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Ouabain (10(-3)M) inhibited the transfer of glucose, proline, methionine and fluid but not of galactose and 3-O-methyl glucose by sacs of rat everted intestine.2. Ouabain (10(-3)M) inhibited both glucose and mannose metabolism. Hexose metabolism was also reduced by varying the [K] from 4.8 m-equiv/l.3. Ouabain (10(-3)M) or absence of K reduced the stimulation of amino acid, hexose and fluid transfer by mannose. Although presence of ouabain or absence of K did not inhibit endogenous galactose transfer, these conditions prevented mannose from stimulating this transfer.4. Ouabain (10(-3)M) exerted greater effects on hexose and amino acid transfer when K was omitted from the saline and its inhibitory effects were partially overcome by increased serosal [K].5. Ouabain (10(-2)M) caused further inhibition of amino acid and glucose transfer and also reduced the transfer of galactose and 3-O-methyl glucose.6. The implications of these results are discussed with respect to the relationships between cations and hexose and amino acid transfer.

哇巴因（10⁻³M）抑制大鼠外翻肠囊对葡萄糖、脯氨酸、蛋氨酸和液体的转运，但不抑制半乳糖和3 - O - 甲基葡萄糖的转运。
哇巴因（10⁻³M）抑制葡萄糖和甘露糖的代谢。通过改变钾离子浓度从4.8毫当量/升，己糖代谢也会降低。
哇巴因（10⁻³M）或钾离子缺失会降低甘露糖对氨基酸、己糖和液体转运的刺激作用。虽然哇巴因的存在或钾离子的缺失并不抑制内源性半乳糖的转运，但这些条件会阻止甘露糖刺激这种转运。
当盐溶液中省略钾离子时，哇巴因（10⁻³M）对己糖和氨基酸转运的影响更大，其抑制作用可通过增加浆膜侧钾离子浓度而部分被克服。
哇巴因（10⁻²M）进一步抑制氨基酸和葡萄糖的转运，也降低半乳糖和3 - O - 甲基葡萄糖的转运。
就阳离子与己糖和氨基酸转运之间的关系对这些结果的意义进行了讨论。

Ouabain (10(-3)M) inhibited the transfer of glucose, proline, methionine and fluid but not of galactose and 3-O-methyl glucose by sacs of rat everted intestine.2. Ouabain (10(-3)M) inhibited both glucose and mannose metabolism. Hexose metabolism was also reduced by varying the [K] from 4.8 m-equiv/l.3. Ouabain (10(-3)M) or absence of K reduced the stimulation of amino acid, hexose and fluid transfer by mannose. Although presence of ouabain or absence of K did not inhibit endogenous galactose transfer, these conditions prevented mannose from stimulating this transfer.4. Ouabain (10(-3)M) exerted greater effects on hexose and amino acid transfer when K was omitted from the saline and its inhibitory effects were partially overcome by increased serosal [K].5. Ouabain (10(-2)M) caused further inhibition of amino acid and glucose transfer and also reduced the transfer of galactose and 3-O-methyl glucose.6. The implications of these results are discussed with respect to the relationships between cations and hexose and amino acid transfer.

哇巴因（10⁻³M）抑制大鼠外翻肠囊对葡萄糖、脯氨酸、蛋氨酸和液体的转运，但不抑制半乳糖和3 - O - 甲基葡萄糖的转运。
哇巴因（10⁻³M）抑制葡萄糖和甘露糖的代谢。通过改变钾离子浓度从4.8毫当量/升，己糖代谢也会降低。
哇巴因（10⁻³M）或钾离子缺失会降低甘露糖对氨基酸、己糖和液体转运的刺激作用。虽然哇巴因的存在或钾离子的缺失并不抑制内源性半乳糖的转运，但这些条件会阻止甘露糖刺激这种转运。
当盐溶液中省略钾离子时，哇巴因（10⁻³M）对己糖和氨基酸转运的影响更大，其抑制作用可通过增加浆膜侧钾离子浓度而部分被克服。
哇巴因（10⁻²M）进一步抑制氨基酸和葡萄糖的转运，也降低半乳糖和3 - O - 甲基葡萄糖的转运。
就阳离子与己糖和氨基酸转运之间的关系对这些结果的意义进行了讨论。

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