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Quantitative excretion of water and sodium load by isolated dog kidney: autonomous renal response to blood dilution factors.

作者信息

Nizet A, Godon J P, Mahieu P

出版信息

Pflugers Arch. 1968;304(1):30-45. doi: 10.1007/BF00586716.

DOI:10.1007/BF00586716
PMID:5748566
Abstract
摘要

相似文献

1
Quantitative excretion of water and sodium load by isolated dog kidney: autonomous renal response to blood dilution factors.离体犬肾对水和钠负荷的定量排泄：肾脏对血液稀释因子的自主反应。
Pflugers Arch. 1968;304(1):30-45. doi: 10.1007/BF00586716.
2
Excretion of water and sodum loading by isolated perfused dog kidney.离体灌注犬肾对水和钠负荷的排泄
Pflugers Arch Gesamte Physiol Menschen Tiere. 1967;297(3):156-61. doi: 10.1007/BF00362707.
3
Influence of serumalbumin and dextran on sodium and water excretion by the isolated dog kidney.血清白蛋白和右旋糖酐对离体狗肾钠和水排泄的影响。
Pflugers Arch Gesamte Physiol Menschen Tiere. 1968;301(1):7-15. doi: 10.1007/BF00412414.
4
Effects of prolonged administration of vasopressin on plasma sodium and on renal excretion of electrolytes and water.血管加压素长期给药对血浆钠以及肾脏电解质和水排泄的影响。
J Pharmacol Exp Ther. 1971 Jul;178(1):141-51.
5
Effects of indomethacin on renal hemodynamics and on water and sodium excretion by the isolated dog kidney.
Pflugers Arch. 1975 Jun 26;357(3-4):243-52. doi: 10.1007/BF00585979.
6
[Adaptation of Rana esculenta to various environments. A special study of renal excretion of water and electrolytes during changes in environment].[食用蛙对各种环境的适应。环境变化期间肾脏对水和电解质排泄的专项研究]
Comp Biochem Physiol. 1969 Apr;29(1):27-50. doi: 10.1016/0010-406x(69)91724-1.
7
Autonomous response of dog kidney to increased blood pressure with and without saline loading.犬肾在有或无盐水负荷情况下对血压升高的自主反应。
Pflugers Arch. 1969;313(3):245-51. doi: 10.1007/BF00586746.
8
The effect of chronic hypotonic volume expansion on the renal regulation of acid-base equilibrium.慢性低渗性容量扩张对肾脏酸碱平衡调节的影响。
J Clin Invest. 1972 Nov;51(11):2928-40. doi: 10.1172/JCI107117.
9
Renal sodium and water excretion after unilateral splanchnicotomy in the dog.犬单侧内脏神经切断术后的肾钠和水排泄
Acta Physiol Acad Sci Hung. 1971;39(4):283-91.
10
Persistence of the natriuretic response to isotonic saline load during hemorrhagic hypotension in the dog.
Proc Soc Exp Biol Med. 1969 May;131(1):265-71. doi: 10.3181/00379727-131-33855.

引用本文的文献

1
Autonomous response of dog kidney to increased blood pressure with and without saline loading.犬肾在有或无盐水负荷情况下对血压升高的自主反应。
Pflugers Arch. 1969;313(3):245-51. doi: 10.1007/BF00586746.
2
[Renal escape phenomenon in hypophyseal diseases].[垂体疾病中的肾逃逸现象]
Klin Wochenschr. 1969 Aug 15;47(16):866-70. doi: 10.1007/BF01879918.
3
The effect of the baroreceptors on activity in efferent sympathetic nerves evoked by peripheral nerve stimulation in the dog.压力感受器对狗外周神经刺激诱发的传出交感神经活动的影响。

本文引用的文献

1
[THE SODIUM CONCENTRATION IN THE MACULA DENSA CELLS AS A REGULATING FACTOR FOR GLOMERULAR FILTRATION (MICROPUNCTURE EXPERIMENTS)].[致密斑细胞中的钠浓度作为肾小球滤过的调节因子（微穿刺实验）]
Klin Wochenschr. 1965 Apr 15;43:410-3. doi: 10.1007/BF01483845.
2
THE EFFECTS OF SALINE INFUSION ON SODIUM REABSORPTION BY THE PROXIMAL TUBULE OF THE DOG.盐水输注对犬近端小管钠重吸收的影响。
J Clin Invest. 1965 Jul;44(7):1160-70. doi: 10.1172/JCI105223.
3
INHIBITION OF RENAL TUBULAR SODIUM REABSORPTION BY HYPERNATREMIA.高钠血症对肾小管钠重吸收的抑制作用
J Physiol. 1969 Sep;204(1):15P-16P.
4
Excretion and tubular reabsorption of sodium, glucose and phosphate by isolated dog kidneys. Influence of blood dilution.离体犬肾对钠、葡萄糖和磷酸盐的排泄及肾小管重吸收。血液稀释的影响。
Pflugers Arch. 1972;332(3):248-58. doi: 10.1007/BF00587452.
5
A humoral component of the natriuretic mechanism in sustained blood volume expansion.持续血容量扩张时利钠机制的一种体液成分。
J Clin Invest. 1972 Oct;51(10):2631-44. doi: 10.1172/JCI107081.
6
Reduction of the glomerular filtration rate by the efferent blood from a kidney taken from a previously dehydrated dog.取自先前已脱水犬的肾脏的传出血液对肾小球滤过率的降低作用。
Pflugers Arch. 1973;345(4):327-33. doi: 10.1007/BF00585851.
7
The mechanisms of fast renal compensation.快速肾代偿的机制。
Pflugers Arch. 1973 Jul 6;341(3):209-17. doi: 10.1007/BF00592790.
8
The control of sodium excretion following saline infusion in dogs.犬静脉输注生理盐水后钠排泄的控制
Pflugers Arch. 1974;350(4):287-98. doi: 10.1007/BF00592637.
9
[Humoral inhibition of epithelial sodium transport after acute expansion of extracellular volume. Further evidence of a natriuretic hormone (author's transl)].[细胞外液量急性扩张后对上皮钠转运的体液抑制作用。利钠激素的进一步证据（作者译）]
Klin Wochenschr. 1974 Sep 1;52(17):801-8. doi: 10.1007/BF01468860.
10
Comparative evaluation of fractional excretion of sodium following saline infusion in transplanted kidneys and in isolated perfused kidneys in conditions of previous high or low dietary sodium intake.在先前高钠或低钠饮食摄入情况下，对移植肾和离体灌注肾输注生理盐水后钠排泄分数的比较评估。
Pflugers Arch. 1976 Jan 30;361(2):121-6. doi: 10.1007/BF00583455.
J Clin Invest. 1965 Jul;44(7):1144-50. doi: 10.1172/JCI105221.
4
Evidence for a direct effect of serum sodium concentration on sodium reabsorption.血清钠浓度对钠重吸收有直接影响的证据。
J Clin Invest. 1962 Apr;41(4):850-9. doi: 10.1172/JCI104542.
5
Studies on the efferent mechanism of the sodium diuresis which follows the administration of intravenous saline in the dog.关于给狗静脉注射生理盐水后出现的钠利尿传出机制的研究。
Clin Sci. 1961 Oct;21:249-58.
6
A comparison of sodium excretion following the infusion of saline into systemic and portal veins in the dog: evidence for a hepatic role in the control of sodium excretion.犬经体静脉和门静脉输注生理盐水后钠排泄的比较：肝脏在钠排泄控制中作用的证据
Clin Sci. 1967 Dec;33(3):481-7.
7
Demonstraton of a role of physical factors as determinants of the natriuretic response to volume expansion.证明物理因素作为容量扩张钠利尿反应决定因素的作用。
J Clin Invest. 1967 Dec;46(12):1963-78. doi: 10.1172/JCI105686.
8
[The biological demonstration of a humoral natriuretic principle in the urine of healthy subjects].[健康受试者尿液中体液性利钠原理的生物学证明]
Klin Wochenschr. 1967 Jan 1;45(1):30-4. doi: 10.1007/BF01745735.
9
Changes in proximal and distal tubular reabsorption produced by rapid expansion of extracellular fluid.细胞外液快速扩充引起的近端和远端肾小管重吸收的变化。
J Clin Invest. 1967 Jul;46(7):1254-63. doi: 10.1172/JCI105618.
10
Functional characteristics of the diluting segment of the dog nephron and the effect of extracellular volume expansion on its reabsorptive capacity.犬肾单位稀释段的功能特性及细胞外液量扩张对其重吸收能力的影响。
J Clin Invest. 1967 Jul;46(7):1178-88. doi: 10.1172/JCI105611.