3型糖原贮积病的研究：人红细胞中淀粉-1,6-葡萄糖苷酶的动力学 - Suppr

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Studies in glycogen storage disease type 3: kinetics of amylo-1,6-glucosidase in human erythrocytes.

作者信息

Garatun-Tjeldstö E, Garatun-Tjeldstö O

出版信息

Scand J Clin Lab Invest. 1971 May;27(3):269-72. doi: 10.3109/00365517109080218.

DOI:10.3109/00365517109080218

PMID:5281102

Abstract

摘要

相似文献

Studies in glycogen storage disease type 3: kinetics of amylo-1,6-glucosidase in human erythrocytes.

Scand J Clin Lab Invest. 1971 May;27(3):269-72. doi: 10.3109/00365517109080218.

Amylo-1,6-glucosidase in human fibroblasts: studies 3 glycogen storage disease.

Biochem Biophys Res Commun. 1970 May 11;39(3):301-6. doi: 10.1016/0006-291x(70)90576-0.

Amylo-1,6-glucosidase activity and glycogen content of the erythrocytes of normal subjects, patients with glycogen storage disease and heterozygotes.

Eur J Biochem. 1967 Oct;2(3):271-4. doi: 10.1111/j.1432-1033.1967.tb00135.x.

Type 3 glycogenosis: atypical enzyme activities in blood cells in two siblings.

J Pediatr. 1972 Nov;81(5):955-61. doi: 10.1016/s0022-3476(72)80549-3.

A rapid micro assay method for amylo-1,6-glucosidase.

Anal Biochem. 1970 Jan;33(1):87-101. doi: 10.1016/0003-2697(70)90442-2.

Glucose and glycogen metabolism in erythrocytes from normal and glycogen storage disease type III subjects.

J Clin Invest. 1968 Jun;47(6):1343-8. doi: 10.1172/JCI105826.

Heredity of leukocyte phosphorylase and amylo-1, 6-glucosidase deficiency.

J Pediatr. 1969 Jan;74(1):90-4. doi: 10.1016/s0022-3476(69)80012-0.

Amylo-1,60glucosidase deficiency (glycogenosis type III) in the Faroe Islands.

Hum Hered. 1975;25(2):115-26. doi: 10.1159/000152716.

Absence of acid maltase in glycogenesis type 2(Pompe's disease) in tissue culture.

Am J Dis Child. 1969 Jan;117(1):108-11. doi: 10.1001/archpedi.1969.02100030110013.

[Glycogenosis caused by amylo-1,6-glucosidase deficiency. Myopathy as a lead finding in adults].

Schweiz Med Wochenschr. 1981 Jun 6;111(23):854-8.

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Studies in glycogen storage disease type 3: kinetics of amylo-1,6-glucosidase in human erythrocytes.

作者信息

Garatun-Tjeldstö E, Garatun-Tjeldstö O

出版信息

Scand J Clin Lab Invest. 1971 May;27(3):269-72. doi: 10.3109/00365517109080218.

DOI:10.3109/00365517109080218

PMID:5281102

Abstract

摘要

相似文献

Studies in glycogen storage disease type 3: kinetics of amylo-1,6-glucosidase in human erythrocytes.

Scand J Clin Lab Invest. 1971 May;27(3):269-72. doi: 10.3109/00365517109080218.

Amylo-1,6-glucosidase in human fibroblasts: studies 3 glycogen storage disease.

Biochem Biophys Res Commun. 1970 May 11;39(3):301-6. doi: 10.1016/0006-291x(70)90576-0.

Amylo-1,6-glucosidase activity and glycogen content of the erythrocytes of normal subjects, patients with glycogen storage disease and heterozygotes.

Eur J Biochem. 1967 Oct;2(3):271-4. doi: 10.1111/j.1432-1033.1967.tb00135.x.

Type 3 glycogenosis: atypical enzyme activities in blood cells in two siblings.

J Pediatr. 1972 Nov;81(5):955-61. doi: 10.1016/s0022-3476(72)80549-3.

A rapid micro assay method for amylo-1,6-glucosidase.

Anal Biochem. 1970 Jan;33(1):87-101. doi: 10.1016/0003-2697(70)90442-2.

Glucose and glycogen metabolism in erythrocytes from normal and glycogen storage disease type III subjects.

J Clin Invest. 1968 Jun;47(6):1343-8. doi: 10.1172/JCI105826.

Heredity of leukocyte phosphorylase and amylo-1, 6-glucosidase deficiency.

J Pediatr. 1969 Jan;74(1):90-4. doi: 10.1016/s0022-3476(69)80012-0.

Amylo-1,60glucosidase deficiency (glycogenosis type III) in the Faroe Islands.

Hum Hered. 1975;25(2):115-26. doi: 10.1159/000152716.

Absence of acid maltase in glycogenesis type 2(Pompe's disease) in tissue culture.

Am J Dis Child. 1969 Jan;117(1):108-11. doi: 10.1001/archpedi.1969.02100030110013.

[Glycogenosis caused by amylo-1,6-glucosidase deficiency. Myopathy as a lead finding in adults].

Schweiz Med Wochenschr. 1981 Jun 6;111(23):854-8.