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Interaction of mixed-function oxidation with biosynthetic processes. 2. Inhibition of lipogenesis by aminopyrine in perfused rat liver.

作者信息

Thurman R G, Scholz R

出版信息

Eur J Biochem. 1973 Sep 21;38(1):73-8. doi: 10.1111/j.1432-1033.1973.tb03035.x.

DOI:10.1111/j.1432-1033.1973.tb03035.x
PMID:4774126
Abstract
摘要

相似文献

1
Interaction of mixed-function oxidation with biosynthetic processes. 2. Inhibition of lipogenesis by aminopyrine in perfused rat liver.混合功能氧化与生物合成过程的相互作用。2. 氨基比林对灌注大鼠肝脏脂肪生成的抑制作用。
Eur J Biochem. 1973 Sep 21;38(1):73-8. doi: 10.1111/j.1432-1033.1973.tb03035.x.
2
Interaction of mixed-function oxidation with biosynthetic processes. 1. Inhibition of gluconeogenesis by aminopyrine in perfused rat liver.
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3
Mixed function oxidation and intermediary metabolism: metabolic interdependencies in the liver.
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4
Alterations in nicotinamide and adenine nucleotide systems during mixed-function oxidation of p-nitroanisole in perfused livers from normal and phenobarbital-treated rats.正常及苯巴比妥处理大鼠灌注肝脏中对硝基苯甲醚混合功能氧化过程中烟酰胺和腺嘌呤核苷酸系统的变化
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5
The effect of cycloheximide on the interaction between mitochondrial respiration and gluconeogenesis in guinea pig and rat liver.放线菌酮对豚鼠和大鼠肝脏线粒体呼吸与糖异生之间相互作用的影响。
J Biol Chem. 1973 Mar 10;248(5):1536-43.
6
Mixed function oxidation in perfused rat liver. The effect of aminopyrine on oxygen uptake.灌注大鼠肝脏中的混合功能氧化。氨基比林对氧摄取的影响。
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7
Action of metyrapone on the redox state of free nicotinamide-adenine dinucleotide and on oxygen consumption of perfused rat livers and isolated mitochondria.甲吡酮对游离烟酰胺腺嘌呤二核苷酸氧化还原状态以及对灌注大鼠肝脏和分离线粒体氧消耗的作用。
Naunyn Schmiedebergs Arch Pharmakol. 1971;268(1):1-16. doi: 10.1007/BF00997168.
8
Effect of 1,3-butanediol on hepatic fatty acid synthesis and metabolite levels in the rat.1,3 - 丁二醇对大鼠肝脏脂肪酸合成及代谢产物水平的影响
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Mitochondrial nicotinamide nucleotide systems: ammonium chloride responses and associated metabolic transitions in hemoglobin-free perfused rat liver.线粒体烟酰胺核苷酸系统：无血红蛋白灌注大鼠肝脏对氯化铵的反应及相关代谢转变
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Activation of ethanol utilization in perfused liver from normal and ethanol-pretreated rats. The effect of hydrogen peroxide generating substrates.正常及经乙醇预处理大鼠灌注肝脏中乙醇利用的激活。过氧化氢生成底物的作用。
Hoppe Seylers Z Physiol Chem. 1974 Mar;355(3):336-40. doi: 10.1515/bchm2.1974.355.1.336.

引用本文的文献

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Glucose regulation of hydroperoxide metabolism in rat intestinal cells. Stimulation of reduced nicotinamide adenine dinucleotide phosphate supply.大鼠肠道细胞中过氧化氢代谢的葡萄糖调节。还原型烟酰胺腺嘌呤二核苷酸磷酸供应的刺激。
J Clin Invest. 1994 Dec;94(6):2426-34. doi: 10.1172/JCI117610.
2
The effects of dietary lipid and phenobarbitone on the production and utilization of NADPH in the liver. A combined biochemical and quantitative cytochemical study.饮食脂质和苯巴比妥对肝脏中烟酰胺腺嘌呤二核苷酸磷酸（NADPH）生成和利用的影响。一项生物化学与定量细胞化学相结合的研究。
Biochem J. 1981 Dec 15;200(3):691-9. doi: 10.1042/bj2000691.
3
NADP-dependent dehydrogenases in rat liver parenchyma. III. The description of a liponeogenic area on the basis of histochemically demonstrated enzyme activities and the neutral fat content during fasting and refeeding.
大鼠肝实质中的NADP依赖性脱氢酶。III. 根据组织化学显示的酶活性及禁食和再喂食期间的中性脂肪含量对一个脂肪生成区域的描述。
Histochemistry. 1981;72(4):579-615. doi: 10.1007/BF00493277.
4
Involvement of glutathione reductase in selenite metabolism and toxicity, studied in isolated rat hepatocytes.谷胱甘肽还原酶在亚硒酸盐代谢及毒性中的作用，以分离的大鼠肝细胞为研究对象。
Arch Toxicol. 1982 Jul;50(2):113-23. doi: 10.1007/BF00373393.
5
Inhibition of fatty acid synthesis and stimulation of glycogen breakdown by vasopressin in the perfused mouse liver.血管加压素对灌注小鼠肝脏中脂肪酸合成的抑制作用及对糖原分解的刺激作用。
Biochem J. 1975 Nov;152(2):389-92. doi: 10.1042/bj1520389.
6
Regulation of p-nitroanisole O-demethylation in perfused rat liver. Adenine nucleotide inhibition of NADP+-dependent dehydrogenases and NADPH-cytochrome c reductase.灌注大鼠肝脏中对硝基苯甲醚O-去甲基化的调节。腺嘌呤核苷酸对NADP⁺依赖性脱氢酶和NADPH-细胞色素c还原酶的抑制作用。
Biochem J. 1979 Dec 15;184(3):675-81. doi: 10.1042/bj1840675.
7
Alterations in nicotinamide and adenine nucleotide systems during mixed-function oxidation of p-nitroanisole in perfused livers from normal and phenobarbital-treated rats.正常及苯巴比妥处理大鼠灌注肝脏中对硝基苯甲醚混合功能氧化过程中烟酰胺和腺嘌呤核苷酸系统的变化
Biochem J. 1977 Sep 15;166(3):583-92. doi: 10.1042/bj1660583.