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Medical aspects of childhood lead poisoning.

出版信息

HSMHA Health Rep. 1971 Feb;86(2):140-3.

PMID:5101535
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1937102/
Abstract
摘要

相似文献

1
Medical aspects of childhood lead poisoning.儿童铅中毒的医学方面
HSMHA Health Rep. 1971 Feb;86(2):140-3.
2
Preventing childhood lead poisoning.预防儿童铅中毒。
Prev Med. 1994 Sep;23(5):634-7. doi: 10.1006/pmed.1994.1105.
3
Minnesota childhood blood lead guidelines.明尼苏达儿童血铅指南。
Minn Med. 2002 Oct;85(10):44-9.
4
Developing a system to monitor and track childhood blood lead levels. Tennessee Childhood Lead Poisoning Prevention Program.开发一个监测和追踪儿童血铅水平的系统。田纳西州儿童铅中毒预防项目。
Tenn Med. 2002 Oct;95(10):409.
5
Childhood lead poisoning associated with lead dust contamination of family vehicles and child safety seats - Maine, 2008.2008年,缅因州儿童铅中毒与家庭车辆及儿童安全座椅的铅尘污染有关。
MMWR Morb Mortal Wkly Rep. 2009 Aug 21;58(32):890-3.
6
Childhood lead poisoning.
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7
Lead hot zones and childhood lead poisoning cases, Santa Clara County, California, 1995.1995年,加利福尼亚州圣克拉拉县的铅热点区域与儿童铅中毒病例
J Public Health Manag Pract. 1999 Mar;5(2):11-2.
8
Evolution of the management and prevention of childhood lead poisoning: dependence of advances in public health on technological advances in the determination of lead and related biochemical indicators of its toxicity.儿童铅中毒管理与预防的演变:公共卫生进展对铅测定及相关毒性生化指标技术进步的依赖。
Environ Res. 2001 Jun;86(2):111-21. doi: 10.1006/enrs.2001.4260.
9
Reassessment of the microcytic anemia of lead poisoning.铅中毒所致小细胞贫血的重新评估
Pediatrics. 1981 Jun;67(6):904-6.
10
Management of elevated blood lead in Tennessee's children: questions a physician might ask.田纳西州儿童血铅水平升高的管理:医生可能会问的问题
Tenn Med. 2002 Oct;95(10):414-7.

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Lead Acetate Exposure and Cerebral Amyloid Accumulation: Mechanistic Evaluations in APP/PS1 Mice.醋酸铅暴露与脑淀粉样蛋白沉积:APP/PS1 小鼠的机制评估。
Environ Health Perspect. 2024 Oct;132(10):107004. doi: 10.1289/EHP14384. Epub 2024 Oct 16.
2
Do Low Levels of Blood Lead Reduce Children's Future Test Scores?低血铅水平会降低儿童未来的考试成绩吗?
Am Econ J Appl Econ. 2018 Jan;10(1):307-341. doi: 10.1257/app.20160404.
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Control of Lead Sources in the United States, 1970-2017: Public Health Progress and Current Challenges to Eliminating Lead Exposure.美国 1970-2017 年的铅污染源控制:消除铅暴露的公共卫生进展和当前挑战。
J Public Health Manag Pract. 2019 Jan/Feb;25 Suppl 1, Lead Poisoning Prevention(Suppl 1 LEAD POISONING PREVENTION):S13-S22. doi: 10.1097/PHH.0000000000000889.
4
Longitudinal changes in blood lead level in children and their relationship to season, age, and exposure to paint or plaster.儿童血铅水平的纵向变化及其与季节、年龄以及接触油漆或石膏的关系。
Am J Public Health. 1979 Apr;69(4):348-52. doi: 10.2105/ajph.69.4.348.

本文引用的文献

1
LEAD POISONING IN CHILDREN.儿童铅中毒
Arch Dis Child. 1964 Feb;39(203):1-13. doi: 10.1136/adc.39.203.1.
2
Studies on urinary excretion of 5-aminolaevulic acid and other haem precursors in lead workers and lead-intoxicated rabbits.铅作业工人和铅中毒家兔尿中5-氨基乙酰丙酸及其他血红素前体排泄情况的研究。
Scand J Clin Lab Invest. 1960;12 Suppl 47:1-128.
3
A reliable qualitative urine coproporphyrin test for lead intoxication in young children.一种用于检测幼儿铅中毒的可靠的定性尿粪卟啉试验。
J Pediatr. 1960 Jun;56:759-67. doi: 10.1016/s0022-3476(60)80312-5.
4
The concentration of lead in plasma, whole blood and erythrocytes of infants and children.
Pediatrics. 1958 May;21(5):793-7.
5
Quantitative urinary coproporphyrin excretion and its relation to edathamil calcium disodium administration in children with acute lead intoxication.急性铅中毒患儿尿中粪卟啉定量排泄及其与依地酸钙钠给药的关系。
J Clin Invest. 1956 Oct;35(10):1131-8. doi: 10.1172/JCI103367.
6
Coproporphyrin studies in children. I. Urinary coproporphyrin excretion in normal children.儿童粪卟啉研究。I. 正常儿童尿粪卟啉排泄情况
Proc Soc Exp Biol Med. 1954 Jan;85(1):86-8. doi: 10.3181/00379727-85-20794.
7
The excretion of delta-aminolaevulinic acid by children.
Acta Paediatr Scand. 1967 May;56(3):265-8. doi: 10.1111/j.1651-2227.1967.tb15376.x.
8
Serum delta-aminolevulinic acid in plumbism.
J Pediatr. 1969 Jun;74(6):917-23. doi: 10.1016/s0022-3476(69)80226-x.
9
Incidence of high blood lead levels in Chicago children.芝加哥儿童高血铅水平的发病率。
Pediatrics. 1969 Nov;44(5):661-7.
10
Chronic lead intoxication in children.儿童慢性铅中毒
Dev Med Child Neurol. 1965 Oct;7(5):529-36. doi: 10.1111/j.1469-8749.1965.tb10961.x.