Inhibition of hypoxia-induced rise in adenosine release and flow by coronary dilators.
作者信息
Kukovetz W R, Pöch G
出版信息
Cardiology. 1971;56(1):107-13. doi: 10.1159/000169350.
PMID:5152736
Abstract
摘要
相似文献
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Inhibition of hypoxia-induced rise in adenosine release and flow by coronary dilators.
Cardiology. 1971;56(1):107-13. doi: 10.1159/000169350.
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Effect of dipyridamole on myocardial adenosine metabolism and coronary flow in hypoxia and reactive hyperemia in the isolated perfused guinea pig heart.双嘧达莫对离体灌注豚鼠心脏在缺氧及反应性充血状态下心肌腺苷代谢和冠状动脉血流的影响。
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[Physiology of the coronary circulation as a basis for evaluating coronary dilators].[作为评估冠状动脉扩张剂基础的冠状动脉循环生理学]
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[Uptake and liberation of 3H-adenosine in the heart during O2 deficiency, increased perfusion pressure and use of coronary dilators].[缺氧、灌注压升高及使用冠状动脉扩张剂时心脏对³H - 腺苷的摄取与释放]
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The potentiation of the action of adenosine on the guinea-pig heart.腺苷对豚鼠心脏作用的增强效应。
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Coronary responses to dilating substances and competitive inhibition by theophylline in the isolated perfused guinea pig heart.离体灌注豚鼠心脏对舒张物质的冠脉反应及茶碱的竞争性抑制作用。
Pflugers Arch. 1975 Jul 28;358(3):213-24. doi: 10.1007/BF00587218.
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Interstitial transudate concentration of adenosine and inosine in rat and guinea pig hearts.大鼠和豚鼠心脏中腺苷和肌苷的间质渗出液浓度。
Am J Physiol. 1988 Jun;254(6 Pt 2):H1125-32. doi: 10.1152/ajpheart.1988.254.6.H1125.
引用本文的文献
1
How does dipyridamole elevate extracellular adenosine concentration? Predictions from a three-compartment model of adenosine formation and inactivation.双嘧达莫是如何提高细胞外腺苷浓度的？腺苷生成与失活三室模型的预测。
Biochem J. 1986 Aug 1;237(3):845-51. doi: 10.1042/bj2370845.
2
Relationship between coronary flow and adenosine release during severe and mild hypoxia in the isolated perfused rat heart with special reference to time-course change.在离体灌注大鼠心脏中，重度和轻度缺氧期间冠状动脉血流与腺苷释放之间的关系，特别关注时间进程变化。
Heart Vessels. 1987;3(3):113-21. doi: 10.1007/BF02058786.
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Adenosine formation and release from neonatal-rat heart cells in culture.新生大鼠培养心肌细胞中腺苷的形成与释放
Biochem J. 1985 Aug 1;229(3):799-805. doi: 10.1042/bj2290799.
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