Letter: Mouse fibroblasts A9 are deficient in HPRT and APRT.
作者信息
Cos R P, Drauss M R, Balis M E, Dancis J
出版信息
Am J Hum Genet. 1974 Mar;26(2):272-3.
Abstract
摘要
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Letter: Mouse fibroblasts A9 are deficient in HPRT and APRT.信件：小鼠成纤维细胞A9缺乏次黄嘌呤磷酸核糖转移酶（HPRT）和腺嘌呤磷酸核糖转移酶（APRT）。
Am J Hum Genet. 1974 Mar;26(2):272-3.
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Absence of demonstrable linkage of human genes for enzymes of the purine and pyrimidine salvage pathways in human-mouse somatic cell hybrids.嘌呤和嘧啶补救途径中人类酶的基因在人-小鼠体细胞杂种中无明显连锁。
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Reexpression of the rat hypoxanthine phosphoribosyltransferase gene in rat-human hybrids.大鼠次黄嘌呤磷酸核糖基转移酶基因在大鼠-人杂种细胞中的重新表达。
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Reappearance of murine hypoxanthine guanine phosphoribosyl transferase activity in mouse A9 cells after attempted hybridisation with human cell lines.在尝试与人类细胞系杂交后，小鼠A9细胞中鼠次黄嘌呤鸟嘌呤磷酸核糖转移酶活性的重现。
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THREE DEGREES OF GUANYLIC ACID--INOSINIC ACID PYROPHOSPHORYLASE DEFICIENCY IN MOUSE FIBROBLASTS.小鼠成纤维细胞中鸟苷酸 - 次黄苷酸焦磷酸化酶缺乏的三个程度
Nature. 1964 Sep 12;203:1142-4. doi: 10.1038/2031142a0.
2
Elevated AMP pyrophosphorylase activity in congenital IMP pyrophosphorylase deficiencey (Lesch-Nyhan disease).先天性次黄嘌呤-鸟嘌呤磷酸核糖转移酶缺乏症（莱施-奈恩病）中腺苷酸焦磷酸化酶活性升高。
J Lab Clin Med. 1969 Nov;74(5):732-41.
3
Communication between normal and enzyme deficient cells in tissue culture.
Exp Cell Res. 1972 Sep;74(1):251-68. doi: 10.1016/0014-4827(72)90503-4.
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