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The control of sodium excretion with normal and reduced nephron populations. The pre-eminence of third factor.

作者信息

Bricker N S

出版信息

Am J Med. 1967 Sep;43(3):313-21. doi: 10.1016/0002-9343(67)90188-x.

DOI:10.1016/0002-9343(67)90188-x
PMID:5341544
Abstract
摘要

相似文献

1
The control of sodium excretion with normal and reduced nephron populations. The pre-eminence of third factor.
Am J Med. 1967 Sep;43(3):313-21. doi: 10.1016/0002-9343(67)90188-x.
2
Mechanism of change in the excretion of sodium per nephron when renal mass is reduced.肾单位数量减少时单个肾单位钠排泄的变化机制。
J Clin Invest. 1969 Jun;48(6):1002-6. doi: 10.1172/JCI106056.
3
Endogenous ANP augments fractional excretion of Pi, Ca, and Na in rats with reduced renal mass.内源性心钠肽可增加肾质量降低大鼠的磷、钙和钠排泄分数。
Am J Physiol. 1988 Dec;255(6 Pt 2):F1091-7. doi: 10.1152/ajprenal.1988.255.6.F1091.
4
Role of renal kallikrein in the increased fractional sodium excretion in the rat remnant kidney model of chronic renal failure.
Adv Exp Med Biol. 1989;247B:449-54. doi: 10.1007/978-1-4615-9546-5_74.
5
Role of atrial natriuretic peptide in adaptation of sodium excretion with reduced renal mass.心房利钠肽在肾单位减少时钠排泄适应性中的作用。
J Clin Invest. 1986 Apr;77(4):1395-8. doi: 10.1172/JCI112447.
6
Renal excretion of sodium in arterial hypertension.动脉高血压时钠的肾脏排泄
Circulation. 1959 Oct;20:498-510. doi: 10.1161/01.cir.20.4.498.
7
[Effect of stimulation of the vagus nerve on the sodium excretion of the kidney].[迷走神经刺激对肾脏钠排泄的影响]
Kiserl Orvostud. 1955 Nov;7(6):636-41.
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[Electrolyte excretion by the kidney in normal pregnancy].[正常妊娠时肾脏的电解质排泄]
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Renal excretion of sodium and the concept of volume receptor.钠的肾排泄与容量感受器的概念
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Angiotensin AT₂ receptors: control of renal sodium excretion and blood pressure.血管紧张素AT₂受体:对肾钠排泄和血压的调控
Adv Exp Med Biol. 2011;707:115-6. doi: 10.1007/978-1-4419-8002-1_25.

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Natriuretic hormone: the ultimate determinant of the preservation of external sodium balance.
利钠激素:维持机体钠平衡的最终决定因素。
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The central mechanism underlying hypertension: a review of the roles of sodium ions, epithelial sodium channels, the renin-angiotensin-aldosterone system, oxidative stress and endogenous digitalis in the brain.高血压的中心机制:钠离子、上皮钠通道、肾素-血管紧张素-醛固酮系统、氧化应激和脑内内源性洋地黄素作用的综述。
Hypertens Res. 2011 Nov;34(11):1147-60. doi: 10.1038/hr.2011.105. Epub 2011 Aug 4.
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The sodium pump and cardiotonic steroids-induced signal transduction protein kinases and calcium-signaling microdomain in regulation of transporter trafficking.钠泵及强心甾体诱导的信号转导蛋白激酶与钙信号微区在转运体转运调节中的作用
Biochim Biophys Acta. 2010 Dec;1802(12):1237-45. doi: 10.1016/j.bbadis.2010.01.013. Epub 2010 Feb 6.
6
Quantitative excretion of water and sodium load by isolated dog kidney: autonomous renal response to blood dilution factors.离体犬肾对水和钠负荷的定量排泄:肾脏对血液稀释因子的自主反应。
Pflugers Arch. 1968;304(1):30-45. doi: 10.1007/BF00586716.
7
Disproportionate inhibition of sodium reabsorption in the unilaterally diseased kidney of dog and man after an acute saline load.急性盐水负荷后,犬和人单侧患病肾脏中钠重吸收的不成比例抑制。
J Clin Invest. 1971 Feb;50(2):422-31. doi: 10.1172/JCI106509.
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Inhibition of transepithelial sodium transport in the frog skin by a low molecular weight fraction of uremic serum.尿毒症血清低分子量组分对蛙皮跨上皮钠转运的抑制作用。
J Clin Invest. 1971 Feb;50(2):303-11. doi: 10.1172/JCI106495.
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Non-occurrence of a natriuretic factor in circulating blood of rats after expansion of the extracellular or the intravascular space.细胞外或血管内空间扩张后大鼠循环血液中利钠因子未出现。
Pflugers Arch. 1970;318(1):21-34. doi: 10.1007/BF00588540.
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[Renal escape phenomenon in hypophyseal diseases].[垂体疾病中的肾逃逸现象]
Klin Wochenschr. 1969 Aug 15;47(16):866-70. doi: 10.1007/BF01879918.