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Regression and persistence of hyperplastic hepatic nodules induced by N-2-Fluorenylacetamide and their relationship to hepatocarcinogenesis.

作者信息

Teebor G W, Becker F F

出版信息

Cancer Res. 1971 Jan;31(1):1-3.

PMID:5540951
Abstract
摘要

相似文献

1
Regression and persistence of hyperplastic hepatic nodules induced by N-2-Fluorenylacetamide and their relationship to hepatocarcinogenesis.N-2-芴基乙酰胺诱导的增生性肝结节的消退与持续存在及其与肝癌发生的关系。
Cancer Res. 1971 Jan;31(1):1-3.
2
Early stages of N-2-fluorenylacetamide-induced hepatocarcinogenesis in male and female rats and effect of gonadectomy on liver neoplastic conversion and neoplastic development.N-2-芴基乙酰胺诱导的雄性和雌性大鼠肝癌发生早期阶段以及性腺切除术对肝脏肿瘤转化和肿瘤发展的影响。
J Natl Cancer Inst. 1984 Jul;73(1):141-9.
3
Suppression of N-nitrosodiethylamine induced hepatocarcinogenesis by silymarin in rats.水飞蓟素对大鼠N-亚硝基二乙胺诱导肝癌发生的抑制作用
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Prevention of hepatocarcinogenesis with phosphatidylcholine and menaquinone-4: in vitro and in vivo experiments.磷脂酰胆碱和维生素K4预防肝癌发生的体内外实验
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Bone marrow-derived cells fuse with hepatic oval cells but are not involved in hepatic tumorigenesis in the choline-deficient ethionine-supplemented diet rat model.在胆碱缺乏且补充乙硫氨酸的饮食大鼠模型中,骨髓来源的细胞与肝卵圆细胞融合,但不参与肝脏肿瘤发生。
Carcinogenesis. 2008 Feb;29(2):448-54. doi: 10.1093/carcin/bgm279. Epub 2008 Jan 3.
6
Bile ductular cell reaction with senescent hepatocytes in chronic viral hepatitis is lost during hepatocarcinogenesis.在慢性病毒性肝炎中,胆管细胞与衰老肝细胞的反应在肝癌发生过程中消失。
Pathol Int. 2009 Jul;59(7):471-8. doi: 10.1111/j.1440-1827.2009.02395.x.
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Enhanced survival and absence of progressive growth of transplanted rat liver altered eosinophilic foci and neoplastic nodules in phenobarbital-treated rats.苯巴比妥处理的大鼠中,移植大鼠肝脏的存活率提高,且无渐进性生长,嗜酸性病灶和肿瘤结节发生改变。
J Natl Cancer Inst. 1983 Oct;71(4):849-54.
8
Molecular pathology of early stage chemically induced hepatocarcinogenesis.早期化学诱导肝癌发生的分子病理学
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Gangliosides of liver tumors induced by N-2 fluorenylacetamide III. Galactosyl and sialyl transferases in single carcinomas and nodules.N-2-芴基乙酰胺诱导的肝肿瘤神经节苷脂III. 单个癌和结节中的半乳糖基转移酶和唾液酸基转移酶
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Analysis of gene expression in different stages of MeIQx-induced rat hepatocarcinogenesis.4-甲基咪唑喹喔啉(MeIQx)诱导大鼠肝癌发生不同阶段的基因表达分析。
Oncol Rep. 2007 Apr;17(4):747-52.

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High-Affinity Low-Capacity and Low-Affinity High-Capacity N-Acetyl-2-Aminofluorene (AAF) Macromolecular Binding Sites Are Revealed During the Growth Cycle of Adult Rat Hepatocytes in Primary Culture.在原代培养的成年大鼠肝细胞生长周期中,揭示了高亲和力低容量和低亲和力高容量 N-乙酰-2-氨基芴(AAF)大分子结合位点。
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N-Acetyl-2-Aminofluorene (AAF) Processing in Adult Rat Hepatocytes in Primary Culture Occurs by High-Affinity Low-Velocity and Low-Affinity High-Velocity AAF Metabolite-Forming Systems.
N-乙酰-2-氨基芴(AAF)在原代培养的成年大鼠肝细胞中的代谢途径为高亲和力低速度和低亲和力高速度 AAF 代谢产物形成系统。
Toxicol Sci. 2018 May 1;163(1):26-34. doi: 10.1093/toxsci/kfy006.
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The potential role of liver stem cells in initiation of primary liver cancer.肝干细胞在原发性肝癌起始过程中的潜在作用。
Hepatol Int. 2016 Nov;10(6):893-901. doi: 10.1007/s12072-016-9730-9. Epub 2016 May 2.
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Age dependence of oval cell responses and bile duct carcinomas in male fischer 344 rats fed a cyclic choline-deficient, ethionine-supplemented diet.雄性 Fischer 344 大鼠周期性胆碱缺乏、乙硫氨酸补充饮食喂养下的卵圆细胞反应和胆管癌的年龄依赖性。
Hepatology. 2010 Nov;52(5):1750-7. doi: 10.1002/hep.23880.
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Stem cells and liver regeneration.干细胞与肝脏再生。
Gastroenterology. 2009 Aug;137(2):466-81. doi: 10.1053/j.gastro.2009.05.044. Epub 2009 May 24.
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Alpha-fetoprotein, stem cells and cancer: how study of the production of alpha-fetoprotein during chemical hepatocarcinogenesis led to reaffirmation of the stem cell theory of cancer.甲胎蛋白、干细胞与癌症:化学性肝癌发生过程中甲胎蛋白产生的研究如何促使癌症干细胞理论得到再次确认。
Tumour Biol. 2008;29(3):161-80. doi: 10.1159/000143402. Epub 2008 Jul 9.
8
Cellular origin of cancer: dedifferentiation or stem cell maturation arrest?癌症的细胞起源:去分化还是干细胞成熟停滞?
Environ Health Perspect. 1993 Dec;101 Suppl 5(Suppl 5):15-26. doi: 10.1289/ehp.93101s515.
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Quantitative in situ image analysis of apoptosis in well and poorly differentiated tumors from rat liver.大鼠肝脏高分化和低分化肿瘤细胞凋亡的定量原位图像分析
Am J Pathol. 1995 Dec;147(6):1626-32.
10
Hepatic nodular lesions in rats and mice: their significance.大鼠和小鼠肝脏结节性病变:其意义。
Br J Cancer. 1980 Mar;41(3):505-6. doi: 10.1038/bjc.1980.85.