多肽抗生素对蛋白质合成的抑制作用。3. 抑制的核糖体位点。 - Suppr

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超能文献

Inhibition of protein synthesis by polypeptide antibiotics. 3. Ribosomal site of inhibition.

作者信息

Ennis H L

出版信息

Mol Pharmacol. 1966 Sep;2(5):444-53.

PMID:5339335

Abstract

摘要

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Inhibition of protein synthesis by polypeptide antibiotics. 3. Ribosomal site of inhibition.多肽抗生素对蛋白质合成的抑制作用。3. 抑制的核糖体位点。

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引用本文的文献

Lasting damage to bacterial ribosomes by reversibly bound virginiamycin M.可逆结合的维吉尼亚霉素M对细菌核糖体造成的持久损伤。

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Bacterial resistance to the synergistic antibiotics of the PA 114, streptogramin, and vernamycin complexes.细菌对PA 114、链阳性菌素和维纳霉素复合物协同抗生素的耐药性。

J Bacteriol. 1967 Jun;93(6):1881-7. doi: 10.1128/jb.93.6.1881-1887.1967.

Mutants of Escherichia coli sensitive to antibiotics.对抗生素敏感的大肠杆菌突变体。

J Bacteriol. 1971 Aug;107(2):486-90. doi: 10.1128/jb.107.2.486-490.1971.

Antibiotic inhibitors of the bacterial ribosome.细菌核糖体的抗生素抑制剂。

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Antibiotics of the virginiamycin family, inhibitors which contain synergistic components.维吉尼亚霉素家族的抗生素，含有协同成分的抑制剂。

Microbiol Rev. 1979 Jun;43(2):145-92. doi: 10.1128/mr.43.2.145-192.1979.

The in vitro binding of virginiamycin M to bacteria ribosomes and ribosomal subunits.维吉尼亚霉素M与细菌核糖体及核糖体亚基的体外结合

Mol Gen Genet. 1978 Oct 25;166(1):53-9. doi: 10.1007/BF00379729.

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