Peuscher F W, van Aken W G, van Mourik J A, Swaak A J, Sie L H, Statius van Eps L W
Scand J Haematol. 1979 Oct;23(4):257-64. doi: 10.1111/j.1600-0609.1979.tb02859.x.
A case of severe haemorrhagic diathesis due to acquired deficiency of factor X (both immunologically and in procoagulant activity) is presented. The clinical and serological features of this case indicated mycoplasma pneumonial infection. Factor X in the peripheral blood did not appear to be influenced by administration of vitamin K, prothrombin-complex concentrate, fresh plasma or fresh whole blood. Circulating inhibitors of blood coagulation were absent and systemic amyloidosis could not be demonstrated. After 20 d, factor X spontaneously returned to normal. In view of the absence of other known causes of factor X deficiency, a possible relationship with mycoplasma pneumonial infection is suggested.
本文报告1例因获得性X因子缺乏（免疫及促凝血活性方面）导致的严重出血素质病例。该病例的临床及血清学特征提示支原体肺炎感染。外周血中的X因子似乎不受维生素K、凝血酶原复合物浓缩剂、新鲜血浆或新鲜全血输注的影响。未发现循环血液凝固抑制剂，也未证实存在系统性淀粉样变性。20天后，X因子自发恢复正常。鉴于不存在其他已知的X因子缺乏原因，提示可能与支原体肺炎感染有关。
