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Delta-aminolevulinic acid dehydratase activity of erythrocytes and liver tissue in man: relationship to lead exposure.

作者信息

Secchi G C, Erba L, Cambiaghi G

出版信息

Arch Environ Health. 1974 Mar;28(3):130-2. doi: 10.1080/00039896.1974.10666453.

DOI:10.1080/00039896.1974.10666453
PMID:4810883
Abstract
摘要

相似文献

1
Delta-aminolevulinic acid dehydratase activity of erythrocytes and liver tissue in man: relationship to lead exposure.人体红细胞和肝脏组织的δ-氨基乙酰丙酸脱水酶活性：与铅暴露的关系。
Arch Environ Health. 1974 Mar;28(3):130-2. doi: 10.1080/00039896.1974.10666453.
2
Effect of lead on delta-aminolevulinic acid dehydratase activity in red blood cells. II. Regeneration of enzyme after cessation of lead exposure.铅对红细胞中δ-氨基乙酰丙酸脱水酶活性的影响。II. 停止铅暴露后酶的再生。
Arch Environ Health. 1974 Sep;29(3):150-3. doi: 10.1080/00039896.1974.10666553.
3
[Experimental studies on porphyrin metabolism in various heavy metal poisoning. (II). Effect of lead, mercury and arsenic compounds on delta-aminolevulinic dehydratase activity (author's transl)].各种重金属中毒时卟啉代谢的实验研究。（二）铅、汞及砷化合物对δ-氨基-γ-酮戊酸脱水酶活性的影响（作者译）
Nihon Eiseigaku Zasshi. 1975 Jun;30(2):337-45.
4
delta-aminolevulinic acid dehydratase in low level lead exposure.低水平铅暴露中的δ-氨基乙酰丙酸脱水酶
Arch Environ Health. 1976 Jul-Aug;31(4):211-5. doi: 10.1080/00039896.1976.10667221.
5
Delta-aminolevulinate dehydratase, a zinc dependent enzyme.
Biochem Biophys Res Commun. 1974 Oct 23;60(4):1418-24. doi: 10.1016/0006-291x(74)90356-8.
6
Activity of erythrocyte delta-aminolevulinic acid dehydrase and its change by heat treatment as indices of lead exposure.红细胞δ-氨基乙酰丙酸脱水酶活性及其热处理变化作为铅暴露指标
Br J Ind Med. 1976 Feb;33(1):36-42. doi: 10.1136/oem.33.1.36.
7
Zeta-aminolevulinic acid dehydratase test for lead exposure.用于铅暴露检测的δ-氨基乙酰丙酸脱水酶试验
Arch Environ Health. 1974 Nov;29(5):274-81. doi: 10.1080/00039896.1974.10666587.
8
[Blood copper and lead levels and cholinesterase as well as delta-aminolevulinic acid dehydratase activity in erythrocytes in children living in the vicinity of a copper smelter].[生活在铜冶炼厂附近儿童的血铜、血铅水平以及红细胞中的胆碱酯酶和δ-氨基乙酰丙酸脱水酶活性]
Pol Tyg Lek. 1976 May 19;31(16):663-6.
9
[Biological effects of lead. (6). Effect of exposure on pH activity curves of hepatic and erythrocyte gamma-aminolaevulinate dehydratase].
Nihon Eiseigaku Zasshi. 1975 Apr;30(1):110.
10
Relationship between activation of delta-aminolevulinic acid dehydratase by heating and blood lead level.加热对δ-氨基乙酰丙酸脱水酶的激活作用与血铅水平之间的关系。
Arch Toxicol. 1975 Nov 20;34(3):253-8. doi: 10.1007/BF00353288.

引用本文的文献

1
δ-Aminolevulinic acid dehydratase single nucleotide polymorphism 2 (ALAD2) and peptide transporter 2*2 haplotype (hPEPT2*2) differently influence neurobehavior in low-level lead exposed children.δ-氨基乙酰丙酸脱水酶单核苷酸多态性2（ALAD2）和肽转运体2 * 2单倍型（hPEPT2 * 2）对低水平铅暴露儿童的神经行为有不同影响。
Neurotoxicol Teratol. 2015 Jan-Feb;47:137-45. doi: 10.1016/j.ntt.2014.12.001. Epub 2014 Dec 13.
2
δ-Aminolevulinic acid dehydratase single nucleotide polymorphism 2 and peptide transporter 2*2 haplotype may differentially mediate lead exposure in male children.δ-氨基乙酰丙酸脱水酶单核苷酸多态性 2 和肽转运蛋白 2*2 单倍型可能在男性儿童中差异介导铅暴露。
Arch Environ Contam Toxicol. 2011 Oct;61(3):521-9. doi: 10.1007/s00244-011-9645-3. Epub 2011 Feb 16.
3
Polymorphisms of delta-aminolevulinic acid dehydratase (ALAD) and peptide transporter 2 (PEPT2) genes in children with low-level lead exposure.低水平铅暴露儿童 delta-氨基乙酰丙酸脱水酶（ALAD）和肽转运蛋白 2（PEPT2）基因的多态性。
Neurotoxicology. 2009 Nov;30(6):881-7. doi: 10.1016/j.neuro.2009.08.006. Epub 2009 Aug 31.
4
Prenatal exposure to lead, delta-aminolevulinic acid, and schizophrenia: further evidence.产前暴露于铅、δ-氨基乙酰丙酸与精神分裂症：进一步的证据
Environ Health Perspect. 2008 Nov;116(11):1586-90. doi: 10.1289/ehp.10464. Epub 2008 Jul 30.
5
Genetic susceptibility to lead poisoning.铅中毒的遗传易感性。
Environ Health Perspect. 2000 Mar;108 Suppl 1(Suppl 1):23-8. doi: 10.1289/ehp.00108s123.
6
A polymorphism in the delta-aminolevulinic acid dehydratase gene may modify the pharmacokinetics and toxicity of lead.δ-氨基-γ-酮戊酸脱水酶基因的多态性可能会改变铅的药代动力学和毒性。
Environ Health Perspect. 1995 Mar;103(3):248-53. doi: 10.1289/ehp.95103248.
7
The action of small doses of lead on erythrocyte D-aminolevulinic acid dehydratase in the mouse.小剂量铅对小鼠红细胞δ-氨基乙酰丙酸脱水酶的作用
Arch Toxicol. 1980 Jan;43(3):213-20. doi: 10.1007/BF00297586.
8
Persistent protoporphyrinemia in hereditary porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency under low lead exposure. A new molecular basis for the pathogenesis of lead intoxication.低铅暴露下遗传性胆色素原合酶（δ-氨基-γ-酮戊酸脱水酶）缺乏所致的持续性原卟啉血症。铅中毒发病机制的一种新分子基础。
Klin Wochenschr. 1982 Jun 15;60(12):599-606. doi: 10.1007/BF01711435.
9
Acute lead poisoning in inherited porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency.遗传性胆色素原合酶（δ-氨基乙酰丙酸脱水酶）缺乏症中的急性铅中毒
Blut. 1982 Aug;45(2):131-9. doi: 10.1007/BF00319941.
10
Comparison of in vivo effect of inorganic lead and cadmium on glutathione reductase system and delta-aminolevulinate dehydratase in human erythrocytes.无机铅和镉对人红细胞中谷胱甘肽还原酶系统及δ-氨基乙酰丙酸脱水酶的体内效应比较。
Br J Ind Med. 1975 Aug;32(3):181-92. doi: 10.1136/oem.32.3.181.