Groman N B, Suzuki G
J Bacteriol. 1965 Oct;90(4):1007-12. doi: 10.1128/jb.90.4.1007-1012.1965.
Groman, Neal B. (University of Washington, Seattle), and Grace Suzuki. Effect of ribonucleic acid phage superinfection on lysis-inhibited Escherichia coli. J. Bacteriol. 90:1007-1012. 1965.-Induced culturesof Escherichia coli K-12(lambda112)F(+) were superinfected with ribonucleic acid phage f2 at various times to test for the specificity of lysis inhibition and the concurrent inhibition of growth. When f2 superinfection occurred within 90 min after induction, lysis was observed in normally lysis-inhibited cultures. Later superinfections produced very little lysis. Following early superinfection, both lambda112 and f2 phages were produced in induced cells. When superinfection occurred during the period in which growth was inhibited, f2 production was totally inhibited. The inhibition of f2 was not due to its inability to adsorb, nor was it due to damage inflicted on cells by ultraviolet irradiation or to exhaustion of the medium. The data suggest that inhibition of lysis of induced K-12(lambda112)F(+) is phage-specific, whereas the accompanying inhibition of growth is nonspecific.
格罗曼，尼尔·B.（华盛顿大学，西雅图），以及格蕾丝·铃木。核糖核酸噬菌体超感染对裂解抑制型大肠杆菌的影响。《细菌学杂志》90:1007 - 1012。1965年。——在不同时间用核糖核酸噬菌体f2对诱导培养的大肠杆菌K - 12（λ112）F（+）进行超感染，以测试裂解抑制的特异性和同时发生的生长抑制。当在诱导后90分钟内发生f2超感染时，在正常情况下有裂解抑制的培养物中观察到了裂解现象。较晚的超感染产生的裂解很少。早期超感染后，诱导细胞中产生了λ112和f2两种噬菌体。当在生长受到抑制的时期发生超感染时，f2的产生被完全抑制。f2的抑制不是由于其无法吸附，也不是由于紫外线照射对细胞造成的损伤或培养基的耗尽。数据表明，诱导的K - 12（λ112）F（+）的裂解抑制是噬菌体特异性的，而伴随的生长抑制是非特异性的。
