1977 Feb;100(2):571-9. doi: 10.1210/endo-100-2-571.
7
[Sexual hormones and relaxation of the symphysis pubis].[性激素与耻骨联合松弛]
Presse Med (1893). 1951 Sep 8;59(57):1159-60.
8
Radiological assessment of relaxin-induced pubic symphyseal changes in guinea pigs: a model for screening disease modifying agents for osteoarthritis.豚鼠中松弛素诱导的耻骨联合变化的放射学评估：一种筛选骨关节炎疾病修饰剂的模型
Inflamm Res. 1995 Aug;44 Suppl 2:S164-5. doi: 10.1007/BF01778313.
9
Relaxin bioactivity and immunoactivity in human corpora lutea.
Endocrinology. 1978 May;102(5):1641-4. doi: 10.1210/endo-102-5-1641.
10
The effect of relaxin on collagen metabolism in the nonpregnant rat pubic symphysis: the influence of estrogen and progesterone in regulating relaxin activity.
Endocrinology. 1996 Sep;137(9):3884-90. doi: 10.1210/endo.137.9.8756561.

引用本文的文献

1
The relaxin family peptide receptor 1 (RXFP1): An emerging player in human health and disease.松弛素家族肽受体 1（RXFP1）：人类健康与疾病中的新兴角色。
Mol Genet Genomic Med. 2020 Apr;8(4):e1194. doi: 10.1002/mgg3.1194. Epub 2020 Feb 26.
2
MicroRNA-144-3p targets relaxin/insulin-like family peptide receptor 1 (RXFP1) expression in lung fibroblasts from patients with idiopathic pulmonary fibrosis.微小 RNA-144-3p 靶向特发性肺纤维化患者肺成纤维细胞中松弛素/胰岛素样家族肽受体 1（RXFP1）的表达。
J Biol Chem. 2019 Mar 29;294(13):5008-5022. doi: 10.1074/jbc.RA118.004910. Epub 2019 Feb 1.
3
Expression of RXFP1 Is Decreased in Idiopathic Pulmonary Fibrosis. Implications for Relaxin-based Therapies.
在特发性肺纤维化中，RXFP1的表达降低。对基于松弛素的治疗的启示。
Am J Respir Crit Care Med. 2016 Dec 1;194(11):1392-1402. doi: 10.1164/rccm.201509-1865OC.
4
Relaxin and its role in the development and treatment of fibrosis.松弛素及其在纤维化发生发展与治疗中的作用。
Transl Res. 2009 Jul;154(1):1-6. doi: 10.1016/j.trsl.2009.03.007. Epub 2009 Apr 22.
5
Guinea-pig interpubic joint (symphysis pubica) relaxation at parturition: underlying cellular processes that resemble an inflammatory response.豚鼠分娩时耻骨联合松弛：类似于炎症反应的潜在细胞过程。
Reprod Biol Endocrinol. 2003 Nov 21;1:113. doi: 10.1186/1477-7827-1-113.
6
Tendon creep is potentiated by NKISK and relaxin which produce collagen fiber sliding.
Iowa Orthop J. 2003;23:75-9.
7
Relaxin inhibits effective collagen deposition by cultured hepatic stellate cells and decreases rat liver fibrosis in vivo.松弛素可抑制培养的肝星状细胞有效沉积胶原蛋白，并在体内减轻大鼠肝纤维化。
Gut. 2001 Oct;49(4):577-83. doi: 10.1136/gut.49.4.577.
8
Relaxin induces an extracellular matrix-degrading phenotype in human lung fibroblasts in vitro and inhibits lung fibrosis in a murine model in vivo.松弛素在体外可诱导人肺成纤维细胞呈现细胞外基质降解表型，并在体内小鼠模型中抑制肺纤维化。
J Clin Invest. 1996 Dec 15;98(12):2739-45. doi: 10.1172/JCI119099.
9
Surgical resections in parous patients with distal ileal and colonic Crohn's disease.经产妇远端回肠和结肠克罗恩病的手术切除
Gut. 1994 Feb;35(2):220-3. doi: 10.1136/gut.35.2.220.
10
Inhibition of relaxin-induced pubic symphyseal "relaxation" in guinea pigs by glycosaminoglycan polysulfates and pentosan polysulfate.糖胺聚糖多硫酸盐和戊聚糖多硫酸盐对豚鼠中松弛素诱导的耻骨联合“松弛”的抑制作用。
Agents Actions. 1994 Aug;42(1-2):74-80. doi: 10.1007/BF02014304.