Buchthal F, Rosenfalck A, Trojaborg W
J Neurol Neurosurg Psychiatry. 1974 Mar;37(3):340-60. doi: 10.1136/jnnp.37.3.340.
In 117 consecutive patients with carpal tunnel syndrome and 11 patients with a compression syndrome of the median nerve at elbow, motor and sensory conduction along the median and ulnar nerves and quantitative electromyography were compared with findings in 190 normal controls of the same age. In 25% of patients with carpal tunnel syndrome in whom motor conduction and EMG were normal, the lesion was located from abnormalities in sensory conduction. The fact that conduction along the same fibres was moderately slowed from digit to palm, severely slowed across the flexor retinaculum, and normal from wrist to elbow indicates that slowing was due to demyelination at the site of compression. Fifteen per cent of the patients with carpal tunnel syndrome had clinical and electrophysiological signs of ulnar involvement. In the other patients conduction along the ulnar nerve was as in 100 normal controls. Compression at the elbow was located by electromyographical findings rather than by abnormalities in conduction.
对117例连续的腕管综合征患者和11例正中神经肘部受压综合征患者，将沿正中神经和尺神经的运动及感觉传导以及定量肌电图与190名同年龄正常对照者的检查结果进行了比较。在运动传导和肌电图正常的腕管综合征患者中，25%的病变是由感觉传导异常所致。从手指到手掌同一纤维的传导中度减慢、跨过屈肌支持带时严重减慢而从腕部到肘部传导正常，这一事实表明减慢是由于受压部位的脱髓鞘所致。15%的腕管综合征患者有尺神经受累的临床和电生理体征。在其他患者中，沿尺神经的传导与100名正常对照者相同。肘部受压是通过肌电图检查结果而非传导异常来定位的。
