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类风湿性鹅颈畸形的发病机制。

The pathogenesis of the rheumatoid swan neck deformity.

作者信息

Heywood A W

出版信息

Hand. 1979 Jun;11(2):176-83. doi: 10.1016/s0072-968x(79)80031-5.

Abstract

Disappointment with the late results of intrinsic release for the rheumatoid "intrinsic-plus" hand has led to a re-appraisal of the role of intrinsic muscle contracture in the pathogenesis of the rheumatoid swan neck deformity. In cadaveric fingers, a properly placed suture typing the lateral band to the middle slip insertion causes a swan neck deformity. It is suggested that the usual "intrinsic-plus" hand and the fixed swan neck deformity of rheumatoid arthritis is caused by adhesions between the extensor tendons on the dorsum of the proximal interphalangeal joint, rather than by intrinsic muscle contracture and/or metacarpo-phalangeal dislocation.

摘要

对类风湿性“内在肌加力型”手部进行内在肌松解术的远期效果不理想,这促使人们重新评估内在肌挛缩在类风湿性鹅颈畸形发病机制中的作用。在尸体手指上,将外侧束正确地缝合固定于中节指骨的插入点,会导致鹅颈畸形。有人提出,类风湿性关节炎常见的“内在肌加力型”手部和固定性鹅颈畸形是由近端指间关节背侧伸肌腱之间的粘连引起的,而非内在肌挛缩和/或掌指关节脱位所致。

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