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Resistance of Wistar-Furth rats to the mast cell-damaging effect of horseradish peroxidase.

作者信息

Cotran R S, Karnovsky M J, Goth A

出版信息

J Histochem Cytochem. 1968 May;16(5):382-3. doi: 10.1177/16.5.382.

DOI:10.1177/16.5.382
PMID:5656148
Abstract
摘要

相似文献

1
Resistance of Wistar-Furth rats to the mast cell-damaging effect of horseradish peroxidase.Wistar-Furth大鼠对辣根过氧化物酶肥大细胞损伤作用的抗性。
J Histochem Cytochem. 1968 May;16(5):382-3. doi: 10.1177/16.5.382.
2
Stimulation of glucose metabolism in rat mast cells by antigen, dextran and compound 48-80, used as histamine releasing agents.作为组胺释放剂的抗原、右旋糖酐和化合物48-80对大鼠肥大细胞葡萄糖代谢的刺激作用。
Acta Physiol Scand. 1974 Jul;91(3):339-53. doi: 10.1111/j.1748-1716.1974.tb05689.x.
3
Mast cells, compound 48-80, and prolonged hypothermia in the rat.大鼠体内的肥大细胞、化合物48 - 80与长时间低温
Am J Anat. 1972 Mar;133(3):379-86. doi: 10.1002/aja.1001330311.
4
Further observations on the inhibition of histamine release by 2-deoxyglucose.关于2-脱氧葡萄糖对组胺释放抑制作用的进一步观察
Acta Physiol Scand. 1968 Apr;72(4):425-32. doi: 10.1111/j.1748-1716.1968.tb03867.x.
5
[Tachyphylaxis of the isolated mast cell].
Naunyn Schmiedebergs Arch Pharmakol. 1969;264(3):303-4.
6
Effect of compound 48/80 on mast cells and histamine levels in rat brain and on corticosterone secretion.
Inflamm Res. 1997 Mar;46 Suppl 1:S47-8.
7
Local degranulation of individual rat peritoneal mast cells induced by compound 48-80.化合物48-80诱导的单个大鼠腹膜肥大细胞的局部脱颗粒
Acta Physiol Scand. 1970 May;79(1):1-5. doi: 10.1111/j.1748-1716.1970.tb04696.x.
8
The response of mast cells to compound 48/80 studied with the electron microscope.用电子显微镜研究肥大细胞对化合物48/80的反应。
Lab Invest. 1965 Oct;14(10):1744-63.
9
Quantitative correlation between degranulation and histamine release following exposure of rat mast cells to compound 48-80 in vitro.大鼠肥大细胞体外暴露于化合物48-80后脱颗粒与组胺释放之间的定量相关性。
Acta Physiol Scand. 1970 Jun;79(2):31A-32A.
10
Distribution of mast cells and their reactivity to a histamine liberator in the trigeminal ganglion of the rat.
J Dent Res. 1972 Jul-Aug;51(4):1092-8. doi: 10.1177/00220345720510041701.

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6
Disruption of iridial blood-aqueous barrier in experimental diabetic rats.
Graefes Arch Clin Exp Ophthalmol. 1982;219(4):159-64. doi: 10.1007/BF02156840.
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The mechanisms of blood-brain barrier impairment by hyperosmolar perfusion. An electron cytochemical study comparing exogenous HRP and endogenous antibody to HRP as tracers.高渗灌注导致血脑屏障损伤的机制。一项比较外源性辣根过氧化物酶（HRP）和内源性抗HRP抗体作为示踪剂的电子细胞化学研究。
Acta Neuropathol. 1982;56(2):99-112. doi: 10.1007/BF00690580.
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A controlled serial ultrastructural tracer study of first-set cardiac allograft rejection in the rat. Evidence that the microvascular endothelium is the primary target of graft destruction.大鼠首次心脏同种异体移植排斥反应的对照系列超微结构示踪研究。微血管内皮是移植物破坏的主要靶点的证据。
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