Endogenous induction of the galactose operon in Escherichia coli K12.
作者信息
Wu H C, Kalckar H M
出版信息
Proc Natl Acad Sci U S A. 1966 Mar;55(3):622-9. doi: 10.1073/pnas.55.3.622.
Abstract
摘要
相似文献
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Endogenous induction of the galactose operon in Escherichia coli K12.大肠杆菌K12中半乳糖操纵子的内源性诱导
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本文引用的文献
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HEREDITARY DEFECTS IN GALACTOSE METABOLISM IN ESCHERICHIA COLI MUTANTS, I. DETERMINATION OF ENZYME ACTIVITIES.大肠杆菌突变体中半乳糖代谢的遗传性缺陷，I. 酶活性的测定
Proc Natl Acad Sci U S A. 1959 Dec;45(12):1776-86. doi: 10.1073/pnas.45.12.1776.
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ENZYME INDUCTION AS AN ALL-OR-NONE PHENOMENON.酶诱导作为一种全或无现象。
Proc Natl Acad Sci U S A. 1957 Jul 15;43(7):553-66. doi: 10.1073/pnas.43.7.553.
3
A method for isolating constitutive mutants for carbohydrate-catabolizing enzymes.一种分离碳水化合物分解酶组成型突变体的方法。
Biochim Biophys Acta. 1962 Jul 2;60:422-4. doi: 10.1016/0006-3002(62)90423-7.
4
Control of inducibility of enzymes of the galactose sequence in Escherichia coli.大肠杆菌中半乳糖序列酶诱导性的控制
Proc Natl Acad Sci U S A. 1962 Jan 15;48(1):32-40. doi: 10.1073/pnas.48.1.32.
5
Separate permeases for the accumulation of methyl-beta-D-galactoside and methyl-beta-D-thiogalactoside in Escherichia coli.大肠杆菌中用于积累甲基-β-D-半乳糖苷和甲基-β-D-硫代半乳糖苷的不同通透酶。
Biochim Biophys Acta. 1959 Apr;32:599-601. doi: 10.1016/0006-3002(59)90659-6.
6
7
MUTATIONS IN ESCHERICHIA COLI THAT AFFECT URIDINE DIPHOSPHATE GLUCOSE PYROPHOSPHORYLASE ACTIVITY AND GALACTOSE FERMENTATION.影响尿苷二磷酸葡萄糖焦磷酸化酶活性和半乳糖发酵的大肠杆菌突变
Biochim Biophys Acta. 1963 Sep 10;74:608-20. doi: 10.1016/0006-3002(63)91412-4.
8
[REGULATORY MECHANISMS IN THE BIOSYNTHESIS OF THE ENZYMES OF GALACTOSE METABOLISM IN ESCHERICHIA COLI K 12. I. THE INDUCED BIOSYNTHESIS OF GALACTOKINASE AND THE SIMULTANEOUS INDUCTION OF THE ENZYMATIC SEQUENCE].[大肠杆菌K12中半乳糖代谢酶生物合成的调控机制。I. 半乳糖激酶的诱导生物合成及酶序列的同步诱导]
J Mol Biol. 1963 Aug;7:164-82. doi: 10.1016/s0022-2836(63)80044-3.
9
A CHEMICAL BASIS FOR THE HOST-INDUCED MODIFICATION OF T-EVEN BACTERIOPHAGES.宿主诱导T偶数噬菌体修饰的化学基础
Proc Natl Acad Sci U S A. 1963 Aug;50(2):300-5. doi: 10.1073/pnas.50.2.300.
10
HOST-INDUCED MODIFICATION OF T-EVEN PHAGES DUE TO DEFECTIVE GLUCOSYLATION OF THEIR DNA.宿主诱导的T偶数噬菌体修饰，归因于其DNA糖基化缺陷
Proc Natl Acad Sci U S A. 1963 Aug;50(2):297-300. doi: 10.1073/pnas.50.2.297.
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