睾酮与雄激素受体结合在5α-还原酶缺乏症患者男性性分化中的作用。 - Suppr

Suppr

超能文献

Role of testosterone binding to the androgen receptor in male sexual differentiation of patients with 5 alpha-reductase deficiency.

作者信息

Maes M, Sultan C, Zerhouni N, Rothwell S W, Migeon C J

出版信息

J Steroid Biochem. 1979 Oct;11(4):1385-92. doi: 10.1016/0022-4731(79)90110-9.

DOI:10.1016/0022-4731(79)90110-9

PMID:513759

Abstract

摘要

相似文献

Role of testosterone binding to the androgen receptor in male sexual differentiation of patients with 5 alpha-reductase deficiency.睾酮与雄激素受体结合在5α-还原酶缺乏症患者男性性分化中的作用。

J Steroid Biochem. 1979 Oct;11(4):1385-92. doi: 10.1016/0022-4731(79)90110-9.

Possible mechanisms of androgen resistance in 5 alpha-reductase deficiency: implications for the physiological roles of 5 alpha-reductases.5α-还原酶缺乏症中雄激素抵抗的可能机制：对5α-还原酶生理作用的启示

J Steroid Biochem. 1983 Jul;19(1B):555-9. doi: 10.1016/0022-4731(83)90217-0.

Comparison of the metabolism and receptor binding of testosterone and 17 beta-hydroxy-5 alpha-androstan-3-one in normal skin fibroblast cultures: influence of origin and passage number.正常皮肤成纤维细胞培养物中睾酮与17β-羟基-5α-雄甾烷-3-酮的代谢及受体结合比较：起源和传代数的影响

J Clin Endocrinol Metab. 1979 Jun;48(6):924-30. doi: 10.1210/jcem-48-6-924.

High dose androgen therapy in male pseudohermaphroditism due to 5 alpha-reductase deficiency and disorders of the androgen receptor.高剂量雄激素疗法治疗因5α-还原酶缺乏和雄激素受体异常所致的男性假两性畸形。

J Clin Invest. 1984 Oct;74(4):1496-508. doi: 10.1172/JCI111563.

Incomplete androgen insensitivity syndrome: partially masculinized genitalia in two patients with absence of androgen receptor in cultured fibroblasts.不完全雄激素不敏感综合征：两名培养的成纤维细胞中缺乏雄激素受体患者的生殖器部分男性化。

Clin Endocrinol (Oxf). 1983 Nov;19(5):565-74. doi: 10.1111/j.1365-2265.1983.tb00032.x.

5-Alpha reductase deficiency: a 40-year retrospective review.5α还原酶缺乏症：40年回顾性研究

Curr Opin Endocrinol Diabetes Obes. 2014 Dec;21(6):483-7. doi: 10.1097/MED.0000000000000116.

Cultured human skin fibroblasts: a model for the study of androgen action.培养的人皮肤成纤维细胞：雄激素作用研究的模型。

Mol Cell Biochem. 1981 Apr 13;36(1):3-22. doi: 10.1007/BF02354827.

Human minimal androgen insensitivity with normal dihydrotestosterone-binding capacity in cultured genital skin fibroblasts: evidence for an androgen-selective qualitative abnormality of the receptor.培养的生殖器皮肤成纤维细胞中具有正常二氢睾酮结合能力的人类最小雄激素不敏感：雄激素受体选择性定性异常的证据。

Am J Hum Genet. 1984 Sep;36(5):965-78.

Different aspects of 5 alpha-reductase deficiency in male pseudohermaphroditism and hypothyroidism.男性假两性畸形和甲状腺功能减退中5α-还原酶缺乏的不同方面。

Clin Endocrinol (Oxf). 1981 May;14(5):459-69. doi: 10.1111/j.1365-2265.1981.tb00635.x.

5 alpha-reductase deficiency in humans: support to the theory that 5 alpha-reduction of testosterone is an essential step in the control of LH secretion.人类5α-还原酶缺乏症：支持睾酮5α-还原是控制促黄体生成素分泌的关键步骤这一理论。

J Endocrinol Invest. 1979 Oct-Dec;2(4):463-4. doi: 10.1007/BF03349351.

引用本文的文献

Genetic Analysis of 25 Patients with 5-Reductase Deficiency in Chinese Population.中国人群中 25 例 5-还原酶缺陷症患者的遗传分析。

Biomed Res Int. 2020 Jun 9;2020:1789514. doi: 10.1155/2020/1789514. eCollection 2020.

Modulation of androgen receptor activation function 2 by testosterone and dihydrotestosterone.睾酮和双氢睾酮对雄激素受体激活功能2的调节作用。

J Biol Chem. 2007 Aug 31;282(35):25801-16. doi: 10.1074/jbc.M703268200. Epub 2007 Jun 25.

The XY female in sport: the controversy continues.体育界的XY女性：争议仍在继续。

Br J Sports Med. 1993 Sep;27(3):150-6. doi: 10.1136/bjsm.27.3.150.

Cultured human skin fibroblasts: a model for the study of androgen action.培养的人皮肤成纤维细胞：雄激素作用研究的模型。

Mol Cell Biochem. 1981 Apr 13;36(1):3-22. doi: 10.1007/BF02354827.

A mutation that causes lability of the androgen receptor under conditions that normally promote transformation to the DNA-binding state.一种在通常促进向DNA结合状态转变的条件下导致雄激素受体不稳定的突变。

J Clin Invest. 1984 Apr;73(4):1095-104. doi: 10.1172/JCI111295.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验