Becker A E, Becker M J, Wagenvoort C A
J Pathol. 1977 Mar;121(3):187-91. doi: 10.1002/path.1711210310.
Necropsy findings are presented of two cases of foetal heart failure secondary to premature, in-utero contraction should lead to a redirection of the right heart output either through the pulmonary vascular bed or to the systemic circulation via the foramen ovale. The muscular pulmonary arteries, however, were not dilated, indicating that no excessive pulmonary flow had occurred. The increased volume load on the right heart may have rendered the foramen ovale restrictive, with right-sided heart failures as a consequence. Premature closure of the ductus arteriosus should be considered a cause of foetal or early neonatal death.
本文呈现了两例胎儿心力衰竭的尸检结果，其继发于子宫内过早收缩，这种收缩会导致右心输出量通过肺血管床或经卵圆孔进入体循环而发生重定向。然而，肌性肺动脉并未扩张，这表明未出现过多的肺血流。右心容量负荷增加可能使卵圆孔受限，进而导致右侧心力衰竭。动脉导管过早关闭应被视为胎儿或早期新生儿死亡的一个原因。
