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Predisposing effect of spontaneous mesenchymal intimal thickenings of rabbit aorta to early lipid deposition.

作者信息

Lopes de Faria J, Lopes de Faria L

出版信息

Virchows Arch A Pathol Pathol Anat. 1971;353(1):1-9. doi: 10.1007/BF00543359.

DOI:10.1007/BF00543359
PMID:5314488
Abstract
摘要

相似文献

1
Predisposing effect of spontaneous mesenchymal intimal thickenings of rabbit aorta to early lipid deposition.
Virchows Arch A Pathol Pathol Anat. 1971;353(1):1-9. doi: 10.1007/BF00543359.
2
THE EFFECT OF SHORT-TERM CHOLESTEROL FEEDING ON THE DEVELOPMENT OF AORTIC ATHEROMATOSIS IN THE RABBIT. I. THE INFLUENCE OF HYPERCHOLESTEROLAEMIA ON LIPID DEPOSITION IN THE AORTA, LIVER, AND ADRENALS.
J Atheroscler Res. 1965 Jul-Aug;5(4):353-68. doi: 10.1016/s0368-1319(65)80071-0.
3
[The pathogenesis of obliterating arterial processes in hypercholesterinemia].
Thromb Diath Haemorrh. 1969 Nov 15;22(2):351-9.
4
Severe pulmonary atherosclerosis in hypercholesterolemic rabbits.高胆固醇血症兔的严重肺动脉粥样硬化
Arch Pathol. 1974 Jul;98(1):47-50.
5
[Study on the reversion of experimental cholesterolic arteriopathy in rabbits. The surfaces of the aortic plaques studied by scanning electron microscopy during and after cessation of the hypercholesterol diet].
Boll Soc Ital Biol Sper. 1975 Feb 15;51(3):93-4.
6
Role of endothelium and hypercholesterolemia in intimal thickening and lipid accumulation.内皮细胞和高胆固醇血症在内膜增厚和脂质蓄积中的作用。
Am J Pathol. 1979 Apr;95(1):131-58.
7
[Dynamics of blood catalase and peroxidase activity in rabbits fed cholesterol for long periods of time].
Biull Eksp Biol Med. 1973 Mar;75(1):61-3.
8
[Development of the connective tissue in the wall of the rabbit aorta in experimental atherosclerosis].[实验性动脉粥样硬化中兔主动脉壁结缔组织的发育]
Biull Eksp Biol Med. 1988 Jan;105(1):90-4.
9
Canine hyperlipoproteinemia and atherosclerosis. Accumulation of lipid by aortic medial cells in vivo and in vitro.犬高脂蛋白血症与动脉粥样硬化。主动脉中膜细胞在体内和体外的脂质蓄积。
Am J Pathol. 1977 Apr;87(1):205-26.
10
Deposition pattern of monocytes and fatty streak development in hypercholesterolemic rabbits.高胆固醇血症兔单核细胞的沉积模式与脂肪条纹形成
Atherosclerosis. 1995 Jul;116(1):103-15. doi: 10.1016/0021-9150(95)05533-3.

引用本文的文献

1
Experimental arteriosclerosis. I. Fibrous plaque formation in primates, an electron microscope study.实验性动脉硬化。I. 灵长类动物纤维斑块形成的电子显微镜研究。
J Exp Med. 1972 Oct 1;136(4):769-89. doi: 10.1084/jem.136.4.769.

本文引用的文献

1
A reliable method for the histologic evaluation of tissue lipid. The joint use of Sudan IV-propylene glycol, phosphin 3R (an aqueous fluorochrome), and the cryostat.一种用于组织脂质组织学评估的可靠方法。苏丹IV-丙二醇、磷光3R（一种水性荧光染料）和低温恒温器的联合使用。
Tech Bull Regist Med Technol. 1962 Mar;32:49-53.
2
The use of sodium chloride-containing fixatives in minimizing cellular distortion in histological and cytochemical preparations.
J Histochem Cytochem. 1961 Jul;9:409-14. doi: 10.1177/9.4.409.
3
Alteration of healing responses in experimental wounds in arteries and other tissues by hypercholesterolemia.高胆固醇血症对动脉及其他组织实验性伤口愈合反应的影响。
Aerosp Med. 1961 Jan;32:25-9.
4
Technics for studying atherosclerotic lesions.研究动脉粥样硬化病变的技术。
Lab Invest. 1958 Jan-Feb;7(1):42-7.
5
Intimal thickening and dietary atherosclerosis.内膜增厚与饮食性动脉粥样硬化。
Arch Pathol. 1967 Jul;84(1):49-53.
6
Role of medial changes in the pathogenesis of the diffuse intimal thickening and in atherogenesis; an approach based upon quantitative histological changes in the media of the ascending aorta.中膜变化在弥漫性内膜增厚发病机制及动脉粥样硬化形成中的作用；基于升主动脉中膜定量组织学变化的研究方法。
J Atheroscler Res. 1965 Sep-Oct;5(5):509-15. doi: 10.1016/s0368-1319(65)80023-0.
7
Comparative studies on tissue cholesterol.组织胆固醇的比较研究。
Arch Pathol. 1968 Dec;86(6):585-96.
8
On the origin of smooth muscle cells in the intimal thickening of rabbit aorta following orthostatic collapse.关于兔主动脉在体位性虚脱后内膜增厚中平滑肌细胞的起源
Beitr Pathol Anat. 1970;140(3):333-44.
9
A model for the rapid production of atheromatous lesions in rabbits.
Lab Invest. 1970 Jan;22(1):94-9.
10
Role of wall factors in the pathogenesis of coronary atherosclerosis. An approach based upon quantitative histological changes in the non-atheromatous intimal thickening and in the tunica media.血管壁因素在冠状动脉粥样硬化发病机制中的作用。基于非动脉粥样硬化性内膜增厚和中膜定量组织学变化的研究方法。
J Atheroscler Res. 1968 Mar-Apr;8(2):291-302.