On the pathogenesis of favism.
作者信息
Sartori E
出版信息
J Med Genet. 1971 Dec;8(4):462-7. doi: 10.1136/jmg.8.4.462.
Abstract
摘要
相似文献
1
On the pathogenesis of favism.论蚕豆病的发病机制。
J Med Genet. 1971 Dec;8(4):462-7. doi: 10.1136/jmg.8.4.462.
2
Favism in GdMediterranean heterozygous females.地中海贫血杂合子女性中的蚕豆病
Pediatr Res. 1979 Jul;13(7):812-6. doi: 10.1203/00006450-197907000-00004.
3
Favism: current problems and investigations.蚕豆病：当前问题与研究
J Med Genet. 1973 Jun;10(2):154-7. doi: 10.1136/jmg.10.2.154.
4
[Favism in Polish families].[波兰家庭中的蚕豆病]
Pol Tyg Lek. 1990;45(38-39):778-81.
5
6
Hemolytic crises of favism in Sicilian females heterozygous for G-6-PD deficiency.患有G-6-PD缺乏症的西西里岛杂合子女性的蚕豆病溶血危象。
Pediatrics. 1972 Jun;49(6):854-9.
7
[Favism - a familial enzymatic defect of the red blood cells in a 7-year-old girl].[蚕豆病——一名7岁女孩红细胞的家族性酶缺陷]
Pediatr Pol. 1982;57(9):731-4.
8
[Favism: hemolytic crisis due to glucose-6-phosphate dehydrogenase deficiency in erythrocytes].[蚕豆病：红细胞葡萄糖-6-磷酸脱氢酶缺乏所致的溶血性危机]
Pol Tyg Lek. 1973 May 14;28(20):744-5.
9
Favism in a Portuguese family due to a deficient glucose-phosphate dehydrogenase variant (Canton) or (cpanton-like) type.一个葡萄牙家庭中因葡萄糖-6-磷酸脱氢酶变异体（广东型）或（类广东型）缺乏而导致的蚕豆病。
Acta Haematol. 1976;56(1):58-64. doi: 10.1159/000207919.
10
The ratio between normal and sensitive erythrocytes in heterozygous glucose-6-phosphate dehydrogenase deficient women.杂合子葡萄糖-6-磷酸脱氢酶缺乏女性中正常红细胞与敏感红细胞的比例。
Acta Haematol. 1970;43(5):291-5. doi: 10.1159/000208740.
引用本文的文献
1
Intestinal transcriptional profiling reveals fava bean-induced immune response in DBA/1 mice.肠道转录组谱分析揭示了豇豆诱导 DBA/1 小鼠的免疫反应。
Biol Res. 2019 Mar 1;52(1):9. doi: 10.1186/s40659-019-0216-9.
2
Glucose-6-phosphate dehydrogenase deficiency.葡萄糖-6-磷酸脱氢酶缺乏症
Postgrad Med J. 1994 Dec;70(830):871-7. doi: 10.1136/pgmj.70.830.871.
3
Favism: current problems and investigations.蚕豆病：当前问题与研究
J Med Genet. 1973 Jun;10(2):154-7. doi: 10.1136/jmg.10.2.154.
本文引用的文献
1
[Favism; geographic and racial factors in the pathogenesis of the disease, with special reference to favism in Sardinia].[蚕豆病；疾病发病机制中的地理和种族因素，特别提及撒丁岛的蚕豆病]
Rass Med Sarda. 1951 May-Jun;2(5-6):89-131.
2
THE MECHANISM OF HAEMOLYSIS IN FAVISM. SOME ANALOGY IN THE ACTIVITY OF PRIMAQUINE AND FAVA JUICE.蚕豆病中溶血的机制。伯氨喹和蚕豆汁活性的某些相似之处。
Acta Haematol. 1965 Mar;33:129-38. doi: 10.1159/000209522.
3
Primaquine sensitivity. Glucose-6-phosphate dehydrogenase deficiency: an inborn error of metabolism of medical and biological significance.伯氨喹敏感性。葡萄糖-6-磷酸脱氢酶缺乏症：一种具有医学和生物学意义的先天性代谢缺陷。
Arch Intern Med. 1962 Feb;109:209-34. doi: 10.1001/archinte.1962.03620140081013.
4
[The in vitro resistance of the reduced glutathione in erythrocytes to fresh bean juice; preliminary note].[红细胞中还原型谷胱甘肽对鲜豆汁的体外抗性；初步报告]
Boll Soc Ital Biol Sper. 1959 Feb 15;35(3):146-8.
5
[New aspects of the biochemical alterations in the erythrocytes of patients with favism; almost complete absence of glucose-6-phosphate dehydrogenase].蚕豆病患者红细胞生化改变的新方面；葡萄糖-6-磷酸脱氢酶几乎完全缺乏
Boll Soc Ital Biol Sper. 1958 Apr 15;34(7):327-9.
6
[Instability of blood glutathione in favism; utilization of a selective test and introduction of the genetic problem].[蚕豆病中血液谷胱甘肽的不稳定性；选择性检测方法的应用及遗传问题的引入]
Boll Soc Ital Biol Sper. 1957 Jul;33(7):1057-60.
7
[Early determinations of serum glutathione in favism].[蚕豆病血清谷胱甘肽的早期测定]
Boll Soc Ital Biol Sper. 1956 Jun;32(6):456-8.
8
[Contribution to the study of favism].[对蚕豆病研究的贡献]
Clin Pediatr (Bologna). 1956 Jul;38(7):507-36.
9
On the familial predisposition to favism.关于蚕豆病的家族易感性。
Am J Hum Genet. 1966 May;18(3):253-63.
10
Bimodal distribution of erythrocytes in heterozygotes for strong Mediterranean glucose-6-phosphate dehydrogenase deficiency.重度地中海型葡萄糖-6-磷酸脱氢酶缺乏症杂合子红细胞的双峰分布。
J Med Genet. 1966 Mar;3(1):42-6. doi: 10.1136/jmg.3.1.42.
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