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The genetic control of xanthine dehydrogenase and urate oxidase synthess in Aspergillus nidulans.

作者信息

Scazzocchio C, Darlington A J

出版信息

Bull Soc Chim Biol (Paris). 1967 Dec 18;49(11):1503-8.

PMID:5583651
Abstract
摘要

相似文献

1
The genetic control of xanthine dehydrogenase and urate oxidase synthess in Aspergillus nidulans.构巢曲霉中黄嘌呤脱氢酶和尿酸氧化酶合成的遗传控制
Bull Soc Chim Biol (Paris). 1967 Dec 18;49(11):1503-8.
2
The induction and repression of the enzymes of purine breakdown in Aspergillus nidulans.构巢曲霉中嘌呤分解酶的诱导与抑制
Biochim Biophys Acta. 1968 Sep 24;166(2):557-68. doi: 10.1016/0005-2787(68)90243-8.
3
A mutation defective in the xanthine alternative pathway of Aspergillus nidulans: its use to investigate the specificity of uaY mediated induction.构巢曲霉黄嘌呤替代途径中的一种缺陷突变:用于研究uaY介导诱导的特异性
Mol Gen Genet. 1978 Sep 8;164(3):303-8. doi: 10.1007/BF00333161.
4
The genetic control of molybdoflavoproteins in Aspergillus nidulans. Allopurinol-resistant mutants constitutive for xanthine-dehydrogenase.构巢曲霉中钼黄素蛋白的遗传控制。对黄嘌呤脱氢酶组成型的别嘌呤醇抗性突变体。
Eur J Biochem. 1973 Jul 16;36(2):428-45. doi: 10.1111/j.1432-1033.1973.tb02928.x.
5
A mutant of Asperigillus nidulans lacking NADP-linked glutamate dehydrogenase.
Mol Gen Genet. 1973 May 9;122(3):261-5. doi: 10.1007/BF00278601.
6
The genetic control of molybdoflavoproteins in Aspergillus nidulans. II. Use of NADH dehydrogenase activity associated with xanthine dehydrogenase to investigate substrate and product inductions.构巢曲霉中钼黄素蛋白的遗传控制。II. 利用与黄嘌呤脱氢酶相关的NADH脱氢酶活性研究底物和产物诱导作用。
Mol Gen Genet. 1973 Sep 5;125(2):147-55. doi: 10.1007/BF00268868.
7
Studies on the stability of immobilized xanthine oxidase and urate oxidase.固定化黄嘌呤氧化酶和尿酸氧化酶的稳定性研究。
Biotechnol Bioeng. 1978 Jul;20(7):1085-85. doi: 10.1002/bit.260200709.
8
Administration of procyanidins from grape seeds reduces serum uric acid levels and decreases hepatic xanthine dehydrogenase/oxidase activities in oxonate-treated mice.给草酸盐处理的小鼠施用葡萄籽原花青素可降低血清尿酸水平,并降低肝脏黄嘌呤脱氢酶/氧化酶活性。
Basic Clin Pharmacol Toxicol. 2004 May;94(5):232-7. doi: 10.1111/j.1742-7843.2004.pto940506.x.
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Spontaneous and induced reversion of ICR-170-induced xanthine dehydrogenase mutants of Aspergillus nidulans.构巢曲霉ICR - 170诱导的黄嘌呤脱氢酶突变体的自发及诱导回复突变
Mutat Res. 1969 Nov-Dec;8(3):521-9. doi: 10.1016/0027-5107(69)90069-4.
10
Biochemical and genetical studies of purine breakdown in Aspergillus.曲霉属中嘌呤分解代谢的生化与遗传学研究。
Nature. 1965 May 8;206(984):599-600. doi: 10.1038/206599a0.

引用本文的文献

1
Purine Acquisition and Synthesis by Human Fungal Pathogens.人类真菌病原体的嘌呤获取与合成
Microorganisms. 2017 Jun 8;5(2):33. doi: 10.3390/microorganisms5020033.
2
Positive regulation in a eukaryote, a study of the uaY gene of Aspergillus nidulans. II. Identification of the effector binding protein.
Mol Gen Genet. 1981;181(1):107-15. doi: 10.1007/BF00339013.
3
Methylammonium resistance in Aspergillus nidulans.构巢曲霉中的甲铵抗性
J Bacteriol. 1969 Jun;98(3):1284-93. doi: 10.1128/jb.98.3.1284-1293.1969.
4
The genetic analysis of regulation of amidase synthesis in Aspergillus nidulans. I. Mutants able to utilize acrylamide.构巢曲霉中酰胺酶合成调控的遗传分析。I. 能够利用丙烯酰胺的突变体
Mol Gen Genet. 1970;108(2):97-106. doi: 10.1007/BF02430516.
5
Molybdate metabolism in Aspergillus nidulans. I. Mutations affecting nitrate reductase and-or xanthine dehydrogenase.构巢曲霉中的钼酸盐代谢。I. 影响硝酸还原酶和/或黄嘌呤脱氢酶的突变
Mol Gen Genet. 1970;108(2):129-45. doi: 10.1007/BF02430519.
6
Regulation of synthesis of glutamate dehydrogenase and glutamine synthetase in micro-organisms.微生物中谷氨酸脱氢酶和谷氨酰胺合成酶合成的调控
Biochem J. 1969 Dec;115(4):769-75. doi: 10.1042/bj1150769.
7
Nitrogen metabolite repression in Aspergillus nidulans.构巢曲霉中的氮代谢物阻遏
Mol Gen Genet. 1973 Nov 2;126(2):111-41. doi: 10.1007/BF00330988.
8
The genetic control of molybdoflavoproteins in Aspergillus nidulans. II. Use of NADH dehydrogenase activity associated with xanthine dehydrogenase to investigate substrate and product inductions.构巢曲霉中钼黄素蛋白的遗传控制。II. 利用与黄嘌呤脱氢酶相关的NADH脱氢酶活性研究底物和产物诱导作用。
Mol Gen Genet. 1973 Sep 5;125(2):147-55. doi: 10.1007/BF00268868.
9
A mutation defective in the xanthine alternative pathway of Aspergillus nidulans: its use to investigate the specificity of uaY mediated induction.构巢曲霉黄嘌呤替代途径中的一种缺陷突变:用于研究uaY介导诱导的特异性
Mol Gen Genet. 1978 Sep 8;164(3):303-8. doi: 10.1007/BF00333161.