Ultrastructural features in galactosamine-induced hepatitis.

Suppr

超能文献

作者信息

Medline A, Schaffner F, Popper H

出版信息

Exp Mol Pathol. 1970 Apr;12(2):201-11. doi: 10.1016/0014-4800(70)90050-x.

DOI:10.1016/0014-4800(70)90050-x

PMID:4907704

Abstract

摘要

相似文献

Ultrastructural features in galactosamine-induced hepatitis.

Exp Mol Pathol. 1970 Apr;12(2):201-11. doi: 10.1016/0014-4800(70)90050-x.

Bile acid metabolism in mammals. Ultrastructural studies on the intrahepatic cholestasis induced by lithocholic and chenodeoxycholic acids in the rat.哺乳动物的胆汁酸代谢。关于大鼠中石胆酸和鹅去氧胆酸诱导的肝内胆汁淤积的超微结构研究。

Lab Invest. 1971 Apr;24(4):292-302.

[Electron optic studies on galactosamine-induced lesions in the rat liver].[半乳糖胺诱导大鼠肝脏损伤的电子光学研究]

Klin Wochenschr. 1971 Nov 15;49(22):1240-1. doi: 10.1007/BF01732738.

Prednisolone-induced hepatic injury. Ultrastructural and biochemical changes in rabbits.泼尼松龙诱导的肝损伤。家兔的超微结构和生化变化。

Arch Pathol. 1971 Jun;91(6):483-92.

[Galactosamine-hepatitis].

Naturwissenschaften. 1968 Oct;55(10):497. doi: 10.1007/BF00599726.

D-galactosamine and acute liver cell injury.D-半乳糖胺与急性肝细胞损伤

Fed Proc. 1973 Apr;32(4):1516-26.

Pathophysiology of cholestasis.胆汁淤积的病理生理学

Hum Pathol. 1970 Mar;1(1):1-24. doi: 10.1016/s0046-8177(70)80002-8.

[Electron microscopic changes in the rat liver after administration of the oral contraceptive lynestrenol].

Acta Hepatosplenol. 1970 Jan-Feb;17(1):1-10.

Ultrastructural studies of the hepatocytes after chronic exposure to low levels of halothane.

Exp Mol Pathol. 1975 Aug;23(1):35-42. doi: 10.1016/0014-4800(75)90004-0.

Ultrastructural studies on the effect of choline orotate on galactosamine induced hepatic injury in rats.乳清酸胆碱对大鼠半乳糖胺诱导的肝损伤影响的超微结构研究

Exp Mol Pathol. 1972 Feb;16(1):33-46. doi: 10.1016/0014-4800(72)90018-4.

引用本文的文献

"Small Hepatocytes" in the Liver.肝脏中的“小肝细胞”。

Cells. 2023 Nov 27;12(23):2718. doi: 10.3390/cells12232718.

Bone marrow-derived mesenchymal stem cells inhibits hepatocyte apoptosis after acute liver injury.骨髓来源的间充质干细胞可抑制急性肝损伤后肝细胞的凋亡。

Int J Clin Exp Pathol. 2015 Jan 1;8(1):107-16. eCollection 2015.

Serum UPLC-MS/MS metabolic profiling in an experimental model for acute-liver injury reveals potential biomarkers for hepatotoxicity.急性肝损伤实验模型中的血清超高效液相色谱-串联质谱代谢谱分析揭示了肝毒性的潜在生物标志物。

Metabolomics. 2012;8(6):997-1011. doi: 10.1007/s11306-011-0329-9. Epub 2011 Jul 14.

Characterization of growth-differentiation factor 15, a transforming growth factor beta superfamily member induced following liver injury.生长分化因子15的特征，一种肝损伤后诱导产生的转化生长因子β超家族成员。

Mol Cell Biol. 2000 May;20(10):3742-51. doi: 10.1128/MCB.20.10.3742-3751.2000.

Tumor necrosis factor-induced hepatocyte apoptosis precedes liver failure in experimental murine shock models.在实验性小鼠休克模型中，肿瘤坏死因子诱导的肝细胞凋亡先于肝衰竭发生。

Am J Pathol. 1995 May;146(5):1220-34.

Activation, proliferation, and differentiation of progenitor cells into hepatocytes in the D-galactosamine model of liver regeneration.在D-半乳糖胺肝再生模型中祖细胞向肝细胞的激活、增殖和分化。

Am J Pathol. 1993 Dec;143(6):1606-20.

Transcription factor and liver-specific mRNA expression in facultative epithelial progenitor cells of liver and pancreas.转录因子与肝脏和胰腺兼性上皮祖细胞中的肝脏特异性mRNA表达

Am J Pathol. 1995 Dec;147(6):1633-48.

Reversible inhibition of RNA synthesis and irreversible inhibition of protein synthesis by D-galactosamine in isolated mouse hepatocytes.在分离的小鼠肝细胞中，D-半乳糖胺对RNA合成的可逆抑制及对蛋白质合成的不可逆抑制。

Mol Cell Biochem. 1982 Jul 7;46(1):25-30. doi: 10.1007/BF00215578.

Correlation of histology and alpha 1-fetoprotein resurgence in rat liver regeneration after experimental injury by galactosamine.半乳糖胺实验性损伤后大鼠肝脏再生中组织学与甲胎蛋白复发的相关性

Virchows Arch A Pathol Anat Histol. 1980;387(1):47-57. doi: 10.1007/BF00428428.

[Distribution and activity of lysosomal protein catabolic-enzymes of rat liver in galactosamine hepatitis: catepsin D, cathepsin A and acid carboxypeptidase].[半乳糖胺性肝炎大鼠肝脏溶酶体蛋白分解代谢酶（组织蛋白酶D、组织蛋白酶A和酸性羧肽酶）的分布与活性]

Klin Wochenschr. 1971 May 1;49(9):550-4. doi: 10.1007/BF01487993.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验