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Biochemical and metabolic aspects of osteoarthritis.

作者信息

Mankin H J

出版信息

Orthop Clin North Am. 1971 Mar;2(1):19-31.

PMID:4940528
Abstract
摘要

相似文献

1
Biochemical and metabolic aspects of osteoarthritis.骨关节炎的生化和代谢方面
Orthop Clin North Am. 1971 Mar;2(1):19-31.
2
Biochemical and metabolic abnormalities in osteoarthritic human cartilage.骨关节炎患者软骨中的生化和代谢异常
Fed Proc. 1973 Apr;32(4):1478-80.
3
Chondrocyte multiplication in osteoarthritic articular cartilage.骨关节炎关节软骨中的软骨细胞增殖
J Bone Joint Surg Br. 1973 Aug;55(3):588-94.
4
[Pathologic, biochemical and metabolic aspects of osteoarthritis].骨关节炎的病理、生化及代谢方面
Nihon Geka Hokan. 1974 Nov 1;43(6):395-6.
5
[Metabolic processes in normal articular cartilage (age aspect) and in osteoarthrosis (literature review)].[正常关节软骨的代谢过程（年龄方面）及骨关节炎（文献综述）]
Ortop Travmatol Protez. 1982 Mar(3):65-70.
6
Changes in collagen metabolism--another look at osteoarthrosis.胶原蛋白代谢的变化——对骨关节炎的另一种审视
Acta Biol Hung. 1984;35(2-4):181-7.
7
[Regulation of collagen from the cartilage by interleukin-1].
Rev Prat. 1996 Dec 1;46(19 Spec No):S32-6.
8
Biochemical and metabolic abnormalities in articular cartilage from osteo-arthritic human hips.骨关节炎患者髋关节软骨的生化及代谢异常
J Bone Joint Surg Am. 1970 Apr;52(3):424-34.
9
Severe disturbance of the distribution and expression of type VI collagen chains in osteoarthritic articular cartilage.骨关节炎关节软骨中VI型胶原链分布和表达的严重紊乱。
Arthritis Rheum. 1998 Jun;41(6):986-96. doi: 10.1002/1529-0131(199806)41:6<986::AID-ART5>3.0.CO;2-N.
10
The reaction of articular cartilage to injury and osteoarthritis (second of two parts).
N Engl J Med. 1974 Dec 19;291(25):1335-40. doi: 10.1056/NEJM197412192912507.

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1
Pathomorphological Evaluation of the Efficiency of Modeling Osteoarthritis of Vascular-Metabolic Genesis in Female Wistar Rats.雌性Wistar大鼠血管-代谢性骨关节炎建模效率的病理形态学评估
Bull Exp Biol Med. 2025 Aug 29. doi: 10.1007/s10517-025-06472-1.
2
LB-P12 Ameliorates Osteoarthritis by Reducing Cartilage Degradation and Inflammation via Regulation of NF-κB/HIF-2α Pathway.LB-P12通过调节NF-κB/HIF-2α信号通路减少软骨降解和炎症来改善骨关节炎。
J Microbiol Biotechnol. 2025 May 2;35:e2504013. doi: 10.4014/jmb.2504.04013.
3
Therapeutic Controlled Release Strategies for Human Osteoarthritis.
人类骨关节炎的治疗性控释策略
Adv Healthc Mater. 2025 Jan;14(2):e2402737. doi: 10.1002/adhm.202402737. Epub 2024 Nov 6.
4
Excessive load promotes temporomandibular joint chondrocyte apoptosis via Piezo1/endoplasmic reticulum stress pathway.过度负荷通过 Piezo1/内质网应激途径促进颞下颌关节软骨细胞凋亡。
J Cell Mol Med. 2024 Jun;28(11):e18472. doi: 10.1111/jcmm.18472.
5
Thyroid hormone induces ossification and terminal maturation in a preserved OA cartilage biomimetic model.甲状腺激素诱导保存的 OA 软骨仿生模型中的骨化和终末成熟。
Arthritis Res Ther. 2024 Apr 25;26(1):91. doi: 10.1186/s13075-024-03326-5.
6
Infrapatellar fat pad adipose tissue-derived macrophages display a predominant CD11c+CD206+ phenotype and express genotypes attributable to key features of OA pathogenesis.髌下脂肪垫脂肪组织来源的巨噬细胞表现出明显的 CD11c+CD206+表型，并表达与 OA 发病机制关键特征相关的基因型。
Front Immunol. 2024 Feb 1;15:1326953. doi: 10.3389/fimmu.2024.1326953. eCollection 2024.
7
Effects of Diclofenac Etalhyaluronate (SI-613/ONO-5704) on Cartilage Degeneration in Arthritic Rats and Inflammatory Cytokine-Stimulated Human Chondrocytes.双氯芬酸乙磺半胱氨酸盐（SI-613/ONO-5704）对关节炎大鼠软骨退变及炎性细胞因子刺激的人软骨细胞的影响
Cartilage. 2024 Feb 5:19476035231224050. doi: 10.1177/19476035231224050.
8
Better post-operative outcomes at 1-year follow-up are associated with lower levels of pre-operative synovitis and higher levels of IL-6 and VEGFA in unicompartmental knee arthroplasty patients.在单髁膝关节置换术患者中，术前滑膜炎水平较低、IL-6 和 VEGFA 水平较高与术后 1 年随访时更好的手术效果相关。
Knee Surg Sports Traumatol Arthrosc. 2023 Oct;31(10):4109-4116. doi: 10.1007/s00167-023-07503-y. Epub 2023 Jul 14.
9
Protective effects of Pudilan Tablets against osteoarthritis in mice induced by monosodium iodoacetate.蒲苓盆炎康颗粒治疗膝骨关节炎的实验研究
Sci Rep. 2023 Feb 16;13(1):2760. doi: 10.1038/s41598-023-29976-0.
10
Elevated Expression of CCN3 in Articular Cartilage Induces Osteoarthritis in Hip Joints Irrespective of Age and Weight Bearing.CCN3 在关节软骨中的高表达导致髋关节骨关节炎，与年龄和负重无关。
Int J Mol Sci. 2022 Dec 4;23(23):15311. doi: 10.3390/ijms232315311.