Anand A
Respir Physiol. 1979 Sep;38(1):59-69. doi: 10.1016/0034-5687(79)90006-9.
Impulses were recorded in single afferent fibres of aortic chemoreceptors of cats anaesthetized with chloralose. As expected raising the blood pressure (BP) passively by occluding the abdominal aorta, consistently reduced or abolished the activity of the chemoreceptors. This reduction persisted for as long as the BP remained high. Stimulating the sensory receptors of the small intestines increased the activity of 10 aortic chemoreceptors. The increased activity persisted after bilateral adrenalectomy but it was abolished by blocking the nerves of the mesentery with xylocaine. The increased activity was noteworthy because it occurred in spite of the rise in blood pressure (that always occurred when the intestines were squeezed) which by itself would tend to reduce the activity of the chemoreceptors. It was concluded that stimulating the intestinal receptors produces a reflex increase in the activity of aortic chemoreceptors by causing a reduction of glomeral blood flow through impulses in the sympathetic fibres.
在使用水合氯醛麻醉的猫的主动脉化学感受器的单根传入纤维中记录冲动。正如预期的那样，通过阻断腹主动脉被动升高血压（BP），会持续降低或消除化学感受器的活性。只要血压保持高位，这种降低就会持续。刺激小肠的感觉感受器会增加10个主动脉化学感受器的活性。双侧肾上腺切除术后，增加的活性仍然存在，但用利多卡因阻断肠系膜神经后，该活性被消除。增加的活性值得注意，因为尽管血压升高（挤压肠道时总是会出现），而血压升高本身会倾向于降低化学感受器的活性，但这种增加的活性仍然出现。得出的结论是，刺激肠道感受器通过交感纤维中的冲动导致肾小球血流量减少，从而使主动脉化学感受器的活性产生反射性增加。
